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← Study guidesNUR 110 · Exam 4 Study Guide · updated Nov 2, 2026
NUR 110 · Foundations and Health Promotion

Exam 4 Study Guide

Fluid, Electrolytes and Acid–Base · IV Therapy and Blood · Urinary Elimination

Coverage: Taylor 10e Chapter 41, Fluid, Electrolyte, and Acid–Base Balance, including IV therapy and blood transfusion, and Chapter 38, Urinary Elimination, bladder only. The guide is ordered by concept, not by chapter: Parts 1–5 are Chapter 41, Parts 6–7 are Chapter 38.
Not on this exam: Chapter 31, Perioperative Nursing, which is taught after the exam, and bowel elimination (Chapter 39).
Format: probably taken online and proctored, as earlier exams were; item types are not published.
About the blue boxes. Until the instructor's lecture slides are posted, a blue box marks one of four things: the instructor's own worksheets (the exam worksheet's bold line and its questions, the electrolyte-imbalance grid, the fluid-balance stick figure); an item the Chapter 41 deck marks or embeds (its asterisk and its five built-in questions); one of the Chapter 38 deck's five review questions; or the highlighted skills-lab line that makes catheter insertion testable.

For exam dates and deadlines, see the semester calendar. Nothing in this guide is tied to a particular term.

First, before anything else

What This Edition Covers

Chapter 41 lecture slides still to come This edition is built from the textbook, the course worksheets, the Chapter 41 deck already posted, the Chapter 38 deck (now posted, and worked into Parts 6 and 7), ATI and the course's Lippincott resources. It will be revised once the instructor's Chapter 41 lecture slides are posted, and anything they stress will gain a blue box. Until then, an unmarked topic is not a safe one; it only means nothing has been singled out yet.

The seven parts, in the order the ideas build on each other:

PartTitleChapter
1Where Body Water Lives, and How It Moves41
2Fluid Volume: Too Little, Too Much, in the Wrong Place41
3Electrolytes: the Seven Numbers and What Happens When They Move41
4Acid–Base: Buffers, Lungs, Kidneys, and the Four Imbalances41
5IV Therapy and Blood41
6Urine: How It Is Made, What Normal Looks Like, How You Test It38
7Urinary Problems and Nursing Care38
Pick a route by the time you have

How to Use This Guide

TimeRoute
20 minutesThe Rapid Review: the numbers to know cold and the easy-to-confuse pairs.
An hourThe whole Rapid Review, then every section flagged top-weighted topic beside its title. They cover six areas: electrolyte imbalances, fluid-volume imbalances, IV solutions, IV complications, urinary problems and catheters.
The whole guideParts 1 to 7 in order, each ending with four scenario questions. Then the Summary Recap, then the practice quiz and practice exam: they are the real question bank.
Orientation

What the course says you must be able to do

The exam is written against the course outline's objectives. Chapter 41's two lecture weeks share one list of seven; the skills-lab rows are the ones an exam item can reach.

Which part covers each objective. Twenty-one rows, folded shut; open it to check that nothing in the outline is missing.
Exam 4 objectives, mapped to the parts of this guide
#ChapterObjective, in shortWhere it is covered
W9·141Composition of the major body-fluid compartments.Part 1, Two compartments, and three spaces outside the cells
W9·241Compare diffusion, osmosis and filtration, and hydrostatic, osmotic and oncotic pressure.Part 1, Four ways across a membrane; Push, pull and albumin · Part 2, Third spacing and edema
W9·341Pathophysiology and signs of sodium, potassium, calcium, magnesium and phosphorus imbalances.Part 3, The imbalance grid; Potassium and sodium; Calcium, magnesium and phosphorus
W9·441Normal and abnormal lab and diagnostic findings in fluid and electrolyte balance.Part 3, Seven numbers, seven jobs · Part 2, Measuring fluid status · Part 4, Reading a gas · Part 6, Normal versus abnormal urine
W9·541Tonicities, purposes and complications of IV solutions.Part 1, Tonicity · Part 5, Solutions by tonicity; IV complications
W9·641Nursing management of fluid and electrolyte imbalances.Part 2, Problems, outcomes and interventions · Part 3, The imbalance grid · Part 5
W9·741The nursing process for fluid and electrolyte imbalances.Part 2, Problems, outcomes and interventions
Topic41Regulatory mechanisms; the buffer, respiratory and renal defences of acid–base balance (topic bullets W9·T6–T7).Part 1, Holding the balance · Part 4, Three defences, fastest first
W11·138The physiology of urination.Part 6, The route, and how voiding works
W11·238Factors that influence urinary elimination and patterns.Part 6, What changes voiding
W11·338Normal versus abnormal characteristics and constituents of urine.Part 6, Normal versus abnormal urine; The eight terms
W11·438Causes and effects of incontinence, retention, urinary tract infection and kidney stones.Part 7, Incontinence; Retention; UTI and CAUTI; Kidney stones
W11·538Urinary diversions.Part 7, Urinary diversions
W11·638Nursing management of altered urinary elimination.Part 7, Promoting voiding and continence; Catheters
W11·738The nursing process for altered urinary elimination.Part 7, Writing the problem statements
L6·241skills lab Tell IV-fluid tonicities apart, and read IV labels correctly.Part 5, Solutions by tonicity
L6·341skills lab Monitor IV sites and recognise IV complications.Part 5, IV complications
L6·1041skills lab IV safety: flow rates, drops per minute versus milliliters per hour, pumps, disposal of equipment.Part 5, Running an infusion
L9·238skills lab Insert an indwelling and a straight catheter on a male and a female; the tested skills are 37-5 / 37-6 in the lab outline (9th-edition numbering; 38-2 / 38-3 in Taylor 10e).Part 7, Inserting an indwelling catheter, female and male
L9·338skills lab Assess for bladder distention; discuss the bladder scanner.Part 6, The bladder scanner and the post-void residual
L9·438skills lab Device care, I&O, specimens and cultures, removal.Part 6, Collecting urine specimens · Part 7, Caring for the catheter, and taking it out
Key terms
ICFECFintravascular interstitialtranscellularsolvent solutesensible lossinsensible loss osmosisdiffusionactive transport capillary filtrationhydrostatic pressure oncotic (colloid osmotic) pressurealbumin osmolalitytonicityADH RAASaldosteroneANP
Objective W9·1 · Ch 41

Two compartments, and three spaces outside the cells

Water is the body's most important nutrient. It does six jobs: it carries nutrients, wastes, hormones and blood cells; supports cell metabolism; dissolves electrolytes and nonelectrolytes; keeps body temperature normal; aids digestion and elimination; and lubricates tissue.

Two barriers divide it. The cell membrane separates the fluid inside cells from everything outside them, and the capillary wall separates the blood from the fluid around the cells. That gives two compartments, and the outer one has three spaces.

CompartmentWhere it isShare of body waterShare of adult body weight
ICF (intracellular)Inside the cells~70%35–40%
ECF (extracellular)Outside the cells: the three spaces below~30%15–20%
· InterstitialAround the cells, including lymph10–15%
· IntravascularPlasma, inside the vessels5%
· TranscellularCSF; pericardial, pleural, synovial and intraocular fluid; sweat; digestive secretionssmall
Total body water—100%50–60%
ADULT BODY WEIGHT = 100% Total body water: 50–60% of body weight Not water 40–50% solids, fat INTRACELLULAR (ICF) inside the cells ~70% of body water = 35–40% of body weight the larger, slower-to-lose store cell membrane EXTRACELLULAR (ECF) ~30% of water = 15–20% of weight Interstitial 10–15% around the cells, including lymph Plasma (intravascular) 5% Transcellular CSF, pleural, joint capillary wall
Two denominators in one picture: the bold figures are shares of body water, the grey ones shares of body weight. The ECF spaces are given as shares of weight.
“ICF is 40%” and “ICF is 70%” are both right An option saying the ICF is about 40% looks wrong to anyone who learned 70%, so students reject it. The two figures use different denominators: 70% of the body's water, 40% of body weight. The stem's last few words decide which it wants. Every percentage has a denominator; find it before you compare numbers.

Age, sex and fat

Fat holds little water and muscle holds a lot, so body water varies with build and age. Three groups differ from the healthy adult's 50–60%:

WhoBody waterWhy it matters
InfantConsiderably more water, and more of it in the ECFECF is lost more easily than ICF, so infants slide into a deficit fast.
Women; people with obesityLessMore of their weight is fat.
Over 60About 45% of body weightMore fat, less muscle: a smaller reserve (box below).
After 60, body water is about 45% Ch 41 deck, slide 7: the deck's only asterisk

After age 60, total body water falls to about 45% of body weight, and that raises the risk of fluid imbalance in the older adult. Expect an age-based risk item to turn on it.

Both ends of life are at risk, for opposite reasons. The infant has more water but keeps a large share in the compartment that drains fastest. The older adult has less water in reserve, and a weaker thirst to warn them.

Topic W9·T2 · Ch 41

In and out: about 2,600 mL a day

Water comes from three sources: drinks (the largest), food, and metabolism (water released as food is oxidized). In a healthy adult, intake and output both average about 2,600 mL a day, and urine output roughly tracks what is drunk. Output leaves by two kinds of loss, and one tempting third category is not a loss at all.

KindHookExamples
SensibleYou can see or measure itUrine (the largest output, about 1,500 mL a day), feces, wound drainage
InsensibleIt evaporates unseenWater vapour breathed out; evaporation through the skin
Not one of these: third-spaced fluidIt is not lost, only unavailableFluid trapped in a body space, such as ascites. It leaves the circulation without leaving the body, so weight does not fall (Part 2).

Sources sort sweat differently: Taylor counts evaporation and perspiration through the skin as insensible and visible sweat as sensible; ATI lists sweat as sensible. Breath and unseen skin evaporation are insensible in every source.

Judge the balance over 2–3 days, not one shift: a single day can run short or long and mean nothing. Fever raises insensible loss: a temperature of 101–103 °F (38.3–39.4 °C) adds at least 500 mL a day, and anything above 103 °F adds at least 1,000 mL.

The deck and Taylor split the 2,600 differently between drinks and food, and among skin, lungs and gut. Only the totals and urine's 1,500 mL agree, so those are the figures to know.

Three fluid numbers, three different jobs 2,600 mL a day is the normal turnover, a description rather than an order. 1.5–2 L is a drinking target to thin secretions (Exam 3). 1–2 L in heart failure is a limit (also Exam 3). Before you pick a number, decide which of the three jobs the stem is asking about. Adding up I&O is in the Dosage Calculation guide; what counts, and how to read the total, is in Part 2.
Objective W9·2 · Ch 41

Four ways across a membrane

Body fluid is solutes (electrolytes, and nonelectrolytes such as glucose and urea) dissolved in a solvent, water. Four processes move them. The exam worksheet matches each process to one phrase, shown in the last column.

ProcessWhat movesWhich wayEnergyExampleWorksheet phrase
OsmosisWaterFrom fewer solute particles toward moreNoneThe main way body water moves between compartmentsLower → higher concentration
DiffusionSolutes and gasesHigh concentration to low (“downhill”)NoneO₂ and CO₂ across the alveolar wall (Exam 3)Gases move by this
Active transportSolutesLow concentration to high (“uphill”)ATPThe sodium–potassium pump, which keeps potassium in the cells and sodium outRequires ATP
Capillary filtrationFluid and small solutesHigher pressure to lowerBlood pressureFluid pushed out at the arterial end of a capillaryFluid-permeable membrane
Two processes go “lower to higher” Osmosis and active transport both move something from lower toward higher concentration, so “lower to higher” pulls students toward active transport. The difference is what moves: osmosis moves water toward the concentrated side for free, while active transport pushes a solute uphill and pays in ATP. Capillary filtration moves water across a membrane as well, so “fluid-permeable membrane” can look like osmosis. Here the driving force separates them: osmosis follows concentration, filtration follows pressure, from higher to lower. Of the two water movers, only osmosis fits “lower to higher concentration”, so filtration takes the membrane phrase. Ask what is moving, what drives it, and whether it costs energy; direction alone never decides.
Objective W9·2 · Ch 41

Push, pull and albumin: why fluid leaves a capillary

Osmotic pressure is the pull that dissolved particles exert on water. When the particles are plasma proteins, chiefly albumin, which are too large to leave the vessel, it is called oncotic (colloid osmotic) pressure. Two pressures oppose each other across every capillary wall.

PressurePush or pullMade byWins atResult
HydrostaticPush, out of the vesselBlood pressing on the vessel wallArterial endFiltration: fluid moves into the interstitium
Oncotic (colloid osmotic)Pull, into the vesselPlasma proteins, mainly albuminVenous endReabsorption: fluid returns to the blood

When the push outruns the pull, or the wall itself leaks, fluid collects in the tissues as edema. The four ways that happens, and third spacing, are in Part 2 (Third spacing and edema); the pitting scale was taught in NUR 120 (Exams 2 and 4).

The same pressures run in reverse when blood volume falls. Fluid moves from the interstitium into the vessels; the interstitium is left concentrated and pulls water out of the cells. The body protects the circulating volume first, at the expense of the tissues and then the cells. That is why the early signs of a deficit show in the mouth, the skin and the pulse before the blood pressure falls (Part 2).

Topic W9·T6 · Ch 41

Holding the balance: thirst, three hormones, nine organs

Thirst is the main control on intake. It is run from the hypothalamus and triggered by cells losing water (concentrated ECF draws water out of them) and by falling blood volume. It weakens with age, so older adults are offered fluid at regular intervals rather than when they ask. Output is controlled by three hormones, all acting on the kidney.

HormoneWhere it comes from, and what releases itWhat the kidney doesNet effect
ADH (antidiuretic hormone, vasopressin)Made in the hypothalamus; stored and released by the posterior pituitary. Released when the ECF is concentrated (high sodium, high glucose) or volume is low; switched off when the ECF is dilute or volume is high.Keeps waterLess urine, more concentrated; the ECF is diluted back toward normal
Aldosterone (the end of RAAS)Poor kidney perfusion → renin → angiotensin I → (ACE) → angiotensin II, which constricts vessels and makes the adrenal glands release aldosterone.Keeps sodium (and water with it); excretes potassiumVolume and blood pressure rise; potassium falls
ANP (atrial natriuretic peptide)The atria, when stretched by a high volume.Excretes sodium and waterVolume falls: the opposite of aldosterone

Taylor does not name ANP, so don't expect an item keyed on it.

Aldosterone keeps sodium and dumps potassium This one sentence carries into Parts 3 and 4: aldosterone keeps sodium and water and excretes potassium. Too little aldosterone runs it backwards, so sodium and water are lost and potassium is kept. Large amounts of cortisol act like aldosterone.

The exam worksheet pairs nine organs with one phrase each, as below.

OrganWorksheet phraseWhat it means
KidneysRegulate the ECFAdjust its volume, osmolality, electrolytes and pH; excrete wastes
Heart and vesselsCirculate nutrients and waterPerfuse the kidneys; stretch receptors sense low volume and trigger retention
LungsPromptly correct acid–baseBlow off or keep CO₂ (Part 4); lose about 300 mL of water a day as vapour
Adrenal glandsRegulate with aldosteroneKeep sodium and water, excrete potassium
PituitaryStores and releases ADHADH is made in the hypothalamus
Nervous systemSenses intracellular dehydrationTriggers thirst; osmoreceptors signal the pituitary to release or hold back ADH
GI tractAbsorbs water and nutrientsAlso a route of loss, through vomiting and diarrhea
ParathyroidsRegulate calcium and phosphatePTH raises calcium and lowers phosphate (Part 3)
ThyroidIncreases blood flow with thyroxineMore blood flow to the kidneys, so more urine
“The pituitary produces ADH” The worksheet pairs the pituitary with ADH, so a statement that the pituitary produces it looks safe. The pituitary stores and releases ADH; the hypothalamus makes it, and the kidney is where it acts. On a matching item the pituitary takes “stores/releases”; a true/false item about where ADH is made is false. Read the verb: making, releasing and acting are different organs' jobs.
Objectives W9·2, W9·5 · Ch 41

Tonicity: what a solution does to a cell

Osmolality is the concentration of particles in a fluid. Tonicity compares a solution's particles with plasma's, and so predicts which way water will move by osmosis. Water always moves toward more particles. There are three tonicities:

TonicityParticles compared with plasmaWater movesThe cell
HypotonicFewerOut of the vessel, into the cellsSwells, and may burst
IsotonicThe sameNo net shift; the fluid stays in the vesselUnchanged
HypertonicMoreOut of the cells, into the vesselShrinks
HYPOTONIC solution fewer particles than plasma ISOTONIC solution same particles as plasma HYPERTONIC solution more particles than plasma Water moves IN cell swells, may burst No net movement cell unchanged Water moves OUT cell shrinks
Each cell starts as concentrated as plasma; the dots outside show how concentrated the solution is. Water heads for the more crowded side: into the cell when the solution is sparser, out of it when the solution is more crowded.
Deck question: does a hypertonic solution shrink the cell? Ch 41 deck, slides 24–25: the deck's question #2

The item: true or false, a hypertonic solution, being more concentrated, draws water out of the cells into the intravascular space, and the cells shrink.

True. The solution has more particles than the cell, so water leaves the cell by osmosis and is drawn into the vessels. The cell shrinks while the circulating volume grows.

“Hyper” sounds like bigger Students read “hyper” as “the cell gets bigger” and pick swelling for a hypertonic solution. The prefix describes the solution, not the cell: a hypertonic solution has more particles, so it pulls water out and the cell shrinks. Water moves toward more particles; find where the particles are and the cell's fate follows.

Not one of these: isotonic exercise (Exam 2) is constant muscle tension with movement. It shares the word with an isotonic solution, not the meaning. Which IV bags fall in each group, and the D5W exception, are in Part 5.

Taylor writes osmolality and puts plasma at about 280 mOsm/L; the deck writes osmolarity. The serum osmolality lab value is in Part 2.

Check yourself · Part 1
An 8-month-old and a 40-year-old each have a day of diarrhea and drink about as much as usual. Who is likelier to become fluid-deficient first, and why? Would an 85-year-old be safer than the 40-year-old?
The infant: more of an infant's water sits in the ECF, which is lost more easily than ICF. No, the 85-year-old is also at higher risk, for the opposite reason: body water is down to about 45%, so there is less in reserve, and thirst is weaker.
A patient with cirrhosis and a very low albumin has swollen ankles and ascites. Pulse 112, BP 94/58. A classmate says the edema proves fluid overload. Does the edema explain the vital signs?
No. With little albumin the oncotic pull is weak, so fluid leaks out of the vessels into the tissues and abdomen. The tissues are wet while the bloodstream runs short, which is what the fast pulse and low BP show. Judge the vessels by the pulse and blood pressure, not by the swelling.
A patient has run a temperature of 39.0 °C (102.2 °F) all day. Her measured intake and output for the day are even. A classmate says that means her fluid balance is fine. Is it?
No. A fever of 101–103 °F adds at least 500 mL a day of insensible loss, breathed out and evaporated through the skin, and no I&O sheet measures it. The even sheet hides a deficit: expect to raise her intake as ordered and watch for early FVD signs. An even I&O usually is reassuring, which is why the call tempts. Count the losses you cannot measure before you trust a balance.
After a large blood loss, a patient's urine falls to 20 mL/h, dark and concentrated. A student says the kidneys must be failing. What is the more likely explanation, and does it change what the nurse does?
The kidneys are doing their job: low volume triggers ADH (water kept) and, through RAAS, aldosterone (sodium and water kept), so the urine turns scant and concentrated. The action does not change: output below 30 mL/h is reported whatever the cause.
Key terms
fluid volume deficit (FVD)hypovolemiadehydration fluid volume excess (FVE)hypervolemiaoverhydration third spacingedemaascites orthostatic hypotensionhemoconcentrationspecific gravity serum osmolalitydaily weightoral rehydration
Objective W9·6 · Topic W9·T9 · Ch 41

Five volume words, sorted by what was lost or gained top-weighted topic

Fluid problems come in two kinds: volume (too little or too much) and distribution (a normal amount in the wrong place). There are five words to keep apart: two deficits, two excesses and one shift. Within each pair, the deciding feature is whether sodium moved in step with the water.

Which kind is it? Read the sodium first, then the cause.
ConditionWhat changedSerum NaTypical causesHook
Hypovolemia (FVD)Water and electrolytes lost together, in near-normal proportionNormalHemorrhage, vomiting, diarrhea, GI suction, burns, heavy sweatingLost salt water
DehydrationWater lost out of proportion to soluteHigh; serum osmolality upNo access to fluids, a patient who cannot sense or ask for a drink, fever, burns, hyperventilationLost water, kept salt
Hypervolemia (FVE)Water and sodium retained together in the ECFUsually normalHeart failure, renal failure, excess sodium, too much IV fluidKept salt water
OverhydrationWater gained in excess of sodium (water intoxication)LowMore water taken in than the kidneys can clear (Part 3, Potassium and sodium)Gained water, not salt
Third-space shiftFluid moves out of the vessels into a space where the body cannot use itVariesBurns, bowel obstruction, liver or heart failure, crush injury, surgery, peritonitisNot lost, not available
Not one of these: edemaA location, not a volume: excess fluid in the interstitium. It comes with FVE and with third spacing, so it cannot tell you which.
Not one of these: interstitial-to-plasma shiftThe body's rescue in FVD, not a disorder of its own (Part 1, Push, pull and albumin).

Taylor files both deficits under “fluid volume deficit” and does not define dehydration separately. The deck and ATI treat dehydration and hypovolemia as two disorders, so a stem that pairs “dehydration” with a high sodium means the water-only loss.

Calling every deficit “dehydration” A patient with a GI bleed and Na 140, and an 88-year-old who stopped drinking and is confused with Na 152: most people call both dehydration, because everyday speech uses the word for any fluid loss. The bleed is hypovolemia and needs salt water replaced. The older adult is dehydration and needs water replaced slowly (Part 5 has the bags). Read the sodium: a same-proportion loss leaves it normal, a water-only loss pushes it up.
Both ends of life tip easily, in different directions Infants and older adults both slide into FVD easily, for the reasons in Part 1 (Two compartments, its age table); infants also have more surface area for their size and cannot fetch or ask for a drink. The twist is at the older end: a heart and kidneys with less reserve tip into FVE just as readily when fluids are pushed.
Objective W9·2 · Topic W9·T2 · Ch 41

Third spacing and edema: fluid in the wrong place

Edema is excess ECF in the interstitium. Fluid leaves a capillary when the hydrostatic push out beats the oncotic pull back in (Part 1, Push, pull and albumin). There are four ways the balance tips.

MechanismWhat happensExamples
More push: ↑ hydrostatic pressureBlood backs up or volume rises, so more fluid is forced out of the capillaryHeart failure, renal failure, excess IV fluid
Less pull: ↓ oncotic pressureToo little albumin to hold water in the vesselsBurns, liver disease, malnutrition
Leaky walls: ↑ capillary permeabilityProtein and fluid escape together, so the pull is lost as wellTrauma, burns, crush injury
Sodium retentionThe kidneys keep sodium, water follows it, and the ECF expands until it spills into the tissuesHeart failure, renal failure

Third spacing. Fluid is trapped in the pleural, peritoneal (ascites) or pericardial space, the joints, the bowel or the interstitium. It has not left the body, but the circulation cannot use it, so the patient shows a deficit: tachycardia, falling BP, low urine output. The causes are low albumin, too much volume (excess IV fluid, kidney failure), high capillary pressure (heart failure), leaky capillaries (trauma, burns) and low serum sodium. The weight does not fall and the loss cannot be measured on the I&O.

Where edema shows. In the lower legs of a patient who walks and the presacral area of a patient in bed. It is not visible until 5–10 lb of fluid has been retained, which is why the scale catches it first. Edema is localized (a thrombophlebitis) or generalized (heart failure, cirrhosis, nephrotic syndrome). The pitting scale is NUR 120 material (Exams 2 and 4); measuring the limb with a tape at the same marked spot each day is more exact than grading it.

“Swollen, so overloaded: slow the IV” The day after a major burn: massive edema, HR 124, urine 15 mL/h. The tempting call is to slow the fluids, because edema usually travels with FVE. Here the capillaries leaked, so the fluid sits in the tissues and the vessels are short. Keep the fluids running as prescribed and report the output. Edema shows where fluid is, not how much the vessels hold.
“Weight unchanged, so volume is fine” Daily weight is the most accurate fluid measure, and that is exactly why this tempts. A patient with a bowel obstruction, HR 118 and scant urine can weigh what she did on admission, because the fluid never left her body. Weight tracks fluid lost from the body, not fluid trapped inside it.
Assignment W10·A1 · Clinical C10·3 · Ch 41

The wet/dry stick figure, head to toe

The assignment is a blank figure: label the signs of hypervolemia (wet) on one side and hypovolemia (dry) on the other. Fill your own in before reading the key; the act of placing each sign on a body part is what makes it stick.

Wet versus dry, labelled Fluid Balance Stick Figure (W10·A1), instructor-assigned
WET · hypervolemia (FVE) runs high and full DRY · hypovolemia (FVD) runs low and empty Confusion, if sodium is diluted Puffy eyelids Engorged neck veins Moist crackles · dyspnea Bounding pulse · BP up Tight rings · swollen fingers Presacral edema in bed · ascites Dependent edema: feet, ankles Restless · mental status change Sunken, soft eyes · no tears (child) Dry mucosa · furrowed tongue · thirst Flat neck veins Poor turgor (test over the sternum) Fast, weak pulse: the earliest sign Orthostatic drop ≥20 / ≥10 mm Hg Weight up over 5% · more urine if kidneys work Hct down (blood diluted) Sudden weight loss · scanty, dark, high-SG urine Hct up (blood concentrated) · BUN up WHOLE BODY
Drawn for this guide. Wet signs on the left, dry signs on the right; the boxes at the foot are the whole-body measures.
AreaWet: hypervolemia (FVE)Dry: hypovolemia (FVD)
BrainConfusion, if the extra water dilutes sodiumRestlessness; change in mental status
Eyes, facePuffy eyelidsSunken, soft eyes (a 10% deficit); pinched face; no tears in a child
Mouth, tongue—Dry where the cheek meets the gum; extra furrows, smaller tongue; cracked lips; thirst
NeckEngorged neck veinsFlat neck veins
LungsMoist crackles, dyspnea, faster breathingUsually clear
PulseBounding (high amplitude)Fast and weak; tachycardia is the earliest sign
Blood pressureUpDown; orthostatic drop of SBP ≥20 or DBP ≥10 mm Hg
Temperature—Often up
SkinTaut, shiny; pitting edemaPoor turgor; dry
HandsTight rings, swollen fingers—
Sacrum, abdomenPresacral edema if in bed; ascites—
Legs, feetDependent edema—
UrineMore, if the kidneys workScanty, dark, high specific gravity (e.g., 1.035)
WeightGain over 5%Sudden loss: 5% in an adult, 10% in an infant; 15% is life-threatening
LabsHct down (diluted)Hct up (concentrated); BUN and creatinine up

Fifteen rows. Most signs split cleanly: wet is full (bounding pulse, distended veins, edema, weight up) and dry is empty (weak pulse, flat veins, weight down). The two that trip people up are the pulse rate and the hematocrit.

The two that trip people up Pulse: the dry pulse is fast but weak; the wet pulse is bounding and can be fast too. A rate of 118 alone does not say wet or dry; amplitude, BP and neck veins do. Hematocrit: it is a ratio of cells to plasma, so it rises when the patient is dry (plasma lost) and falls when the patient is wet (plasma diluted). A falling Hct can also mean bleeding.
Objective W9·4 · Clinical C10·2–C10·9 · Ch 41

Measuring fluid status: what each measure is worth

Assessment rests on four parameters: the history and physical, intake and output, daily weights, and labs. Look hardest at the four risk groups: acute or chronic illness (diabetes, heart failure, renal failure); abnormal losses (vomiting, diarrhea, draining wounds, fistulas); burns, trauma and surgery; and therapies that shift fluid (diuretics, steroids, IV therapy, parenteral nutrition).

Daily weight, the most accurate measure

1 kg ≈ 1 L ≈ 2.2 lb (NUR 120 Exam 4). Weigh at the same time, on the same scale, in similar clothing, best in the morning after voiding and before breakfast. Weight is more accurate than I&O, and in an infant it is the most reliable indicator of fluid balance. A rapid loss of 5% in an adult or 10% in an infant signals FVD; a gain over 5% signals FVE. The lb↔kg arithmetic is in the Dosage Calculation guide.

Intake and output: what counts

RecordIncludesEasy to miss
IntakeOral fluids; IV fluids, including IVPB; tube feedings and flushes (NG, G, J); ice chips at about half their volume; gelatin, popsicles, water iceThe IVPB and the flushes, because they feel like medication
OutputUrine; vomitus; diarrhea; fistula and suction drainage; note heavy sweating and wound drainageLiquid stool and drain output. Diapers are weighed: 1 g = 1 mL

Tell the patient, the family and every caregiver that everything is measured. Normal turnover is about 2,600 mL in and out a day (Part 1, In and out), and the balance is judged over 2–3 days, not one shift. Intake well below output signals a risk of FVD; well above, a risk of FVE. Refresher (Dosage Calculation guide): report the balance with its sign, e.g. −350 mL; assistive personnel may measure, but the nurse interprets.

Urine output and specific gravity are fluid-status measures too; the report threshold, the SG range and how to read SG against the volume are in Part 6 (Normal versus abnormal urine).

Bedside technique, and what fools you

FindingHow to check itWhat fools you
PulseRate and amplitudeThe rate alone (the exceptions above)
Orthostatic BPLying, sitting, then standing; a sustained drop of SBP ≥20 or DBP ≥10 mm Hg is postural hypotensionA single seated reading misses it
Skin turgorOver the sternum, inner thigh or forehead; the abdomen or medial thigh in a childNormally reduced with age; severe malnutrition depresses it in infants without any fluid loss
TongueOne lengthwise furrow is normal; more furrows and a smaller tongue mean FVDNothing: tongue turgor does not change with age
MouthDryness where the cheek meets the gumMouth-breathing and dry air dry the mouth too
Tears, salivaAbsent in a child means FVDBoth decrease normally with age

NUR 120 taught the back of the hand as the turgor site. For fluid status, use the sternum, inner thigh or forehead instead: hand skin loses elasticity with age, so it tents in older adults who are not dry. In an older adult, confirm with the tongue, the mucosa and an orthostatic BP.

Labs

LabDry (FVD)Wet (FVE)Note
HematocritUpDownHemoconcentration vs dilution; also down after bleeding or hemolysis (Exam 3)
BUNUp—Also up in renal impairment, heart failure, burns, DKA
CreatinineUp—More kidney-specific than BUN
BUN:creatinineOver 20:1—Low blood flow to the kidneys; both rising together in proportion points to kidney damage instead
Serum osmolalityUp in dehydrationDown in overhydration285–295 mOsm/kg; the lab most used to judge fluid status. It tracks the sodium, so it stays normal in a same-proportion loss or gain
Serum sodiumUp in dehydrationDown in overhydrationNormal in pure hypovolemia or hypervolemia (Part 3, Potassium and sodium)

Taylor uses about 280 mOsm as the reference point for plasma.

A high hematocrit read as “too many red cells” After three days of vomiting and diarrhea the Hct is 54%. Most people read it as a cell count and reach for polycythemia. The cells did not multiply; the plasma shrank around them. Rehydrate and expect it to fall. A ratio moves when either side changes: before blaming the numerator, check the denominator.
Assignments W10·A3–A5 · Ch 41

Three patients, three shapes

The unit's three video cases are the shapes scenario items come in: an excess with electrolytes piling up, an excess from a failing pump, and a deficit in a child.

ShapeWhat drives itWhat you watchThe trap inside it
Chronic renal failure: FVEThe kidneys cannot excrete water, sodium, potassium, magnesium or phosphate; calcium falls as phosphate risesWeight, I&O, lungs; potassium (Part 3); low output with low, fixed SGA fluid bolus for low output overloads a kidney that cannot respond; question lactated Ringer's, which contains K (Part 5)
Heart failure: FVEA weak pump raises hydrostatic pressure, and the kidneys retain sodium and waterDaily weight, crackles, neck veins, edema, I&O“Encourage fluids” is wrong here; the HF picture itself is NUR 120 Exam 4 material
Pediatric dehydration: FVDMore ECF and surface area, no way to fetch a drink; vomiting and diarrheaDaily weight; tears, eyes, urine output; sodium, because hypernatremia can leave permanent brain damage in a childA 10% loss in an infant matches a 5% loss in an adult; oral rehydration comes first if the child can drink
Same age group, opposite fluid orders Older adults need fluids offered, at least 1,500 mL a day at regular intervals, because their thirst is weak. An older adult with heart failure or renal failure may need the opposite: a restriction. Read the diagnosis before you push fluids.
Objective W9·7 · Topical TO-FE·3 · Ch 41

Problems, outcomes and interventions

There are three NANDA labels: Excess fluid volume, Deficient fluid volume and Risk for deficient fluid volume. Items may instead use the three newer problem labels below, each with its own cues.

Problem labelCues that point to it
Fluid overloadTight wedding ring, clothes that no longer fit, dyspnea on exertion, pitting edema, taut shiny skin, adventitious breath sounds, BP up, a 10-lb (4.5-kg) gain in a month
Fluid imbalanceMental status change, temperature and pulse up, BP down, dry mucosa, cracked lips, furrowed tongue, poor turgor, scanty dark urine, a sudden 5-lb (2–3 kg) loss
Impaired fluid intakeLimits drinks to avoid trips to the bathroom; SG 1.035; intake below 1,500 mL a day

Exam 3's red rule still applies: no medical diagnosis and no symptom after R/T. The three expected outcomes: intake and output balanced at about 2,500 mL a day, averaged over 3 days (a round goal near the ~2,600 mL turnover); urine SG within 1.010–1.025; and the patient practises self-care that keeps the balance (for example, losing 4 lb a month after cutting sodium).

Nursing care, dry versus wet

One table for both problems, in the order you work: assess, act, teach, delegate, evaluate. Most rows are mirror images. The how-to for fluids, diet and diuretics is in the table after it.

StepDry: deficient fluid volume (hypovolemia)Wet: excess fluid volume (hypervolemia)
Assess and monitorHeart rate first (the earliest sign), then orthostatic BP and pulse: lying, sitting, standing. LOC: restlessness comes early. Mucosa and turgor. Hourly urine output when the deficit is acute; report under 30 mL/h (Part 6). SG, Hct, BUN, sodium.BP and pulse amplitude. Lung sounds, RR and SpO₂ at every assessment: crackles mean the fluid has reached the lungs. Neck veins; edema at the ankles, or the sacrum in bed. LOC, if sodium is diluted. Hct, sodium.
Monitor, bothStrict I&O and a daily weight by the technique above. A change of 1 kg is 1 L.
Position, safety, oxygenFall precautions for orthostasis: bed low, call light and a drink within reach; the patient dangles at the bedside, rises slowly and walks with assistance.Semi- or high-Fowler's for dyspnea, so the lungs can expand; oxygen as ordered.
Fluids and dietOral first if the patient can drink (Oral rehydration, below); otherwise isotonic IV fluid as ordered (Part 5). While replacing, watch for overload: lungs, neck veins, output. A water-only dehydration is corrected slowly.Fluid and sodium restriction as ordered (Restrict fluids and Diet, below). IV fluids and flushes count against the allowance.
MedicationsTreat the cause as ordered. A diuretic can be the cause: report dizziness, a falling BP or low output in a patient taking one.Diuretics as ordered, then weight, I&O and potassium, which loop diuretics waste (Diuretics, below).
Skin and mouthFrequent mouth care; lubricate cracked lips; recheck turgor and mucosa.Stretched skin breaks down easily: turn at least every 2 hours, elevate swollen limbs, protect taut skin, and avoid tape and tight clothing. Thirst on a restriction: Restrict fluids, below.
TeachRise in stages. Drink to a goal tied to the day's routine. Avoid casual laxatives, enemas and “water pills” (below).Weigh every morning at home and report a rapid gain. Keep to the fluid and sodium limits, taught to whoever cooks.
DelegateAssistive personnel may measure and record I&O, weigh, take routine vital signs on a stable patient, offer fluids within the order, help the patient walk and turn, and report to the RN (dizziness, low output, shortness of breath). The RN keeps assessment (lung sounds, interpreting orthostatic readings), IV fluids, interpreting the I&O and weights, teaching and evaluation.
EvaluateHeart rate down, no orthostatic drop, output 30 mL/h or more, SG back in range, LOC clear, weight back up.Weight down, lungs clearing, SpO₂ up, less edema, intake and output nearer even.
Age changes the actionInfant: the daily weight is the best indicator; weigh diapers (1 g = 1 mL); oral rehydration by syringe (below). Older adult: weak thirst, so offer fluids at set times; turgor misleads, so use the tongue, mucosa and orthostatic readings; orthostasis makes a fall likelier.Older adult: a heart and kidneys with little reserve, so IV replacement can tip a dry patient wet. Listen to the lungs often and report a wet cough, dyspnea or new edema. Child: infusions by pump (Part 5).

Third spacing. Nurse it as a deficit in the vessels: heart rate, BP, LOC, hourly output and fluids as ordered. Weight and I&O miss the loss (above), so they cannot show improvement either. When the cause settles and the fluid returns to the vessels, the risk flips to overload, so keep listening to the lungs.

“She walks fine at home” An 81-year-old after two days of diarrhea, with a 24 mm Hg drop on standing, sat up steadily and asks to walk to the bathroom alone. Most people let her, because encouraging independence is usually right, and that is what makes the habit dangerous: the drop comes as she stands, not while she sits. Dangle, stand in stages, walk beside her. A dry patient becomes a fall risk the moment she stands.
How to carry out five of the actions above. IV fluids and blood are Part 5.
InterventionHow
Increase fluidsSplit the 24-hour order into shift totals, heaviest by day (e.g., 1,200 day, 900 evening, 300 night for 2,400 mL), so the night is not spent voiding. Offer variety at the temperature the patient likes; gelatin and popsicles for children; keep a drink within reach; count tube flushes.
Restrict fluidsSet shift totals so one shift cannot use up the allowance. Use small cups; offer fluids every 1–2 hours between meals; avoid dry, salty or sweet foods; keep other drinks out of sight. For thirst: frequent mouth care, lip lubricant, sugar-free gum or hard candy.
Oral rehydrationFirst-line for anyone who can drink, at any age, if renal function is adequate: an electrolyte-and-glucose solution, with I&O recorded hourly or per protocol and SG watched. Child: check the swallow; 5 mL every 5–10 minutes by syringe into the side of the mouth, more as tolerated; 50 mL/kg over at least 4 hours for mild dehydration, 100 mL/kg for moderate; back to a normal diet once rehydrated and not vomiting.
DietFVD: high-water foods such as citrus, melons and celery. FVE: limit sodium. Give a written list and use teach-back with whoever cooks.
DiureticsRisk of FVD and electrolyte loss, especially potassium; monitor I&O, daily weight and electrolytes (Exam 1 drug table; Part 3, Potassium and sodium).

Teach the patient to report four things: a rapid weight gain or loss; swollen fingers, feet or ankles, or puffy eyelids; scanty or very heavy urine; and muscle weakness or a change in skin sensation. Warn against casual use of enemas, laxatives, antacids and over-the-counter “water pills”.

Evaluate with five questions: is intake about equal to output; do the weight and the I&O agree; are the abnormal losses responding; are the signs resolving without new ones caused by the therapy; can the patient manage self-care?

Unlimited ice chips “because they're not really drinking” A thirsty patient on a 1,000 mL restriction, and a kind nurse offers all the ice chips he wants. The instinct to relieve thirst is right, which is what makes the habit dangerous: ice is water, and uncounted water is hidden intake that can undo the restriction. Count it at half its volume, and treat thirst with mouth care. All water counts, whatever form it arrives in.
Check yourself · Part 2
Patient A has had 3+ pitting ankle edema for days, with clear lungs and SpO₂ 96%. Patient B has heart failure, is up 1.2 kg since yesterday, has 1+ edema and new bibasilar crackles. Whom do you assess first?
Patient B. 1.2 kg is about 1.2 L retained in a day, and fluid in the lungs threatens oxygenation. The 3+ grade is the more dramatic number and the less urgent finding: rank by the function threatened.
Skin pinched on the back of the hand stays tented in an 84-year-old who is eating and drinking normally, and in a 28-year-old after two days of diarrhea. What does each finding mean?
Expected with age in the 84-year-old; check the tongue, the mucosa and an orthostatic BP, and retest over the sternum. FVD in the 28-year-old. The same sign means different things because elasticity falls with age.
A patient with heart failure and excess fluid volume is up 1 kg since yesterday. Four tasks: (1) weigh him before breakfast, (2) listen to his lungs, (3) empty and record the urinal, (4) teach his wife the low-sodium list. Which go to assistive personnel?
(1) and (3). Weighing and measuring collect numbers. Listening to the lungs is assessment, and teaching and deciding what the 1 kg means stay with the RN. Handing off the lungs tempts because the aide is already at the bedside doing vital signs. Assistive personnel collect the numbers; the RN assesses, interprets and teaches.
The order is fluids restricted to 1,000 mL a day. Which instruction is best? (a) “Limit your fluids.” (b) “Your fluids are restricted per protocol.” (c) “Stay under 1 liter a day.” (d) “Use this 240 mL cup: one with breakfast, lunch and dinner and one at bedtime is your whole day, and a cup of ice chips counts as half.”
(d). (a) is vague and (b) is jargon. (c) is correct but leaves him to measure and add, which is why it tempts. Tie the limit to a cup he can see and meals he already eats.
Key terms
electrolytenonelectrolyteion cation · anionmEqICF · ECF aldosteronePTHvitamin D hypo- / hyperkalemiahypo- / hypernatremia hypo- / hypercalcemiahypo- / hypermagnesemia hypo- / hyperphosphatemiahypo- / hyperchloremia tetanyChvostek's signTrousseau's sign DTRsrefeeding
Topic W9·T3 · Objective W9·4 · Ch 41

Seven numbers, seven jobs

An electrolyte breaks apart in water into charged particles, ions. Cations are positive (Na⁺, K⁺, Ca²⁺, Mg²⁺, H⁺); anions are negative (Cl⁻, HCO₃⁻, phosphate). In health total cations equal total anions, and both are counted in milliequivalents (mEq), a unit of chemical activity. A nonelectrolyte stays intact in solution; glucose and urea are the two examples to know.

Deck question: electrolyte or nonelectrolyte? Ch 41 deck, slides 16–17 (embedded question 1)

True or false: molecules in the body's compounds that stay intact are called electrolytes. False. Molecules that stay intact are nonelectrolytes; electrolytes are the ones that separate into ions.

Splits into charged ions: electrolyte. Stays whole (glucose, urea): nonelectrolyte.

Items match each electrolyte to its range and to its job, so learn the range and the job together. Two of the seven are in mg/dL (calcium and phosphorus); the other five are in mEq/L.

The seven electrolytes: course normal ranges, where each mainly lives, and its chief job.
ElectrolyteNormal (course)Lives mainlyChief job (the worksheet's matching phrase in bold)Hook
Sodium, Na⁺135–145 mEq/LECFChief electrolyte of the ECF. Controls and regulates the volume of body fluid; nerve and muscle function.Where sodium goes, water follows.
Potassium, K⁺3.5–5.0 mEq/LICFMajor cation of the ICF. Regulates cell enzyme activity and water content; carries nerve, cardiac, skeletal-muscle and intestinal impulses.The heart electrolyte.
Calcium, Ca²⁺8.6–10.2 mg/dLECF; 99% stored in bone and teethMost abundant electrolyte in the body. Clotting, nerve impulses, muscle contraction, bones and teeth.mg/dL, like phosphorus.
Magnesium, Mg²⁺1.3–2.3 mEq/LICF (the second ICF cation)Neuromuscular function. Carbohydrate and protein metabolism; enzyme reactions.Calcium's partner in nerve and muscle.
Chloride, Cl⁻97–107 mEq/LECFMajor ECF anion. Holds osmotic pressure with sodium; makes stomach acid (HCl).Travels with sodium.
Bicarbonate, HCO₃⁻25–29 mEq/LECFAnion chemical buffer, the body's primary buffer.The serum value; an ABG uses a different range (Part 4, Reading a gas).
Phosphate (phosphorus)2.5–4.5 mg/dLICFMajor ICF anion and buffer. ATP and energy; bones; muscle and red-cell function.Moves opposite to calcium.

Taylor gives calcium as 8.6–10.5 mg/dL (ionized 4.5–5.6), and its imbalance table uses Mg above 2.1 and Cl below 98 or above 108. ATI uses Na 136–145, Ca 9–10.5 and Mg 1.3–2.1. Use the course numbers.

Inside or outside. Four live mainly in the ECF: sodium, chloride, calcium and bicarbonate. Three live mainly in the ICF: potassium, phosphate and magnesium. That is why a sodium problem shows up as water moving into or out of cells (Part 1, Tonicity).

Sodium for “most abundant” Sodium is the most-picked answer for both “most abundant electrolyte in the body” and “major cation of the ICF”, because it is the electrolyte everyone has heard of. Sodium is chief of the ECF. Calcium is most abundant in the body, because of bone. Potassium is the major cation inside cells. Calcium also works in nerves and muscles, but it must take “most abundant”, which leaves “neuromuscular function” for magnesium. Read the scope word (body, ECF or ICF) before you match an electrolyte to a superlative.
Topic W9·T4 · Ch 41

Where each comes from, how it is lost, and what controls it

Most of the seven arrive in food and leave through the kidneys, the gut or the skin. The food column is what you name when you teach.

Sources, losses and regulation.
ElectrolyteFood sourcesLost through, or blocked byControlled by
Na⁺Table salt, processed foods, preservatives: canned soups and vegetables, lunch meats, processed cheese, salty snacks.GI tract, kidneys, skin (sweat).Aldosterone keeps it (RAAS); kidneys.
K⁺Bananas, citrus, apricots, melons, raisins; broccoli, potatoes, lima beans; dried peas and beans, whole grains, milk, meat.Kidneys (the main exit), stool, sweat, vomit.Aldosterone excretes it. Shifts between ICF and ECF.
Ca²⁺Dairy, dried beans, green leafy vegetables, small fish eaten with the bones.Absorbed only with vitamin D and stomach acid.PTH raises it; calcitonin lowers it.
Mg²⁺Leafy greens, nuts, seafood, whole grains, dried peas and beans, cocoa.Urine (loop diuretics), diarrhea, NG suction.Kidneys; PTH.
Cl⁻Salt, processed foods.Vomiting and gastric suction (it leaves as HCl), with sodium.Follows sodium; aldosterone.
HCO₃⁻None to teach. Antacids and sodium bicarbonate add it.Diarrhea, diuretics.Kidneys.
PhosphateMeat, poultry, eggs, milk, bread, cereal.Absorption blocked by antacids containing calcium, magnesium or aluminum.Kidneys, PTH, vitamin D.
Three control rules that answer most “why” questions
  1. Aldosterone keeps sodium (and water) and dumps potassium (Part 1, Holding the balance), so too little aldosterone is a cause of high K.
  2. The kidneys cannot conserve potassium. A patient who stops eating, vomits or takes a loop diuretic keeps losing it.
  3. Calcium and phosphate move in opposite directions. When the kidneys fail, phosphate climbs and calcium falls.
Teaching a potassium-rich diet “Eat a potassium-rich diet” is jargon and “eat more fruit” is vague. “A banana with breakfast, orange juice instead of soda at lunch, a baked potato with dinner,” with a written list and teach-back with whoever cooks, is an instruction. Oral potassium is diluted and taken right after breakfast and right after dinner, which is clearer than “BID” and is not the same as every 12 hours.
The salt substitute A patient told to cut salt reaches for a salt substitute. The advice sounds heart-healthy and usually is, which is why the swap tempts. Salt substitutes are potassium chloride. For a patient with renal failure, a potassium-sparing diuretic or a high K, the substitute is the danger. Ask what replaces the thing you told the patient to stop.
Objective W9·3 · Ch 41

The imbalance grid: four patterns before the details top-weighted topic

Electrolyte Imbalances Worksheet: build the grid Electrolyte Imbalances Worksheet (instructor-posted), the unit's assigned electrolyte worksheet

The worksheet is a blank grid of ten imbalances, low and high for potassium, sodium, calcium, phosphorus and magnesium. Each row prints the cut-off and leaves room for risk factors, manifestations and interventions. The tables in the next two sections are that grid filled in, on the worksheet's own cut-offs. Fill a blank copy from memory first, then check it against them.

ElectrolyteHypo- (below)Hyper- (above)
Potassium3.5 mEq/L5.0 mEq/L
Sodium135 mEq/L145 mEq/L
Calcium8.6 mg/dL10.2 mg/dL
Phosphorus2.5 mg/dL4.5 mg/dL
Magnesium1.3 mEq/L2.3 mEq/L

No chloride row. Learn the grid as patterns first; the details hang on them.

POTASSIUM high or low, same danger the heart muscle weakness, paresthesias dysrhythmias, up to arrest the history says which way SODIUM water follows sodium Na high cell shrinks normal Na low cell swells the brain confusion, restlessness, seizures either direction CALCIUM · MAGNESIUM direction decides the picture LOW normal HIGH twitchy tingling, cramps tetany, seizures brisk reflexes sluggish lethargy slow breathing (Mg) weak or absent reflexes low chloride is twitchy too
Three of the four patterns on one page. Phosphorus has no tidy picture of its own.

The four patterns.

  1. Potassium, high or low, threatens the heart. Both directions cause muscle weakness, paresthesias and dysrhythmias. The history tells them apart: renal failure or a KCl infusion points high; vomiting, suction or a loop diuretic points low.
  2. Sodium, high or low, threatens the brain. Low sodium lets water into cells, which swell; high sodium pulls water out, and they shrink.
  3. Low calcium and low magnesium are twitchy: tingling, cramps, tetany, hyperactive reflexes, seizures.
  4. High calcium and high magnesium are sluggish: lethargy, weak or absent reflexes; high magnesium also slows the breathing.
Objective W9·3 · W9·6 · Ch 41

Potassium and sodium: the heart and the brain top-weighted topic

Potassium

ImbalanceRisk factorsManifestationsInterventions
Hypokalemia
K <3.5
Vomiting, gastric (NG) suction, diarrhea, loop and thiazide diuretics, alkalosis, poor intake.Skeletal muscle first: weakness, leg cramps, fatigue, paresthesias. Dysrhythmias. Raises the risk of digoxin toxicity.K-rich foods. Oral K diluted, after meals. IV K only diluted, by pump (Part 5, IV potassium). Confirm urine output before replacing. Monitor K on diuretics.
Hyperkalemia
K >5.0
Renal failure (the commonest), too little aldosterone, KCl and salt substitutes, potassium-sparing diuretics, ACE inhibitors, NSAIDs, heparin. Often present in metabolic acidosis.Muscle weakness, paresthesias, paralysis. Cardiac irregularities, up to arrest.Stop every K source; question any K-containing IV, such as LR, and notify. Low-K diet. As ordered: calcium gluconate to protect the heart (it does not lower K), insulin with dextrose (watch the glucose), a K-binding resin, dialysis.

Why potassium is the dangerous one. Its range is narrow, only 1.5 mEq/L from floor to ceiling, so a small move is a big one. It carries the heart's impulses. The kidneys cannot hold on to it when intake stops, yet they are its main exit, so when they fail it piles up. That is why IV potassium is never given as a bolus: a bolus raises the serum level suddenly and causes a fatal dysrhythmia. Never IV push (NUR 110 Exam 1). How it is diluted, run and capped: Part 5, IV potassium.

“Redraw it; it's probably hemolyzed” A routine draw reads K 6.4 in a patient taking spironolactone and lisinopril. Redrawing in the morning is the most-picked plan, and the reasoning is usually right: hemolysis is a common cause of a falsely high K. That is what makes the habit dangerous when two potassium-raising drugs are on the MAR. Assess, check the rhythm and notify now; redraw as well, not instead. Treat a dangerous value as real until it is proven false, especially when the history explains it.

Sodium

Deck question: a sodium deficit from losing sodium or gaining water Ch 41 deck, slides 33–34 (embedded question 4)

Which imbalance is a sodium deficit in the ECF caused by a loss of sodium or a gain of water? Hyponatremia. Hypernatremia is a sodium surplus; hypokalemia and hyperkalemia are a potassium deficit and excess.

Low sodium is a ratio: too little salt, or too much water diluting it.

ImbalanceRisk factorsManifestationsInterventions
Hyponatremia
Na <135
(severe <120)
Sodium loss: vomiting, diarrhea, fistulas, sweating, diuretics (thiazides are the commonest drug cause). Water gain: excess water, too much hypotonic IV fluid, heart failure, cirrhosis.Water moves into cells, which swell. Lethargy and confusion come first; headache, cramps, weakness. Below 120: twitching, hyperreflexia, seizures, coma.Treat the volume problem behind it: fluid restriction when water is diluting it; sodium and fluid (NS) when both were lost. Raise it slowly.
Hypernatremia
Na >145
Water loss or sodium excess: fluid deprivation; anyone who cannot sense, reach or ask for water (infants, confused or older adults); diarrhea; burns; hyperventilation; prolonged suction; rapid sodium-containing IV.Cells shrink and the brain is hit hardest: restlessness, agitation, disorientation, hallucinations, seizures. Red, swollen tongue; dry, sticky membranes. Permanent brain damage, especially in children.Hypotonic IV fluid (0.45% NaCl) lowers it without dropping it too fast. Avoid table salt and processed foods; watch sodium-containing IVs. Offer water on a schedule to anyone who cannot ask; mouth care.
“Low sodium, so give sodium” The most-picked plan for any low sodium, because the name seems to say what is missing. Usually the water is the problem. Na 128 in heart failure, with edema, crackles and a rising weight, needs a fluid restriction; Na 128 after three days of vomiting, with dry mucosa and an orthostatic drop, needs sodium and volume. Edema or low BP tells you the volume, not the sodium. Decide wet or dry first, then read the sodium.
Objective W9·3 · W9·6 · Ch 41

Calcium, magnesium and phosphorus: the nerve-and-muscle minerals top-weighted topic

Calcium

ImbalanceRisk factorsManifestationsInterventions
Hypocalcemia
Ca <8.6 mg/dL
Low intake; poor absorption (low vitamin D); low PTH, most often after thyroid or neck surgery; renal failure; low albumin; many transfusions; loop diuretics.Numbness and tingling of the fingers, toes and around the mouth; cramps; tetany; seizures; laryngeal spasm; dysrhythmias. Chvostek's sign (tap the face just in front of the ear: that side twitches) and Trousseau's sign (a BP cuff held above systolic: the hand goes into spasm).Calcium and vitamin D foods. IV calcium gluconate as ordered. Check Chvostek and Trousseau.
Hypercalcemia
Ca >10.2 mg/dL
Cancer and hyperparathyroidism (the two commonest); prolonged bed rest; thiazides; too much vitamin D.GI first: nausea, vomiting, constipation. Bone pain; excessive urination and thirst; confusion, lethargy, slurred speech; weak reflexes; kidney stones.NS IV, then a loop diuretic, as ordered. Mobilize, with weight-bearing. Fluids; strain the urine.
The day after thyroid or neck surgery Tingling around the mouth or in the fingertips after thyroid or neck surgery is low calcium until proven otherwise, because that surgery is the commonest cause of low PTH. Check the calcium, listen for wheezing or noisy breathing (laryngeal spasm), and report. A new tingle in a high-risk patient is a cue, not a comfort issue.

Magnesium

ImbalanceRisk factorsManifestationsInterventions
Hypomagnesemia
Mg <1.3
NG suction, diarrhea, chronic alcohol use, loop diuretics, tube feeds or PN, burns, sepsis.Weakness, tremors, tetany, seizures, hyperactive reflexes, dysrhythmias, mental-status change. The same Chvostek and Trousseau signs.Mg foods. IV magnesium sulfate is high-alert (second-nurse check); during it, check reflexes and respiratory rate. It is working when restlessness, tremors and seizures settle. Low Mg, low K and low Ca travel together: correct them together.
Hypermagnesemia
Mg >2.3
Renal failure (the commonest); Mg-containing antacids and laxatives (milk of magnesia).Flushing, nausea, weakness, lethargy; hypoactive or absent reflexes; respiratory depression; bradycardia, hypotension, arrest.Stop Mg products. Calcium gluconate (the antidote) as ordered; dialysis.

Phosphorus, and chloride in one line

ImbalanceRisk factorsManifestationsInterventions
Hypophosphatemia
<2.5 mg/dL
Feeding a malnourished patient (refeeding); alcohol withdrawal; DKA; hyperventilation; malabsorption; diuretics. Antacids with Ca, Mg or aluminum block absorption.Weakness, paresthesias, slurred speech, trouble swallowing, weak respiratory effort, confusion, seizures, coma.Replace as ordered; phosphate foods; refeed slowly.
Hyperphosphatemia
>4.5 mg/dL
Kidneys not excreting it (renal failure); low PTH.Hyperreflexia, anorexia, weakness, dysrhythmias; and, because calcium falls as phosphate rises, low-calcium signs.Phosphate binders with meals; limit dairy, meat and cola; dialysis.

Chloride. It follows sodium. Low chloride (vomiting, gastric suction, diuretics, metabolic alkalosis) is twitchy like low calcium; high chloride travels with metabolic acidosis and high sodium. Care follows the partner problem: the sodium row, or the acid–base imbalance (Part 4).

“Feeding will fix the weakness” A malnourished patient starts tube feeding and two days later is weaker and breathing shallowly. Blaming the malnutrition is the most-picked answer, because feeding usually does cure weakness. But feeding a malnourished patient is itself a cause of low phosphorus, and low phosphorus weakens respiratory effort. Check the level and refeed slowly. When a treatment starts and a new symptom follows, ask whether the treatment caused it.
Objective W9·6 · Ch 41

Reading a patient: which electrolyte, and which one first

Read the deciding sign, then check the history for the cause.

Sign to electrolyte: the reverse lookup.
The picture in the stemThinkDeciding feature
Leg cramps and weakness after days of furosemideLow KSkeletal muscle first, plus a K-wasting drug.
Weakness and rhythm changes in renal failureHigh KSame symptoms as low K; the kidneys decide.
Confusion and headache in a patient drinking litres of waterLow NaBrain signs plus water gain.
Agitation, red swollen tongue, an older adult who cannot reach waterHigh NaBrain signs plus water loss.
Tingling around the mouth, a facial twitch when the cheek is tappedLow Ca (or low Mg)Twitchy, with Chvostek's sign.
Constipation, thirst, passing a lot of urine, bone painHigh CaGI first; cancer or bed rest in the history.
Absent knee jerks and RR 10 on milk of magnesiaHigh MgSluggish, plus an Mg source and weak kidneys.
Shallow breathing after tube feeding is startedLow phosphorusRefeeding.
Not one of these: tingling after vomiting with a normal CaMetabolic alkalosisAn acid–base picture, not a grid row (Part 4, The four imbalances).

When two values are abnormal, rank by the function each value threatens, not by how many points it sits outside its range. The order is the ABCDE framework from Exam 3, applied to electrolytes. Four tiers, in order:

  1. Airway and breathing. Low calcium with laryngeal spasm; high magnesium with a falling RR or lost reflexes; low phosphorus or a potassium below 2.5 with weak respiratory effort.
  2. Circulation: heart rhythm. Any potassium out of range with weakness or rhythm changes, or with a cause that will keep pushing it (renal failure, a KCl infusion running). Potassium is the commonest electrolyte answer to “who first”, because a breathing problem is rarer.
  3. Disability: the brain. Sodium with confusion or seizures, in either direction, or below 120; tetany or seizures from low calcium or magnesium.
  4. The rest. An abnormal value in an alert, stable patient whose cause is already being treated.
The bigger number is the wrong answer Na 128 (alert, mild nausea) or K 2.9 (on furosemide, legs weak)? Sodium is the most-picked answer: it sits 7 points below its range and potassium only 0.6. But potassium's range is only 1.5 wide; below 3 it brings weakness and dysrhythmias, and below 2.5 it can paralyse the breathing muscles. Rank by the organ a value threatens, not by its distance from normal.
What first with a high potassium K 6.2, new weakness, and a KCl infusion running. Every option may be correct; the order is the item. Stop the potassium (the cause is in your hands), assess and put the patient on a monitor, notify, then redraw and document. Calling first, or redrawing first, leaves the cause running while you wait.
Objective W9·6 · W9·7 · Ch 41

What the nurse does, across every imbalance top-weighted topic

Each grid row holds its own treatment; the care that repeats across rows lives here once, in nursing-process order.

Nursing care for electrolyte imbalances: five steps, twelve actions.
StepWhat you doWhich imbalances
1 Assess and monitorHeart: apical pulse and rhythm; cardiac monitor as ordered.Any K out of range; Ca and Mg, either direction.
Brain: neuro checks (level of consciousness, orientation, behaviour), more often if they change.Na, either direction; high Ca; low phosphorus.
Breathing and reflexes: RR and depth, DTRs; listen for stridor or wheeze.High Mg; low Ca (laryngeal spasm); low phosphorus; K below 2.5.
Volume: I&O and daily weight (Part 2); urine output before any K.Na; renal failure; diuretics; high Ca on IV NS.
Labs: recheck the level after each replacement.All; check Mg when a low K will not correct.
2 ActFall precautions: bed low, call light in reach, help to walk.The weak and the sluggish: low K, high Ca, high Mg; low phosphorus; confused Na.
Seizure precautions: padded rails, suction and oxygen at the bedside.Na below 120 or with neuro change; low Ca; low Mg.
Airway ready: suction and oxygen near; for high Mg, calcium gluconate available.Low Ca (laryngeal spasm); high Mg (respiratory depression).
Medication safety: potassium is never pushed (Part 5, IV potassium); know the K before digoxin; IV Mg is high-alert; stop every K or Mg source when high.K and Mg.
3 TeachA written food list for whoever cooks; report weakness, cramps, tingling, palpitations or confusion; no salt substitute with high K; no Mg antacids or laxatives with weak kidneys.All (foods: the sources table).
4 DelegateAssistive personnel may weigh, measure I&O, take vital signs, walk the patient and offer fluids within the plan, and report new weakness, numbness or confusion. The RN keeps neuro and reflex checks, the rhythm, the labs, IV electrolytes, teaching and evaluation.All.
5 EvaluateLevel in range on recheck; signs gone (regular rhythm, oriented, normal reflexes, no tetany); no new problem from the treatment: sodium corrected too fast, low glucose after insulin, K overshooting after replacement.All.

The care-plan line. For a patient not yet out of range, the NANDA label is Risk for electrolyte imbalance: risk factors, and no “as evidenced by”. Example: Risk for electrolyte imbalance related to gastric fluid loss through NG suction and loop-diuretic therapy. Measurable outcome: serum K stays 3.5–5.0 mEq/L and Mg 1.3–2.3 mEq/L on daily labs, with a regular apical pulse and no leg cramps, through discharge.

Slowing the magnesium drip During IV magnesium sulfate, the RR falls to 10 and the knee jerks are gone. Most people slow it and recheck in an hour, because slowing usually fixes a rate problem. Here the drug is the cause: stop it, assess, notify, and have calcium gluconate ready. When a drug produces the sign of its own excess, stop it; don't titrate it.
Objective W9·6 · Topic W9·T4 · Ch 41

The links: drugs, acid–base, the kidneys and immobility

Most electrolyte items hide the cause in the medication list or the history. Eight links to know:

What moves the numbers.
The linkWhat movesWhat you do
Loop and thiazide diuretics (furosemide)K falls; loops also lower Mg and Ca. Thiazides are the commonest drug cause of low Na, and can raise Ca.Monitor K and I&O; teach K-rich foods.
Potassium-sparing diuretics (spironolactone), ACE inhibitors (lisinopril), NSAIDs, heparin, KCl, salt substitutesK rises.Question a new K source; report a rising K.
DigoxinLow K raises the risk of digoxin toxicity.Know the K before the dose (NUR 110 Exam 1).
Mg-containing antacids and laxativesMg rises when the kidneys cannot clear it.Avoid them in renal failure.
Vomiting, gastric suctionAcid, K and Cl are lost: metabolic alkalosis, and low K travels with it (Part 4, The four imbalances).Replace K as ordered.
Metabolic acidosisHigh K is often present (the shift: Part 4).Watch the K as the acidosis is treated.
Renal failureK, Mg and phosphate rise; calcium falls. The kidneys are the exit for all three.Stop every source; expect binders and dialysis.
ImmobilityCalcium leaves demineralizing bone: high Ca and calcium kidney stones (NUR 110 Exam 2).Mobilize with weight-bearing; fluids. Stones: Part 7.
Same potassium, two meanings K 3.4 is barely low. In a healthy patient eating normally it is a diet conversation. In a patient on digoxin and furosemide it is a reason to report before the next digoxin dose, because low K raises the risk of toxicity and the diuretic will keep pushing it down. The medication list decides what a borderline value means.
“The water pill did it” A patient on furosemide reads K 5.8, and the note blames the diuretic. Agreeing is the most-picked answer, half-remembering that diuretics upset potassium. Loop diuretics lower K. Look at the rest of the list: lisinopril, ibuprofen, a potassium-sparing diuretic, KCl, failing kidneys. Know which way a drug pushes a value before you blame it for the value.
Check yourself · Part 3
Patient A: Na 151, alert, oriented, thirsty and drinking. Patient B: renal failure, has been taking milk of magnesia, Mg 2.9, RR 10, knee jerks absent, drowsy. Who do you see first?
Patient B. The magnesium is only 0.6 above range, but absent reflexes and an RR of 10 are respiratory depression in progress. The sodium is further out, and the patient is correcting it by drinking.
After two days of NG suction a patient has tingling fingers and tight, hypertonic muscles. Ca is 9.2 mg/dL. A classmate blames hypocalcemia. Does the calcium explain it, and what else could?
No: 9.2 is normal. Suction removes acid, potassium and chloride, producing metabolic alkalosis, which itself causes tingling and hypertonic muscles. Check the K and an ABG (Part 4, The four imbalances).
A patient with metastatic breast cancer has been on bed rest for two weeks. She is constipated, thirsty, passing large amounts of urine, reports aching bones and is newly drowsy. Her glucose is normal. A classmate puts the constipation down to immobility. What one problem explains all five findings, and what do you expect to be ordered?
High calcium. Cancer and prolonged bed rest are both causes, and GI signs, thirst, heavy urination, bone pain and lethargy are its picture. Immobility does slow the bowel, which is why the classmate's answer tempts, but it explains none of the rest. Report and check the calcium; expect NS IV, then a loop diuretic, weight-bearing as allowed, fluids and strained urine. Prefer the one cause that explains every finding to a separate excuse for each.
Which two show hyperactive reflexes: Ca 7.9 mg/dL · Mg 3.1 mEq/L · Ca 11.4 mg/dL · Mg 1.0 mEq/L?
Ca 7.9 and Mg 1.0, the two lows: twitchy. The two highs are sluggish, with weak or absent reflexes. Sort by direction, not by electrolyte.
Key terms
acidbasepH buffercarbonic acid–bicarbonate20 : 1 ratio PaCO₂HCO₃⁻serum bicarbonate hypoventilationhyperventilationKussmaul respirations ketonesH⁺/K⁺ exchangecompensation (the rescue)
Topic W9·T7 · Ch 41

Acids, bases and the 20 : 1 ratio

An acid releases hydrogen ions (H⁺); a base traps them. The more free H⁺, the lower the pH. On the 1–14 scale 7 is neutral, but blood lives in a narrow band: 7.35–7.45, slightly alkaline. Below 7.35 is acidosis, above 7.45 is alkalosis, and below 6.80 or above 7.80 life cannot continue.

Two chemicals carry the whole story. Carbonic acid is the acid the lungs control, by blowing CO₂ off or holding it in, which is why CO₂ acts as an acid. Bicarbonate (HCO₃⁻) is the base, and the kidneys control it. What sets the pH is not the amount of either but their ratio: about 20 parts bicarbonate to 1 part carbonic acid. Every imbalance in this part is that ratio tipping one way; every rescue is the other side tipping to match.

Topic W9·T7 · Ch 41

Three defences, fastest first

Three systems guard the ratio. Learn them in order of speed, because that is how items ask for them.

DefenceHow it worksSpeed
1 · BuffersChemical pairs that take up extra H⁺ or release it. Three systems: the carbonic acid–sodium bicarbonate buffer (the main one); phosphate, which works inside cells, especially the kidney tubules; and protein, meaning plasma proteins and the globin part of hemoglobin. Fast, alongside the lungs.
2 · LungsRising CO₂ stimulates chemoreceptors in the medulla and the carotid and aortic bodies. Breathing becomes faster and deeper, CO₂ is blown off, and the pH rises. Falling CO₂ makes breathing slower and shallower, so CO₂ is kept. Minutes, but only short term.
3 · KidneysIn acidosis they excrete H⁺ and form or keep HCO₃⁻. In alkalosis they keep H⁺ and excrete HCO₃⁻. Hours to days, up to about 3 days. Slow, but the lasting fix.
Deck question: the most important buffer Ch 41 deck, slides 28–29 (embedded Question 3)

Which buffer system matters most? The carbonic acid–sodium bicarbonate system, which buffers as much as 90% of the hydrogen ions in the ECF. Phosphate and protein are real buffers with smaller shares. The fourth option, a “hydrogen buffer system”, is not one of the three.

Across the lifespan. Newborns cannot acidify their urine well. In older adults the lungs and kidneys both work less efficiently, especially with lung disease, so both ends of life have less room for error.

Objective W9·4 · Ch 41

Reading a gas: the refresher

The normals and method are unchanged from NUR 120 Exam 3 (and NUR 110 Exam 3 for the respiratory pair).

ValueNormalWhat it tells you
pH7.35 – 7.45Below 7.35 is acidosis, above 7.45 is alkalosis.
PaCO₂35 – 45 mmHgThe respiratory number, controlled by the lungs, which respond in minutes. CO₂ acts as an acid.
HCO₃⁻22 – 26 mEq/LThe metabolic number, controlled by the kidneys, which take hours to days. HCO₃ acts as a base.
PaO₂80 – 100 mmHgDoes not affect acid–base balance. It is read separately, for oxygenation.
NUR 120's four steps, in the handout's own wording.
StepWhat to do
1Look at the pH. Is it on the acid side, the alkaline side, or normal? <7.35 acid · >7.45 alkaline
2Look at the PaCO₂ — this is the respiratory number. High means acid, low means alkaline.
3Look at the HCO₃ — this is the metabolic number. Low means acid, high means alkaline.
4Which one matches the pH? That one names the imbalance.

Say it as a sentence: “The pH is acid. The CO₂ is acid. They match — so it is respiratory acidosis.” ATI's ROME is the same rule seen through direction: Respiratory Opposite (pH and PaCO₂ move opposite ways), Metabolic Equal (pH and HCO₃ move the same way). If both numbers match an acid pH, both are causing it: a combined acidosis, the one picture the four steps cannot name alone.

Two tests, two numbers Bicarbonate has two normal ranges, and both are right. Serum bicarbonate on an electrolyte panel (BMP), often reported as “CO₂”, is 25–29 mEq/L. ABG HCO₃⁻ is 22–26 mEq/L. So a value of 27 is normal on a BMP and high on an ABG. Before you judge a bicarbonate, name the test it came from. Part 3's table (Seven numbers, seven jobs) lists the 25–29.

Drawing one (NUR 110 Exam 3): Allen test before a radial stick; the O₂ setting on the requisition; the specimen on ice and to the lab promptly; pressure 5–10 minutes, longer on anticoagulants.

Topic W9·T8 · TO-FE·2 · Ch 41

The four imbalances

Each name has two halves: which system started it (respiratory means carbonic acid is off; metabolic means bicarbonate is off) and which way the pH went. The respiratory pair is a refresher, one row each; the metabolic pair is new, and the next section gives its causes in depth.

ImbalanceMatching numberCausesSigns
Respiratory acidosispH <7.35
PaCO₂ >45
carbonic acid excess: hypoventilation
Opioid or sedative overdose, anesthesia, COPD, asthma, pneumonia, atelectasis, pulmonary edema, neuromuscular weakness, abdominal distention Drowsiness, mental cloudiness, confusion, dizziness, twitching; chronic: weakness, dull headache
Respiratory alkalosispH >7.45
PaCO₂ <35
carbonic acid deficit: hyperventilation
Anxiety (the most common), pain, fever, hypoxemia, early sepsis, pregnancy, a ventilator set too fast Lightheadedness, tingling, palpitations, tinnitus, sweating, tremor, poor concentration; convulsions if severe
Metabolic acidosispH <7.35
HCO₃⁻ <22
bicarbonate deficit, or acid gained
Diarrhea, DKA, renal failure, starvation, lactic acidosis (shock, cardiac arrest), salicylate (aspirin) toxicity, fistulas Headache, confusion, drowsiness, Kussmaul breathing (deep and rapid), nausea and vomiting, warm flushed skin; hyperkalemia often present
Metabolic alkalosispH >7.45
HCO₃⁻ >26
bicarbonate excess, or acid lost
Vomiting, NG suction, potassium-wasting diuretics, hypokalemia, antacid overuse, steroids Dizziness, tingling fingers and toes, twitching or hypertonic muscles, slow shallow breathing; hypokalemia

Acidosis depresses the brain; alkalosis excites it (NUR 120 Exam 3): drowsy and confused on one side, tingling and twitchy on the other. The tingling and twitching of alkalosis look like low calcium: with a normal calcium after vomiting or suction, think metabolic alkalosis (Part 3, Calcium, magnesium and phosphorus).

Read five gases. Cover the right-hand columns, run the four steps, then check.

pH / PaCO₂ / HCO₃⁻The number that matches the pHName
7.30 / 36 / 17HCO₃⁻ low (acid)Metabolic acidosis
7.52 / 30 / 24PaCO₂ low (alkaline)Respiratory alkalosis
7.28 / 54 / 25PaCO₂ high (acid)Respiratory acidosis
7.49 / 41 / 32HCO₃⁻ high (alkaline)Metabolic alkalosis
7.33 / 35 / 18HCO₃⁻ low; a PaCO₂ of 35 sits on the edge but is still normalMetabolic acidosis
Hyperventilating and panicky is not always anxiety A post-op patient breathing at 34, tingling and frightened, with SpO₂ 86%. Coaching slow breathing “for anxiety” is the most-picked answer, because anxiety is the commonest cause of respiratory alkalosis, and that usually-right reasoning is the trap. Hypoxemia causes the same picture, and rebreathing makes it worse. Rule out hypoxemia before you call it anxiety.

What the nurse does

Work each row left to right. Medications, fluids and O₂ are as ordered.
ImbalanceAssess and monitorActTeach · delegate
Respiratory acidosis Airway, rate and depth, lung sounds, SpO₂; LOC (drowsier means the CO₂ is climbing); repeat ABGs Sit the patient upright; cough, deep breathing, incentive spirometer; fluids to thin secretions; O₂ and bronchodilators. Hold the next opioid or sedative dose and report; reversal agent if ordered. In a chronic CO₂ retainer (COPD), give exactly the O₂ ordered and watch for rising drowsiness. Lower CO₂ slowly. Teach the spirometer and a splinted cough. Assistive personnel (AP) may reposition, prompt the spirometer, and take VS and SpO₂ if stable; LOC, lung sounds, O₂ changes and the gas stay with the RN.
Respiratory alkalosis SpO₂ first; rate, pain, temperature, anxiety; tingling and dizziness; on a ventilator, the set rate Stay with the patient, speak calmly, breathe slowly with them; treat the cause (analgesic, antipyretic, anxiolytic); rebreathe CO₂ (a rebreather mask or paper bag) only once hypoxemia is ruled out; report a ventilator rate set too fast; fall precautions. Teach slow, paced breathing for the next episode. AP may stay with the patient and report; ruling out hypoxemia stays with the RN.
Metabolic acidosis Cardiac rhythm and K⁺; LOC; breathing pattern; glucose in DKA; I&O; an ABG after each 50–100 mEq of sodium bicarbonate Treat the cause: IV fluids and insulin in DKA, fluid replacement for diarrhea, sodium bicarbonate if ordered. Don't slow Kussmaul breathing; it is the rescue. Bed low, call light in reach, fall precautions for confusion. Teach glucose checks and insulin as prescribed in diabetes; report diarrhea that won't stop. AP may record I&O and VS; the rhythm, K⁺ and insulin stay with the nurse.
Metabolic alkalosis Rate and depth (slow, shallow); tingling, twitching, tetany-like spasms; K⁺ and rhythm; I&O including emesis and NG output Reverse the cause: replace fluid, chloride (0.9% NaCl) and K⁺ (Part 5, IV potassium); antiemetic; irrigate an NG tube with normal saline, not water, and suction no more than ordered; no unneeded antacids; fall precautions. Teach: no casual antacid use; potassium foods or supplements with a K-wasting diuretic as prescribed. AP may measure emesis and NG output and report a jump; NG irrigation and IV K⁺ stay with the nurse.

Evaluate four things in every row: the repeat gas moving toward pH 7.35–7.45; mental status back to baseline; breathing at a normal rate and depth (in metabolic acidosis, easing as the acid clears); and K⁺ within 3.5–5.0.

“She came in with a high potassium” DKA, K⁺ 5.9 on arrival and 3.4 two hours into insulin and fluids. Holding the ordered potassium because of the admission value is the most-picked error, because hyperkalemia really does travel with acidosis. But insulin and a rising pH push K⁺ back into cells, and the serum level can fall fast. Correcting an acidosis moves potassium: recheck it and watch the rhythm.
Topic W9·T7 · Ch 41

Metabolic causes: what was lost, what was gained

Every metabolic cause comes down to one of four moves: acid lost, acid gained, base lost or base gained. Name the move and the imbalance follows.

CauseWhat happensResult
Vomiting, NG suctionStomach acid (HCl) leaves; potassium and chloride go with it.Alkalosis
Diarrhea, intestinal fistulaBicarbonate-rich intestinal fluid leaves; chloride is kept.Acidosis
DKA, starvationKetones, acids from fat breakdown, build up.Acidosis
Renal failureThe kidneys can no longer excrete H⁺ or rebuild bicarbonate, and potassium stays in.Acidosis + hyperkalemia
Shock, cardiac arrest; aspirin overdoseLactic acid, or salicylate, is added.Acidosis
Antacid or bicarbonate overuseBase is added.Alkalosis
Loop or thiazide diureticsPotassium and chloride are lost in the urine.Alkalosis
Large volumes of 0.9% NaClNormal saline carries more chloride than plasma; as chloride rises, bicarbonate falls.Acidosis

The GI rule from NUR 120 Exam 3 still settles most items: vomiting loses acid, so alkalosis; diarrhea loses bicarbonate, so acidosis. Sort by what leaves, not by the route.

Potassium rides along

Hydrogen and potassium trade places across the cell membrane. In acidosis, H⁺ moves into cells and K⁺ moves out, so serum K⁺ rises; that is why hyperkalemia so often comes with metabolic acidosis. In alkalosis the trade reverses and serum K⁺ falls. Low potassium also feeds metabolic alkalosis, so the two sustain each other. Insulin pushes K⁺ into cells too. Treat every acid–base patient as a potassium patient, and so as a heart patient (Part 3, Potassium and sodium).

“Acidosis lowers potassium” The most-picked wrong direction, because acid sounds bad and “low” sounds bad, so students pair them. The exchange runs the other way: H⁺ goes into the cell and pushes K⁺ out into the blood. Acid in means K⁺ out; alkalosis and insulin push K⁺ in.
Topic W9·T7 · TO-FE·2 · Ch 41

Compensation: the rescue

When one system tips the ratio, the other system pushes back. NUR 120's rule still holds: only the number that matches the pH names the imbalance; a number reading the other way is the rescue, not the disease. The lungs rescue metabolic problems within minutes. The kidneys rescue respiratory problems over hours to days; in respiratory alkalosis they start after about 6 hours.

Each system rescues the other system's imbalance LUNGS control CO₂, the acid respond in minutes KIDNEYS control HCO₃⁻, the base respond over hours to days rescue rescue a METABOLIC problem Acidosis: deep, rapid (Kussmaul) breaths blow CO₂ off, so PaCO₂ falls Alkalosis: slow, shallow breaths keep CO₂, so PaCO₂ rises a RESPIRATORY problem Acidosis: kidneys keep HCO₃⁻ and excrete H⁺, so HCO₃⁻ rises Alkalosis: kidneys excrete HCO₃⁻, so HCO₃⁻ falls The rescue reads the opposite way to the pH. It pulls the pH back toward normal; it never names the imbalance.
Lungs fix metabolic problems fast; kidneys fix respiratory problems slowly. A rescue number is the body's repair, not a second disease.

Why culprit and rescue move together on paper. High CO₂ means acid but high HCO₃⁻ means base, so a rescue pushes the second number the same direction as the first: both up, or both down. A COPD patient who has retained CO₂ for years also carries a high bicarbonate. That is the kidneys' rescue, not a second disease.

How far this exam goes. The program limits ABG work to non-compensatory acid–base balance, so expect the partner number to be normal in most items. The course deck's ABG patterns, though, print the rescue in: respiratory acidosis with HCO₃⁻ “normal or slightly elevated”, respiratory alkalosis with HCO₃⁻ <22, metabolic acidosis with PaCO₂ <35, metabolic alkalosis with PaCO₂ >45. Meet one of these and step 4 still names it; the extra number is the rescue.

A high PaCO₂ after two days of NG suction pH 7.50, HCO₃⁻ 34, PaCO₂ 48. Adding “respiratory acidosis” as a second problem is the most-picked error, because step 2 says a high PaCO₂ means acid. But the pH is alkaline, and only the bicarbonate agrees: metabolic alkalosis, with slow breathing keeping CO₂ as the rescue. A number that disagrees with the pH cannot name the imbalance.
Ask the instructor How far does compensation go on this exam? The program outline says non-compensatory, the deck prints rescue values, and Taylor goes on to grade compensation as partial or full. Until that is answered, name the imbalance from the number that matches the pH, call the other number the rescue, and don't spend time on partial versus full.
Check yourself · Part 4
Both patients have pH 7.32. A has COPD, PaCO₂ 62, HCO₃⁻ 31, and is chatting at their baseline. B is post-op on PCA morphine, PaCO₂ 48, HCO₃⁻ 24, and drowsy. Whom do you see first?
B. Both have respiratory acidosis, but B's bicarbonate is normal: no rescue yet, so the problem is new and opioid-driven. A's kidneys have had days to catch up. A's bigger PaCO₂ is the tempting wrong pick. (The pair is chosen so the pH matches; you are not being asked to grade compensation.)
HCO₃⁻ 28 mEq/L appears on one patient's morning BMP and on another patient's ABG (pH 7.47, PaCO₂ 40). Interpret each.
BMP: normal (serum 25–29). ABG: high (22–26), and it matches the alkaline pH, so metabolic alkalosis. Name the test before you judge the number.
A patient hyperventilating from anxiety has pH 7.50, PaCO₂ 30 and K⁺ 6.0. Does the alkalosis explain the potassium?
No. Alkalosis pushes K⁺ into cells and lowers the serum level. Look for another cause: renal failure, potassium-sparing drugs or ACE inhibitors, or a sample hemolyzed during the draw.
A patient in DKA breathes deep and fast at 32/min. A classmate wants to coach slower breathing, as for anxiety. Your response?
Don't. Kussmaul breathing is the lungs' rescue, blowing off acid; slowing it would let the pH fall further. Treat the cause as ordered, give IV fluids, and watch the K⁺ and the rhythm.
Key terms
isotonichypotonichypertonic PIVCmidlinePICC nontunneled CVCtunneled CVCimplanted port IVPBdrop factormacrodripmicrodrip infiltrationextravasationphlebitis speed shockair embolism ABORh factorcrossmatchhemolytic reaction
Exam worksheet: review IV sites, placement, fluids Exam worksheet (instructor-posted), the one bold line

The exam worksheet has one bold line, “REVIEW IV SITES/PLACEMENT/FLUIDS!”, and seven open questions follow it. Each one is worked in its own blue box in this part:

#The questionWorked in
1How do you choose the best place on an extremity for an IV?Venous access
2Give an example of a hypertonic, a hypotonic and an isotonic fluid.Solutions
3Infection vs infiltration vs phlebitis vs emboli.Complications
4The important concepts when giving blood.Blood
5The signs of a transfusion reaction.Blood
6How IV potassium is given, its maximum, and why.IV potassium
7The important concepts in caring for a central line.Venous access
Objective W9·5 · Lab L6·2 · Ch 41

Solutions by tonicity: what each bag does to the cells top-weighted topic

Tonicity compares a fluid's particle concentration with plasma's, and water moves toward the side with more particles (Part 1, Tonicity). So the label on the bag tells you which way water will move once it is running: into the cells, out of them, or nowhere.

Exam worksheet question: a hypertonic, hypotonic and isotonic example Exam worksheet, open question 2; NS, 0.45% NaCl and D10W from deck slide 22
TonicityExampleWater and cellsWho gets itWatch for
Isotonic0.9% NaCl (normal saline)No net shift: it stays in the vessels and expands the ECFHypovolemia; the only fluid hung with blood. Not a routine maintenance fluidFluid overload in heart or kidney failure; hypernatremia
IsotonicLactated Ringer'sThe sameHypovolemia, burns, GI lossesIt contains potassium: question it when K is high
Isotonic in the bagD5WActs hypotonic once the dextrose is used (trap below)Hypernatremia: it supplies free waterNot for raised intracranial pressure or for fluid resuscitation
Hypotonic0.45% NaCl (half-normal saline)Out of the vessels into the cells, which swellMaintenance; hypernatremia, lowered gentlyLow BP as the vessels empty; avoid with raised intracranial pressure, burns, trauma
HypertonicD10WOut of the cells into the vessels: cells shrinkPulls fluid into the circulation; supplies glucoseOverload and pulmonary edema in heart or kidney disease; irritates veins, so use a large one

Taylor's hypertonic examples are D5LR and D5 0.9% NaCl (and D5½NS): dextrose added to a saline or LR base makes the bag hypertonic.

Apply the deck's hypertonic question (Part 1, Tonicity) to the bedside: the water a hypertonic bag pulls out of the cells lands in the circulation, so the patient to watch is the one whose heart or kidneys cannot handle more volume. Listen for crackles.

D5W: isotonic in the bag, hypotonic in the body Most people call D5W hypertonic, because “5% dextrose” sounds like added solute. In the bag it is isotonic. Once the body metabolises the dextrose, what is left is free water, which moves into cells the way a hypotonic fluid does. That is why it is kept away from a swelling brain. Classify a fluid by what is in the bag, then ask separately what it becomes once the body uses the sugar.
An IV fluid is a drug Check every fluid order against today's labs and against the heart and kidneys. LR carries potassium, so LR ordered for a patient whose K is 5.6 is a call to the prescriber, not a bag to hang. Normal saline running into a patient with heart failure gets the same scrutiny.
Lab L6·5 · Objective W9·5 · Ch 41

Venous access: choosing the site and the device

Exam worksheet question: the best place on an extremity Exam worksheet, open question 1; the four factors from deck slide 55

Four factors decide the site: how accessible the vein is, what condition it is in, what fluid will run, and for how long. In practice they become seven rules:

  1. Use the nondominant arm, and involve the patient in the choice.
  2. Use the forearm for a short peripheral catheter: it dwells longer, hurts less and is harder to pull out. Hand veins are acceptable short-term.
  3. Start distal and work up: never below a recent site, a flexion area, or painful or damaged skin.
  4. Avoid the antecubital fossa if any other vein is available (trap below).
  5. Never use a limb with a same-side mastectomy with node removal, an AV fistula or dialysis catheter, or burns, infection or trauma.
  6. No leg veins in adults except in an emergency: they risk thrombophlebitis and ulceration.
  7. Hypertonic, irritant, fast or thick fluids, blood included, need a large vein.

Gauge. 20–24 g for most adults; 22–26 g for neonates, children, older adults and anyone with poor veins; blood 20–24 g, or 18–20 g for a rapid transfusion. Children: hand, forearm, or the foot if not yet walking; scalp veins only as a last resort. Older adults' thin veins infiltrate easily, so limit the tourniquet (or use a BP cuff), lower the insertion angle, and watch the rate closely: with less heart and kidney reserve, overload comes sooner, from a bag or a transfusion.

Technique, the testable points. A tourniquet 3–4 inches above the site, with the radial pulse still present. Skin prep with chlorhexidine in alcohol, left to air-dry: no wiping, fanning or blowing, and no touching the site afterwards. Confirm a blood return and a flush without resistance. Transparent dressing, labelled with date, time, site, and catheter type and size. Two attempts per nurse at most, then escalate. Starting an IV is surgical asepsis (Exam 1) and is never delegated to assistive personnel.

The big vein in the elbow crease It is the easiest vein to see and feel, so it is the most-picked site. Every bend of the elbow kinks or dislodges the catheter, and damage there costs you the veins below it and the future PICC site. Pick the vein that will last the whole therapy, not the one easiest to hit.

The six devices

DeviceWhere it goesChosen forKey facts
Short PIVCPeripheral vein, forearm preferredShort therapy, under about 4 daysNo vesicants, irritants or parenteral nutrition (PN)
MidlineUpper-arm vein; tip at or below the axilla5–14 daysNot a central line: the same limits as a PIVC
PICCInserted in an upper-arm vein; tip centralLong term, weeks to monthsA trained RN can insert one; least pneumothorax risk of the central lines; replaced only PRN
Nontunneled CVCInternal jugular, subclavian or femoral veinUnder 14 days; critical, unstable patientsPneumothorax risk at the subclavian
Tunneled CVCJugular or subclavian, run under the skinLong termA Dacron cuff anchors it; fewer infections than nontunneled; no dressing once healed
Implanted portUnder the skin of the upper chestLong termLowest infection rate; accessed with a noncoring needle; nothing visible

PICC veins: the deck lists basilic, median cubital, brachial and cephalic; Taylor lists basilic, brachial (preferred) and cephalic, above the antecubital fossa. Don't key the median cubital on its own.

A central device is considered when therapy will run longer than about 15 days, or the fluid would injure a small vein. Choose the least invasive device, with the smallest diameter and fewest lumens that will do the job.

Right atrium superior vena cava internal jugular vein subclavian vein upper-arm vein PICC in at the upper arm, tip threaded to the SVC Midline tip stops at or below the axilla: NOT central Nontunneled straight in at the jugular or subclavian (or femoral); under 14 days Tunneled exits lower on the chest after running under the skin; Dacron cuff Implanted port under the skin; noncoring needle Every central tip ends here: lower third of the SVC, near the right atrium. X-ray before first use.
Where each device enters, and where every central tip ends. A midline enters the same arm vein as a PICC but stops short of the chest, which is why it is not a central line.
Deck question: what can a central line be used for? Ch 41 deck, slides 53–54 (Question #5)

True: a central venous access device carries IV fluids, medications, blood products and parenteral nutrition, and it also allows hemodynamic monitoring and blood sampling. That range is why it is chosen for the fluids a peripheral vein cannot take, and why its infection risk gets the care in the next box.

Exam worksheet question: caring for a central line Exam worksheet, open question 7

Seven concepts:

  1. X-ray confirms the tip before the first use.
  2. Assess the site, dressing and securement with each infusion and at least daily.
  3. Asepsis at every access: scrub the hub for 5–15 seconds with chlorhexidine-alcohol and let it dry.
  4. Flush and aspirate for a blood return before and after each use. Lock a CVAD with heparin 10 units/mL or saline; a PIVC is locked with saline.
  5. Change the dressing on schedule (table below, How often things are changed), and at once if damp, loose or soiled.
  6. Clamp the catheter whenever the system is opened, and never let a bag run dry: both let air in.
  7. Not changed on a schedule: review the need daily and remove it once it is no longer needed or infection is suspected. Never submerge it; cover it to shower.

Watch for the five complications: thrombosis, bloodstream infection (CLABSI), air embolism, phlebitis, occlusion. An RN may remove a PICC or midline; most other CVADs are removed by advanced practitioners.

Lab L6·4–L6·10 · Ch 41

Running an infusion: priming, labelling, piggybacks, pumps and rates

Hanging a bag uses the same rights and checks as a medication: compare it with the MAR, check the expiry date, scan it, and inspect the fluid for clarity and particles. Priming, in five steps:

  1. Close the roller clamp and spike the bag without touching the port.
  2. Fill the drip chamber at least halfway.
  3. Prime the whole tubing until every bubble is gone, then clamp.
  4. Label the bag (name, solution, additives, date, time) and add a time tape; label the tubing with the date of its next change.
  5. Recheck the site and rate 30 minutes after starting, then check the infusion at least hourly.
IVPB 50–250 mL Primary time tape PUMP to the patient Secondary hangs HIGHER, so it runs first. When it empties the primary resumes: reset its rate. Label the bag; add a time tape. Tubing label: the date of its next change. Drip chamber at least half full. Prime until every bubble is gone. Back-prime the IVPB only if the fluids are compatible. Roller clamp regulates; slide clamp stops. Pump runs in mL/h. On gravity, count the drops: gtt/min.
A primary line with a piggyback. Height decides which bag runs: the higher one wins.

Pumps. They are standard care, and a smart pump does not take away your responsibility for programming it. The alarms mean air, occlusion, low volume or rising pressure; rising pressure can be an infiltrating site (trap at the end of this section). Keep a time tape on the bag even on a pump, and run children's infusions on a pump, not by gravity. Too fast causes fluid overload; too slow leaves a deficit.

Flow rates: two formulas, two units Pump: mL/h = total mL ÷ hours. Gravity: gtt/min = mL/h × drop factor ÷ 60. The drop factor is on the tubing package: macrodrip 10, 15 or 20 gtt/mL for rates above about 75 mL/h, microdrip 60 gtt/mL below it (the drop rate then equals the mL/h), and 10 gtt/mL for a blood set. The constants, the 15-second count and the worked examples are in the Dosage Calculation guide (IV flow rates).
A short drill. Cover the right two columns and work each line.
Order and setPumpGravity
1,000 mL over 5 h · 15 gtt/mL200 mL/h200 × 15 ÷ 60 = 50 gtt/min
IVPB 50 mL over 20 min · 10 gtt/mL150 mL/h150 × 10 ÷ 60 = 25 gtt/min
360 mL over 6 h · 60 gtt/mL60 mL/h60 gtt/min: microdrip, the same number
Speeding up a bag that has fallen behind Raising the rate so the bag finishes on time is the most-picked answer, because it seems logical and in a healthy adult it is often harmless. In heart or kidney failure that extra volume is exactly what causes overload. A volume order is not a deadline: never catch up; recalculate at the ordered rate and consult the prescriber.

How often things are changed

ItemChange
Continuous primary or secondary setNo more often than every 96 h, and at least every 7 days; also with a new catheter, or at once if contaminated
Intermittent (IVPB) setEvery 24 h
PN set · lipid setEvery 24 h · every 12 h
Blood setA filter takes 4 units at most; don't reuse it after a unit that ran 4 h
Transparent dressing · gauzeAt least every 7 days · every 2 days; either at once if damp, loose or soiled
Needleless connectorNo more often than every 96 h; scrub it before every access
The PIVC itselfWhen clinically indicated: no longer needed, unused for 24 h, or a complication. Never by dwell time alone

ATI and older test banks give a routine PIVC change every 72–96 h; Taylor replaces it when clinically indicated.

Discontinuing a PIVC, in six steps. (1) Clamp the tubing. (2) Remove the dressing while stabilising the hub. (3) Withdraw the catheter without bending it, and don't press on it as it comes out. (4) Then press dry sterile gauze on the site until the bleeding stops; an alcohol pad stings and does not stop bleeding. (5) Check that the tip is intact; if it is not, put a tourniquet high on the limb and notify at once. (6) Occlusive dressing, then document.

When something goes wrong at the site, five rules. Resistance on flushing: never force it; restart elsewhere. Catheter partly out: never re-advance it. Tubing disconnected: discard the set. Artery hit, or tingling on insertion: remove, apply pressure, notify. Hematoma forming: remove and use a new site.

Caring for a running IV: the nurse's five jobs

JobWhat you do
AssessBeyond the 30-minute and hourly checks (step 5 above), a full site check, inspect then palpate for temperature, at least every 4 h; every 1–2 h with a vesicant or irritant, a confused patient or a site over a joint; hourly in children. Then the patient, not the pump: vital signs, lung sounds, I&O, daily weight (Part 2)
ActRun the ordered rate on a pump; keep the bag, tubing and dressing labelled; change the bag before it runs dry; act on the first sign of a complication (table below)
Teach“Press your call light if the skin around the tube hurts, burns, swells, feels cold, tight or wet, or turns red, even a little. Don't wait for the pump to beep, and don't touch the pump or the clamps.” Cover the site to shower
DelegateAssistive personnel measure and record I&O and report any site complaint, wet dressing or alarm. They never start, adjust, flush or discontinue an IV, or restart a pump. An LPN's IV role is set by state law and facility policy
EvaluateIs the fluid problem correcting (weight, I&O over 2–3 days, vital signs, labs), and is the site still quiet?
Restarting the beeping pump An occlusion alarm usually means a kinked line or a bent wrist, so most people (and most assistive personnel) just press restart. The reasoning is usually right, which is what makes the habit dangerous: rising pressure can be an infiltrating site, and restarting pushes more fluid into the tissue. An alarm is a cue to assess the site and the patient, and only the nurse touches the pump.
Objective W9·5 · Lab L6·3 · Ch 41

IV complications: read the site, then act top-weighted topic

Local complications are more common; systemic ones (overload, air embolism, sepsis) are more dangerous. Pain and swelling go with every local complication, so they decide nothing. Temperature and colour decide. The table below has eight complications.

Read the site in four cues Cool and pale = a leak (infiltration or extravasation). Warm and red = inflammation (phlebitis or infection). Pus = infection. A cord along the vein = phlebitis or a clot. It is the same logic as NUR 120's cold limb with an arterial block versus the warm limb with a venous clot.
Exam worksheet question: infection vs infiltration vs phlebitis vs emboli Exam worksheet, open question 3; deck slide 56 lists the complications
ComplicationWhat happenedSignsFirst actionThen
InfiltrationNon-vesicant fluid leaks into tissueCool, pale, swollen, sore; flow slows; leaking at the siteStop the infusionRemove the catheter; outline the area; check circulation, sensation and movement below it; elevate; restart at a new site
ExtravasationVesicant leaks into tissueThe same look, plus burning or stinging; tissue damageStop the infusionFollow the protocol (aspirate, antidote if one exists); notify
PhlebitisVein wall inflamed, mechanically or chemicallyWarm, red, tender along the vein; streak; palpable cordStop and removeWarm compress; grade it; restart elsewhere, never in that vein
ThrombusClot from vein traumaLike phlebitis; the flow may stopStopNever rub, massage or flush it through; restart elsewhere
InfectionOrganisms at the site, or in the bloodRedness, induration, pus; fever, chills, malaiseStop and notifyCulture per protocol; strict asepsis
Speed shockToo-rapid infusionPounding headache, fainting, racing pulse, apprehension, chills, back pain, dyspneaStop or slow; notifyPrevented by the right tubing, a pump and a time tape
Fluid overloadToo much volumeEngorged neck veins, rising BP, dyspnea, crackles, bounding pulseSlow the infusionRaise the head of the bed; notify; vital signs
Air embolismAir enters, e.g. through a break in the systemSudden respiratory distress, rising HR, cyanosis, falling BP, change in LOCClose the system: clamp or pinchLeft side, head down; call for help; vital signs and SpO₂

Phlebitis is graded 0–4: 1 redness; 2 pain with redness or swelling; 3 adds a streak and a palpable cord; 4 a cord longer than 1 inch with pus.

“Emboli” is plural on the worksheet: three things can travel. Air, through a break in the system (the last row). A clot: a thrombus at the site can break loose, which is why a clotted line is never rubbed, massaged or flushed through. A piece of the catheter: inspect the tip whenever a catheter comes out; if it is not intact, apply a tourniquet high on the extremity and notify at once, to keep the fragment from travelling. Infiltration and phlebitis stay at the site; an embolus leaves it, and so can an infection, into the bloodstream.

Left side, head down is the second step The position is the famous fact about air embolism, so most people pick it first. While you turn the patient, air is still entering through the open line. Close the leak before you treat its effects: clamp, then position. The same order holds for every complication in the table: stop the cause, then manage the damage.
Two sites that look the same A cool, pale, swollen site running saline and one running a vesicant look identical, so most people call both infiltration. The second is extravasation, and its tissue damage makes it the urgent one, even when it is the smaller swelling. Infiltration and extravasation are separated by what leaked, not by how the site looks.
Objective W9·3 · W9·5 · Ch 41

IV potassium: the speed limit

Exam worksheet question: how is IV potassium given, what is the maximum, and why? Exam worksheet, open question 6
HowNever IV push or bolus (Exam 1: concentrated KCl is never pushed, under any circumstance). Always diluted, always on a pump. Check urine output first: the kidneys are the main way potassium leaves.
MaximumNo faster than 10 mEq per hour. So 20 mEq takes at least 2 hours, and 40 mEq at least 4.
WhyA bolus raises serum potassium suddenly, and sudden hyperkalemia can cause a fatal dysrhythmia (Part 3, Potassium and sodium).
AlsoIt can sting the vein: warn the patient, and watch the site.
Running it faster because the potassium is lower With K 2.9, most people want the 20 mEq bag in over 30 minutes, the usual piggyback time, because urgent deficits are usually corrected quickly. With potassium that habit is lethal. Potassium has a speed limit that urgency does not change: 10 mEq/h at most, by pump, never pushed.
Topic: blood and blood products · Ch 41

Blood: compatibility, giving it, and reactions

Typing identifies the blood type; crossmatching tests the donor's blood against the recipient's. A red cell carries antigens, and the plasma carries antibodies against the antigens the person lacks.

TypeAntigen on red cellsAntibodies in plasmaCan receive red cells from
AAAnti-BA, O
BBAnti-AB, O
ABA and BNone: universal recipientA, B, AB, O
ONone: universal donorAnti-A and anti-BO only

Rh is a second, separate match. The D antigen present means Rh-positive. An Rh-negative patient receives Rh-negative blood: Rh-positive blood makes them form antibodies, and a later transfusion then destroys red cells. An Rh-negative donor can give to an Rh-positive patient.

Components, five. Packed red cells raise the hemoglobin without much volume; fresh frozen plasma gives clotting factors and volume; platelets treat a low count; albumin treats low albumin and hypovolemic shock; cryoprecipitate treats low fibrinogen and hemophilia. Whole blood is rare.

“A matches A” For an A-negative patient, A-positive is the most-picked unit, because the letter matches. It fails the Rh match. O-positive tempts next, as the “universal donor”, and fails the same way. Check two separate matches: no foreign ABO antigen, and Rh-negative patients get Rh-negative blood.
Exam worksheet question: the important concepts when giving blood Exam worksheet, open question 4

Nine steps, in order:

  1. Verify the order and the informed consent.
  2. Before collecting the unit: vital signs within 30 minutes of starting, lung sounds, risk conditions (fever, heart failure, kidney disease) and a patent IV, 20–24 g, or 18–20 g for a rapid transfusion.
  3. Collect the unit and start it promptly, within about 30 minutes or per policy.
  4. Two qualified people check at the bedside, with the patient: two identifiers against the band, the patient's ABO and Rh against the unit's, the donation number and the expiry. Any mismatch: don't spike it; call the blood bank.
  5. Use a blood set with a filter, primed with 0.9% NaCl only. Never dextrose, which destroys red cells, and never add a medication to the line.
  6. Start slowly and stay for the first 15 minutes: major reactions usually appear within the first 50 mL.
  7. Vital signs at 15 minutes; with no reaction, raise the rate as ordered. Assess at least every 30 minutes, with vital signs per policy.
  8. Finish within 4 hours.
  9. Vital signs at the end and 1 hour later. Watch for 4–6 hours for a delayed fever or breathing trouble, and teach the patient those signs.

Document the donation (unit) number, the times started and finished, the volume (on the I&O), the vital signs and the patient's response. The bedside check and the monitoring stay with licensed nurses; assistive personnel report any new symptom at once.

ATI's modules give three different gauges for blood (18–20, 18–24, 20–24 g); Taylor's 20–24 g, or 18–20 g for a rapid transfusion, is the one to learn.

Exam worksheet question: the signs of a transfusion reaction Exam worksheet, open question 5; deck slide 58 lists the five

Every reaction starts with the same six steps: (1) stop the transfusion; (2) keep the vein open with 0.9% NaCl through new tubing, so the blood in the line is not pushed in; (3) stay, assess, and compare vital signs with the baseline; (4) notify the provider and the blood bank; (5) send the bag and tubing, and collect blood and urine samples per protocol; (6) document. The signature tells you which reaction it is. The one exception is circulatory overload: the problem is volume, so the textbook allows slowing rather than stopping, and the saline that keeps the vein open runs at the slowest rate.

ReactionSignatureAfter stop and saline
AllergicHives, itching; can progress to anaphylaxisNotify; antihistamine as ordered
FebrileFever, chills, headache, malaiseNotify; treat the symptoms
HemolyticIncompatible blood, immediate: flushing, fever, chills, headache, low back pain, falling BP, shockNotify at once; blood samples and the first voided urine; treat shock; send the unit, tubing and filter to the lab
Circulatory overloadDyspnea, dry cough, crackles, rising BPSlow or stop; sit upright with feet dependent; vital signs; notify
BacterialFever, dry flushed skin, abdominal pain; blood pressure changes (see the note below)Blood cultures; return the bag; antibiotics as ordered
Not one of theseSwelling only at the IV siteInfiltration: site care, as in the complications table

Blood pressure is where the sources split. For bacterial contamination, Taylor's table prints hypertension while ATI lists hypotension, the usual septic picture; key on the fever and the contaminated unit, not on the blood pressure. For circulatory overload, the rising BP is ATI's; Taylor lists dyspnea, dry cough and pulmonary edema.

Calling first during a reaction “Notify the provider” is the most-picked first step, because reporting feels like the safe move. Every minute the unit keeps running pushes in more of the blood causing the reaction. When the infusion is the cause, stop it first, keep the vein open with saline, then call.
Breathless during a transfusion is not always allergy An older patient with heart failure who becomes short of breath during a second unit tempts most people toward an allergic reaction. With no hives or wheeze, but crackles and a rising BP, it is volume. Classify a reaction by its signature: hives allergic, fever febrile, back pain and shock hemolytic, crackles and rising BP overload.
Check yourself · Part 5
Two patients on IV saline. A: a forearm site swollen 4 inches, cool and pale. B: a 78-year-old with heart failure at 150 mL/h, with a new dry cough, RR 26 and distended neck veins. Whom do you go to first?
B. Overload is systemic and threatens breathing: slow the infusion, raise the head of the bed, notify. A's swelling looks worse but is local. Rank by threatened function.
D5W is ordered for two patients: one with Na 151 after days of poor intake, one with a head injury and rising intracranial pressure. Is it right for both?
Only the first. Once the dextrose is used, D5W is free water: it dilutes a high sodium, and it swells brain cells. Question the second order.
Ten minutes into a unit of red cells, the patient has chills and low back pain, and the BP has fallen from 132/80 to 100/60. Put in order: notify, run saline, stop, send the unit and urine.
Stop → saline through new tubing → notify → send the unit and first voided urine. This is a hemolytic picture; stopping comes before calling.
The patient is AB-positive. Which red cells are safe: A-negative, B-positive, O-negative, AB-positive?
All four. AB plasma has no anti-A or anti-B, so any ABO type is accepted, and an Rh-positive patient can take Rh-negative cells. Most people strike out the negatives, because the Rh rule usually forbids a mismatch; it forbids only positive into negative. Apply each match in the direction it protects.
Key terms
micturitiondetrusorinternal / external sphincter specific gravityoliguriaanuria polyuriadysurianocturia hematuriaclean-catchsampling port 24-hour urineC&Sbladder scanner PVR
Objective W11·1 · Ch 38

The route, and how voiding works

Urine passes through four structures: kidney, ureter, bladder, urethra. How the kidneys decide how much to make is Part 1, Holding the balance.

kidney → ureter → bladder → urethra Kidney ~1 million nephrons each; the whole blood volume passes through about every 30 min Ureter peristalsis carries urine down Bladder wall = detrusor smooth muscle; the urge to void begins at 150–250 mL Internal sphincter involuntary, at the bladder neck Slanted entry a mucosal fold closes over the opening: no backflow to the kidney External sphincter voluntary: start, hold back, stop Urethra female ~4 cm · male ~20 cm Two nerve supplies, two jobs SYMPATHETIC → STORE detrusor relaxes internal sphincter tightens the bladder fills at low pressure PARASYMPATHETIC → EMPTY detrusor contracts internal sphincter relaxes you relax the external one by choice Anticholinergics block emptying: retention. Cholinergic drugs make the bladder empty.
The urinary tract from the front, and the two nerve supplies that switch the bladder between storing and emptying.

Voiding (micturition) is a spinal reflex that the brain learns to control after infancy. Stretch receptors fire as the bladder fills, and the urge begins at about 150–250 mL. To void, the detrusor contracts, the internal sphincter, perineal muscles and external sphincter relax, and the abdominal wall tightens slightly. Voluntary control covers three things only: starting, holding back and interrupting. Normal voiding is painless. Embarrassment can stop it altogether, which matters when you need a specimen. A bladder cut off from the brain by injury or disease still empties by reflex: an autonomic bladder (Part 7, Incontinence).

The female urethra is short and close to the vagina and rectum, which is why women get more UTIs (Part 7). The male urethra is long and curved and passes through the prostate, which can obstruct it.

ATI says the adult bladder comfortably holds 300–400 mL and signals at 250–400 mL. Use 150–250 mL for the point at which the urge begins.

Objective W11·1 · Ch 38

Normal voiding and normal output

Healthy adults void roughly every 3–4 hours while awake and usually not during sleep. The first morning void is the most concentrated, which is why some tests ask for it. How often someone voids depends on how much urine is being made, so a change from the person's usual pattern, not the pattern itself, is what suggests illness. Habitually holding urine for 8–12 waking hours breeds UTIs.

The output numbers, with the arithmetic to do in your head.
NumberWhat it meansThe arithmetic
≥ 30 mL/hThe minimum adult output. Below it, report.240 mL per 8-hour shift · 720 mL a day
< 400 mL/24 hOliguriaabout 17 mL/h
< 50 mL/24 hAnuriaabout 2 mL/h: essentially none
6–8 wet diapers/dayExpected for a newbornA diaper dry for more than 3 h signals dehydration
30 mL an hour is the adult version of the floor The same floor per kilogram is about 0.5 mL/kg/h, and infants normally make more, 1–2 mL/kg/h. Judge a child's output by weight, not by 30.

Taylor gives typical adult output as about 1,500 mL a day, roughly 40–80 mL/h. Those describe average output; 30 mL/h is the report threshold.

Objective W11·2 · Ch 38

What changes voiding

Seven factors. The medications have their own two lists below.
FactorWhat it does
Infants and childrenNewborn urine is pale and odourless. Voluntary control comes at 18–24 months; most children are dry between 2 and 5, day before night, girls before boys. Regression in hospital is expected and short-lived. Nocturnal enuresis usually resolves by 6.
Older adultsLess concentrating ability: nocturia. Lower tone: frequency, urgency. Weaker contraction: retention, stasis, UTI. Enlarged prostate in men over 50. Incontinence is not a normal part of ageing: it is a treatable symptom.
Food and fluidLow intake: less, darker urine. Alcohol inhibits ADH, and caffeine also increases output. High-sodium foods hold fluid back, so less urine.
PsychologicalNo privacy can stop voiding; stress can cause small, frequent voids.
Activity and toneImmobility causes stasis and poor control. An indwelling catheter costs bladder tone, because the bladder is never stretched by filling. Childbirth and menopause weaken the pelvic floor.
Surgery and anaesthesiaPelvic or lower abdominal surgery, epidural or spinal anaesthesia, opioids and delayed walking favour retention (Part 7).
DiseaseFever and sweating: less, concentrated urine. Heart failure: less urine. High blood glucose: osmotic diuresis, so polyuria. Arthritis and Parkinson's make toileting hard.

Eight diseases linked to kidney problems: congenital urinary tract abnormalities, polycystic kidney disease, UTI, urinary stones, hypertension, diabetes mellitus, gout and connective tissue disorders. The last four are diseases of other systems, so they are the ones most often missed on a select-all item. Ask about all eight in the history (next section).

Drugs that change the amount or emptying. Retention: anticholinergics, antihistamines and OTC cold remedies, antidepressants, calcium-channel blockers, opioids and epidural analgesia. More urine: diuretics. Blunted reflex or awareness: analgesics, sedatives and tranquillisers. Nephrotoxic: NSAIDs such as aspirin and ibuprofen, and gentamicin. Watch I&O and the renal labs, because nephrotoxicity does not announce itself as frequency, incontinence or nocturia.

Colour changes. Only one row may be blood.
CauseUrine colourIs it blood?
AnticoagulantsPink or redPossibly: assess for bleeding
Beets, blackberries, rhubarbRedNo
Phenazopyridine (Pyridium), a urinary analgesicOrange to orange-redNo; expected
DiureticsPale yellowNo; dilution
Amitriptyline; B-complex vitaminsGreen or blue-greenNo
Levodopa; injectable ironBrown or blackNo
Bile pigments (liver disease)Brown, tea-colouredNo, but abnormal
“It's just the beets”, on warfarin Reassuring her is the most-picked answer, because beets really do turn urine red. On an anticoagulant, red urine may be genuine bleeding. Test the urine for blood, look for other bleeding and report. When a finding has a harmless and a dangerous explanation, test before you reassure.

Nocturia, a refresher (NUR 120 Exam 4): alone in an older adult it is ageing; with ankle edema and orthopnea it points to heart failure.

Lab L9·3, L9·4 · Ch 38

Assessing urinary function: ask, inspect, palpate, measure

A focused urinary assessment runs in four steps, in this order: ask, inspect, palpate or percuss, measure. Judge every finding against the patient's own usual pattern.

1. Ask: the history, under five headings

HeadingAsk, in plain wordsWhat it gives you
Usual pattern“How often do you pass water in the day? Do you get up at night to go? What does your urine usually look like?”The baseline
Recent changes“Any change in how often, how much, or how strong the stream is? Any leaking when you cough or sneeze, or on the way to the bathroom?”A change, which is what suggests illness
Past or present problems“Any urgency, burning, trouble starting, dribbling? Bladder infections or kidney trouble before? Do you use pads?”Dysuria, hesitancy, incontinence; the renal history (the eight diseases above)
Aids“Is there anything you do that helps you go?”Routines to keep in the care plan
Urinary diversion“How do you look after your stoma, and what equipment do you use?”Their routine and supplies, recorded (Part 7, Urinary diversions)

Use the patient's own words for urinating; for a child, record the child's word for needing to go, and for an infant count wet diapers. Review the medication list against the drugs above. When a problem comes up, explore how long, how bad, what brings it on and what they already do about it.

2–3. Inspect, then palpate and percuss: five sites

SiteWhat you doReport or act on
SkinInspect colour, texture, turgor (Part 2) and the perineal skinExcoriation or IAD from incontinence (Part 7)
Urethral meatusInspect for inflammation, discharge and odour. Female: dorsal recumbent, labia retracted. Male: retract the foreskin if uncircumcised, then replace itInfection cues: report
UrineInspect colour, odour, clarity and sediment; SG, pH and constituents when indicated. In acute care, at least once a shift, even if the patient measures their ownAnything on the abnormal list (next section)
BladderAsk when they last voided first. Inspect above the symphysis pubis for swelling, palpate for tenderness, percuss, or use the bedside scanner (The bladder scanner, below). An empty bladder can't be felt; a full one rises above the symphysis, sometimes to just below the umbilicusDistended: scan, then retention care (Part 7)
Not yours: kidneysDeep palpation, done by an advanced practitioner—

4. Measure: three situations

PatientHow
ContinentGloves. Void into a urinal, bedpan or specimen hat under the toilet seat. Pour into a calibrated container, set it on a flat surface and read it at eye level, record. Discard in the toilet unless a specimen is needed.
IncontinentCount the episodes and note colour and odour. Weigh the pad: wet minus dry, 1 g = 1 mL. Toileting about every 2 hours gives urine to measure and prevents episodes.
Indwelling catheterDrain the bag into the patient's own graduated container at least every 8 hours; the bag's own markings are only approximate (spout care: Part 7). For hourly output in the acutely ill, a urimeter: read it, then tip it into the main bag.

Who does what. An AP may measure, empty and record. The nurse makes sure the AP knows the procedure, validates the accuracy, and does the assessment and the interpreting (Part 2, Measuring fluid status).

Report, in five findings Output under 30 mL/h (by weight in a child); a distended bladder or a PVR over 100 mL; meatal discharge, or cloudy, foul urine with fever; red urine on an anticoagulant; and any change from the patient's usual pattern that the history can't explain.
Palpating before asking Going straight to the hands-on step is the most-picked first action, because a distended bladder is the finding you are hunting for. But a bladder you can't feel means one thing ten minutes after a void and another after eight hours without one, and only the history says which. Ask before you touch: the history decides what the hands-on findings mean.
Objective W11·3 · Ch 38

Normal versus abnormal urine

Seven characteristics. Specific gravity measures how well the kidneys concentrate.
FeatureNormalAbnormal, and what it suggests
Volume≥ 30 mL/hLess: report. Large volumes: polyuria (terms below).
ColourPale yellow, straw or amber, by concentrationDark amber: concentrated. Red or pink: blood or food. Brown: bile. Drugs: the table above.
ClarityClear when freshCloudy fresh urine: RBCs, WBCs, bacteria, vaginal discharge, sperm or prostatic fluid.
OdourFaintly aromaticSweet: glucose. Fetid: heavy infection. Musty after asparagus.
Specific gravity1.010–1.025High: concentrated, usually dehydration. Low: dilute, usually overhydration. Glucose or protein raise it.
pH4.6–8.2A protein-rich diet acidifies; citrus, dairy and vegetables alkalinise.
ConstituentsUrea, creatinine, uric acid, electrolytesAbnormal: blood, pus, protein, glucose, ketones, casts, gross bacteria, bile.

Taylor gives specific gravity as 1.005–1.030 in Ch 41 and 1.015–1.025 in Ch 38, and urine pH as 4.5–8 (usually 5–6). A value well outside every range, such as 1.035 or 1.002, means the same thing under all of them.

The reagent strip (dipstick) screens at the bedside. Hold the thick end, dip so every pad is soaked, tap off the excess, lay it on its side and read it against the colour chart at the time the manufacturer specifies. An abnormal strip needs lab confirmation.

Pad positiveSuggests
GlucoseHigh blood glucose spilling over (diabetes); also sweet odour and a higher SG
KetonesFat breakdown: uncontrolled diabetes, starvation
ProteinKidney damage
BloodCatheter or cystoscopy trauma, stones, infection, anticoagulants. Note menstruation on the slip.
Leukocyte esterase, nitritesWBCs and bacteria: infection (Part 7)
BilirubinLiver or biliary disease
Deck question: does normal fresh urine smell of ammonia? Ch 38 deck, slides 21–22

False. Fresh urine smells faintly aromatic. An ammonia smell develops as urine stands, from bacterial action, so it tells you about the specimen's age before it tells you anything about the patient.

The specimen that sat on the counter Three hours later it is cloudy, alkaline and smells of ammonia, and most people call it a UTI because every finding is on the infection list. Standing alone produces all three. Recollect. Before interpreting a finding, ask whether the way the specimen was handled explains it.
SG 1.035 with 4+ glucose does not prove dehydration “High SG means dehydrated” is usually right, which is exactly what makes the habit dangerous: glucose and protein raise SG without the urine being concentrated. Judge hydration from I&O, weight and vital signs. Check what else is dissolved in the urine before reading SG as hydration.
Read SG with the output SG 1.003 after a litre of water is a kidney doing its job. SG 1.003 with 15 mL/h and a rising creatinine is a kidney that has lost the ability to concentrate. Dilute urine is reassuring only when there is plenty of it.
Lab L9·1 · Ch 38

The eight terms, as one family

Each term answers a different question about the urine. The hook names it.
TermMeansHook: the question it answers
AnuriaLess than 50 mL in 24 h: kidney shutdownHow much is made? Essentially none
OliguriaLess than 400 mL in 24 hHow much is made? Too little
PolyuriaExcessive output (diuresis); no fixed numberHow much is made? Too much: high glucose, diuretics, alcohol, a big intake
FrequencyVoiding more often than usualHow often?
UrgencyA strong, sudden need to voidHow hard does it push?
NocturiaWaking at night to voidWhen?
DysuriaPainful or difficult voidingHow does it feel? Expected briefly after catheter removal or cystoscopy
HematuriaBlood in the urineWhat is in it?
Not one of these: retentionUrine made normally but not emptiedA full bladder and an empty bag (Part 7, Retention)

Also: glycosuria, proteinuria, pyuria (pus) and bacteriuria (bacteria without symptoms, not a UTI: Part 7).

Deck question: which term means under 50 mL of urine in 24 hours? Ch 38 deck, slides 9–10

Anuria, which the deck calls synonymous with kidney shutdown or renal failure. The three distractors each answer a different question in the table: dysuria is how voiding feels, glycosuria is sugar in the urine, pyuria is pus in it. And an empty bag over a full bladder is retention, not anuria. Anuria and oliguria are about how much the kidneys make; scan before you name it.

Some sources set anuria at less than 100 mL in 24 h. Taylor and the Ch 38 deck both use less than 50 mL.

Lab L9·4 · Ch 38

Collecting urine specimens

Every specimen is labelled in front of the patient (two identifiers, date and time) and bagged. Urine changes as it stands, so it reaches the lab promptly (ideally within 2 hours) or is refrigerated.

Eight specimens. Only a catheter route gives a sterile one.
SpecimenSterile?HowAmount, use
Routine UANoVoid into a clean bedpan, urinal or hat; keep out stool and toilet tissue.≥ 10 mL
Clean-catch midstreamClean, not sterileClean the meatus (female: separate the labia, front to back; male: retract the foreskin, and replace it afterwards). Void the first ~30 mL into the toilet, catch the middle of the stream without touching the meatus, finish in the toilet.3–5 mL minimum; the usual culture specimen from a patient who can void
Straight catheterSterileSurgical asepsis, for a patient who can't void.Culture ~3 mL
Indwelling catheterSterileFrom the sampling port with a syringe (steps below). Never from the drainage bag, where urine is stale, and never by opening the closed system.Culture ~3 mL; UA ≥ 10 mL
24-hour (timed)NoStart by voiding and discarding; record the time. Keep every void for 24 h, cold, with any preservative the lab specifies. At the end, void and add it. A missed void restarts the test. Post a sign on the bathroom door.Creatinine clearance, protein
First morningNoThe first void after waking.Most concentrated; pregnancy test
Infant or childVariesMidstream is possible over age 2 with help. Infants: an adhesive collection bag; never squeeze a wet diaper. Suprapubic aspiration is not an RN skill.—
Urinary diversionUA no; culture yesFor a culture, never use the existing pouch; catheterise the stoma (Part 7).—

A culture and sensitivity (C&S) names the organism and the antibiotics that work on it, in 24–48 hours; a UA shows bacteria but not which ones.

From the port of an indwelling catheter, in six steps:

  1. Gather a syringe sized to the test, an antiseptic swab, a sterile container, clean gloves and a clamp.
  2. If the tubing is empty, clamp it below the port for no more than 30 minutes so fresh urine collects.
  3. Scrub the port with the swab and let it dry.
  4. Attach the syringe and aspirate slowly.
  5. Remove the syringe and release the clamp.
  6. Transfer to the sterile container, label at the bedside, bag and send.
Deck question: is a catheter specimen taken from the collection bag? Ch 38 deck, slides 24–25

False. It comes from the catheter itself, which in practice means the sampling port, by the six steps above. Bag urine has been standing, so it is stale and contaminated; disconnecting the tubing to reach fresher urine opens the closed system. A specimen is only as fresh and as sterile as the place you took it from: from a catheter, the port, never the bag.

The Ch 38 deck now confirms the port, as Taylor does. The only outlier is the insertion checklist, which takes a specimen at insertion from the brand-new drainage bag. For any exam item, answer with the port.

A clean-catch is not sterile Most people call it sterile: the cup is sterile, the meatus is cleaned, and a common flashcard says so. Cleaning only minimises contamination; the urine still crosses the meatus and skin. A specimen is sterile only when it comes straight out of the bladder, by straight catheter or through the port.
Sterile specimen, clean gloves A port specimen is sterile because of its route. You collect it with clean gloves, an antiseptic swab and a sterile container, not a sterile field. “Sterile specimen” and “sterile gloves” are two separate questions.
Keeping the first void of a 24-hour collection The classic error, because “start the collection” sounds like “collect this one”. That urine was made before the clock started. Discard the first, keep the last, and tie both to a clock time.
Lab L9·3 · Ch 38

The bladder scanner and the post-void residual

A portable ultrasound bladder scanner estimates the volume in the bladder. It is non-invasive, painless and carries almost no infection risk, so it is preferred over catheterising just to find out, and it cuts catheter use and CAUTI. The post-void residual (PVR) is what is left straight after voiding.

Scanning, in seven steps:

  1. Patient supine and draped; a right-handed nurse stands on the right.
  2. Turn it on, choose SCAN and select the patient's biological sex.
  3. Clean the scanner head and palpate the symphysis pubis.
  4. Put a generous amount of ultrasound gel, never lubricant, midline 1–1.5 in above the symphysis.
  5. Point the direction icon toward the head and aim slightly down toward the coccyx.
  6. Centre the bladder on the crosshairs, rescanning until it is centred, then read the volume.
  7. Clean up; document why you scanned, the volume and the response.

A PVR under 50 mL is adequate emptying; over 100 mL, the bladder is not emptying properly. Measuring the residual does not help the patient void; what comes next is retention care (Part 7). Ascites makes the reading inaccurate.

Pressing “female” after a hysterectomy Choosing the female setting for a woman is right almost every time, which is why it misleads here: that setting subtracts a uterus that is no longer there. Use the male setting. The setting describes the anatomy behind the bladder, not the patient's sex.
The scanner reads 12 mL over a firm, tender bladder Most people trust the device, because devices usually are right. Recheck the gel, position, aim and crosshairs, and rescan. When the patient and the device disagree, fix the measurement before you trust the number.

The same scan tells retention (a full bladder) from low production (a nearly empty one): Part 7, Retention.

Topic W11·T4 · Ch 38

Other diagnostic tests, briefly

Four tests. The aftercare column is the testable one.
TestBeforeAfter: watch for
Urodynamic studiesBladder full; tell them a catheter will be inserted8–10 glasses of water over 24 h; teach the signs of UTI
Cystoscopy (urethra and bladder)Informed consent; sedation and analgesiaDysuria and hematuria are expected at first; generous fluids; output for at least 24 h; watch for retention and infection
IV pyelogramNPO; allergy history; not with a raised BUN or creatinine, or in pregnancy; bowel prep; consentContrast reaction (rash, nausea, hives); contrast can cause acute kidney failure, so watch I&O
CT with contrastNPO about 8 h; allergy history; remove metalA delayed contrast reaction
Check yourself · Part 6
Two bladder scans read 150 mL. A was scanned right after voiding 300 mL. B is 8 hours post-op and has not voided. What does each mean?
A: a PVR over 100 mL, incomplete emptying; report. B: the bladder is not full, so the problem is production (about 19 mL/h). Assess fluid status and notify; a catheter won't help.
Urine output has been 8 mL/h for 3 hours in a 6-kg infant and in a 70-kg adult. What does each mean?
Infant: expected, about 1.3 mL/kg/h, inside the infant range of 1–2. Adult: report now, far below 30 mL/h. Calling both low because both are under 30 is the common slip. Judge a child's output per kilogram.
Best 24-hour urine instruction? (a) “Collect all your urine for a day.” (b) “Begin with your first morning specimen.” (c) “Discard the initial void and include the terminal void.” (d) “When you get up at 0700, empty your bladder into the toilet. Save every drop after that in the jug in the fridge, including the 0700 void next morning.”
(d), anchored to the patient's own morning. (a) is vague, (b) keeps the void to discard, (c) is jargon.
A: urine SG 1.035, dark amber, alert and now drinking well, HR 88. B: 22 mL/h for 3 hours, HR 112, newly restless. Who first?
B. A's SG is the only value out of range, which is why it tempts, but B's output and heart rate threaten perfusion now. Rank by threatened function.
Key terms
stressurgemixed overflowfunctionalreflex transienttotal (continuous)urinary retention cystitispyelonephritisCAUTI IADPFMT (Kegel)bladder training prompted voidingintermittent catheterindwelling catheter suprapubiccoudéfenestrated drape ileal conduitcontinent diversionnephrostomy
Objective W11·4 · Ch 38

Incontinence: sort it by where the failure is top-weighted topic

Three rules hold for every type. Behavioural measures come first. An indwelling catheter is the last choice, because of infection. Absorbent products are not a treatment: they protect skin and clothing, and used carelessly they cause skin breakdown and UTI. Choose one on six factors: functional disability, type and severity, gender, caregivers available, earlier treatment failed, and patient preference. Eight types:

TypeWhere it fails, and whyThe cue in a stemKey interventionHook
StressWeak pelvic floor or sphincter plus a rise in abdominal pressure. Childbirth, menopause, obesity, chronic constipation.Small leak with cough, sneeze, laugh, lift or standing up. No urge first.Kegels, weight loss, treat constipation; pessary; surgery last.Effort squeezes it out.
UrgeOveractive bladder. Caffeine, alcohol, faecal impaction, UTI, poor toileting habits; diuretics, especially in older adults.“I can't make it in time.” Frequency, nocturia.Bladder training; cut caffeine, alcohol, artificial sweeteners; diuretic in the morning; treat UTI or impaction.The urge wins the race.
MixedStress and urge together.Leaks with effort and with urgency.Kegels plus bladder training.Both.
OverflowBladder never empties: weak signal or a blocked outlet. Enlarged prostate, diabetes, neurological disease, drugs.Constant dribbling, distended bladder, high PVR.Empty the bladder: intermittent catheter, double voiding; treat the obstruction; review drugs.A full cup spilling over.
FunctionalThe urinary tract works; reaching the toilet doesn't. Mobility limits, memory loss, disorientation, barriers.Knows she needs to go; can't get there or undo her clothes in time.Fix the environment: commode, clear path, call light, elastic or Velcro clothing; prompted or scheduled toileting.Bladder fine, access broken.
ReflexNo sensation of fullness: the nerves to the brain are damaged (spinal cord injury, MS). Empties by reflex.Empties without warning, at fairly predictable volumes.Scheduled intermittent (self-)catheterisation; timed voiding; skin care.No message gets through.
TransientAppears suddenly and lasts 6 months or less. A temporary, treatable cause: confusion from acute illness or infection, diuretics, IV fluids.New, during an admission.Find and treat the cause; continence should return.Here today, gone with the cause.
Total (continuous)Unpredictable, constant loss: surgery, trauma, fistula.Never dry.Containment, external device, skin care; repair.Never closes.
Not one of theseRetention without leaking (next section); enuresis, wetting past toilet-training age, is a developmental term, not a mechanism.———
ASK IN THIS ORDER YES MEANS 1 Sudden and new (6 months or less), with acute illness, confusion, a diuretic, IV fluids? Transient find and treat the cause 2 Bladder distended, or PVR high after voiding? Overflow empty it; training makes it worse 3 No sensation of fullness (spinal cord injury, MS)? Reflex scheduled intermittent catheter 4 Voids normally once the barrier is removed? Functional fix the environment, prompt 5 Leaks with cough, sneeze, laugh, lift or standing up? Stress Kegels, weight loss 6 A strong urge first, and can't make it in time? Urge bladder training, cut irritants nonono nono Yes to both 5 and 6: mixed. Never dry at all: total.
The first three questions catch the look-alikes before the familiar answers (stress, urge) can.
“Goes every hour and dribbles” is not urge An older man with an enlarged prostate who voids hourly and dribbles sounds like urge, so most people pick urge and bladder training. Feel the bladder: if it is distended, or the PVR is high, it is overflow, and teaching a full bladder to hold longer makes it worse. Classify incontinence by mechanism, not by how it feels to the patient: check emptying before you name the type.
Leaking on standing up is not functional A woman who leaks when she gets up from a chair or lifts groceries sounds like a mobility problem, and the movement tempts “functional”. But she walks without difficulty, and standing raises abdominal pressure on a weak pelvic floor. A leak triggered by effort is stress incontinence, even when the effort looks like a mobility problem.
Deck question: diuretics can bring on urge incontinence Ch 38 deck, slides 13–14

True. A diuretic raises urine production, so the bladder fills faster and the urge comes sooner. Read the medication list before you name the type, and teach a morning dose so the peak doesn't fall at night. Leaking that starts suddenly with a new diuretic during an illness is still transient on the ladder.

Nursing care, any type:

StepWhat you do
Assess and monitorHistory, including the environment and walking aids; diet (caffeine, sweeteners, too little fluid); a voiding diary of times, amounts and leaks; palpation, a PVR and a urine screen for UTI, a frequent cause; the perineal skin.
ActThe type's key intervention (table); call light and urinal or commode within reach; clean and protect the skin after every episode (IAD, below).
TeachThe type's plan (Kegels, bladder training); fluids up, irritants down; incontinence is not a normal part of ageing.
DelegateAP: toileting schedules, perineal care, pads, I&O. The nurse: the type, the skin, the plan, teaching.
EvaluateFewer leaks and longer intervals in the diary; intact skin.

Priority (Exam 3): incontinence is an actual problem and skin breakdown only a risk, so incontinence ranks first.

Objective W11·4 · Ch 38

Retention: urine made but not emptied top-weighted topic

Six causes: an enlarged prostate (men far more often); drugs that block the emptying reflex, such as anticholinergics, antihistamines, antidepressants and opioids (Part 6, What changes voiding); surgery on the lower abdomen or pelvis, especially with an epidural, delayed walking or low intake; swelling after childbirth; nerve damage; and obstruction or prolapse. Immobility lowers bladder tone and leaves urine standing (Exam 2).

The cues: a bladder distended above the symphysis pubis, discomfort, frequent small voids, hesitancy, a weak stream, and dribbling (overflow). The proof is a high PVR: above 100 mL means the bladder is not emptying (Part 6, The bladder scanner). Untreated, retention leads to UTI, hydronephrosis and kidney damage.

Nursing care:

StepWhat you do
Assess and monitorAsk when they last voided and what they have drunk, then palpate above the symphysis and bladder-scan (non-invasive, no infection risk); look for the causes above, and after surgery track the time since the last void.
ActNursing measures first: privacy, usual position, running water within earshot, warm water over the perineum (cold tightens the muscles), analgesia if pain holds them back; double voiding, or Credé with an order (both below). Then intermittent (straight) catheterisation per order, the preferred route, post-op too; indwelling only for gradual decompression or retention that won't resolve. Drain no more at once than facility policy allows; treat the cause; walk early after surgery.
TeachVoid at the first urge; report hesitancy, a weak stream or fullness; ask before taking OTC decongestants or antihistamines.
DelegateAP records every void and reports a missed one; scan per agency policy. The nurse assesses and catheterises.
EvaluateVoids comfortably in adequate amounts; PVR back in range on re-scan.
“No urine” is two different problems Two patients, neither has voided in 6 hours. A scans 650 mL: retention, a problem of emptying, so nursing measures and then a catheter. B scans 40 mL: a problem of production (fluid volume, perfusion or kidney), so assess and notify; a catheter cannot drain urine that isn't there. Classify by what is in the bladder, not by what is in the bag.
Objective W11·4 · Ch 38

UTI and CAUTI top-weighted topic

UTI is one of the commonest health care–associated infections and a common source of systemic infection in older adults. E. coli from the bowel is the usual organism. A lower UTI is the bladder (cystitis) and urethra; an upper UTI is the kidneys and ureters (pyelonephritis), and an untreated lower UTI can climb there.

Seven risk factors: female anatomy (a short urethra near the vagina and rectum) and sexual activity; a diaphragm or spermicide, which kills protective flora; menopause; an indwelling catheter; diabetes; older age, including an enlarged prostate; and stasis from retention, immobility or habitually putting off voiding.

PictureWhat you see
Lower UTIDysuria, frequency, urgency with small volumes, suprapubic discomfort, cloudy, foul-smelling urine, hematuria.
Upper UTIAdds flank or low-back pain, fever and chills, nausea and vomiting.
With a catheterBurning or irritation at the meatus, cloudy urine, strong odour, fever, chills.
Older adultClassic signs may be missing. New confusion, agitation, falls, new incontinence or poor appetite may be the only sign, often without fever.

Nursing care:

StepWhat you do
Assess and monitorThe signs above and temperature; mental status in an older adult; urine colour, clarity and odour; I&O.
ActCulture before the first antibiotic dose (Exam 1; how: Part 6, Collecting urine specimens). Antibiotic as ordered, a short course for a lower UTI, longer for an upper; generous fluids unless restricted; ask whether any catheter is still needed.
TeachFinish the whole course; the six prevention points below.
DelegateAP: perineal and catheter care, fluids, I&O; reports fever, new confusion, cloudy or foul urine. The nurse assesses and teaches.
EvaluateSymptoms and fever settle; mental status back to baseline. New flank pain, fever or chills mean it has climbed: report.

Prevention, six points in a patient's words: six to eight 8-oz glasses of fluid a day; go when you first feel the urge and take time to empty fully; wipe front to back; urinate before and after sex; cotton-crotch underwear and no tight clothes; shower rather than bathe, with no douches or sprays. If infections recur, discuss alternatives to a diaphragm or spermicide.

CAUTI. About 75% of hospital UTIs are catheter-associated, and CMS treats CAUTI as preventable and does not pay for its treatment. Bacteria form a biofilm on the catheter and climb inside the lumen or along the outside. The bundle (Exam 1): insert only for a valid indication, with surgical asepsis; maintain it by the seven care rules (Caring for the catheter); remove it as early as possible.

“Most CAUTIs start with a break in sterile insertion” When a unit's CAUTI rate rises, retraining everyone on insertion is the most-picked fix, because insertion technique does matter. But most organisms get in after placement, through handling of the catheter and drainage system, and the risk grows with every catheter day. The best CAUTI prevention is not inserting one, and the next best is the removal date.
Objective W11·4 · Ch 38 and Ch 41

Kidney stones: the calcium bridge top-weighted topic

Refresher (Exam 2): with immobility, calcium leaves demineralising bone and can precipitate as renal calculi. That is the bridge from high serum calcium (Part 3, The links) to this chapter. Other risks include low urine volume, a high urine calcium, hyperparathyroidism, excess calcium or calcium antacids, gout, obesity, diuretics, recurrent UTI, male sex and a prior or family history of stones.

The picture is sharp flank pain radiating to the lower abdomen or groin, hematuria, frequency, urgency and dysuria. A stone can block the outflow and cause retention. Fever and chills mean infection behind an obstruction: report. For a small stone, strain all urine and send any stone to the lab to be identified, push fluids unless restricted, give analgesia as ordered, and track I&O and temperature; a large or obstructing stone is broken up by shock-wave lithotripsy or removed.

Objective W11·6 · Ch 38

Promoting voiding and continence

Deck question: incontinence can be prevented Ch 38 deck, slides 36–37; the four headings from slides 27 and 29

“No interventions are effective for preventing incontinence” is false. Four kinds work, and this section follows them: regular voiding habits; fluid intake, kept up, not cut; muscle tone (pelvic floor training); and help with toileting (scheduled voiding, the environment). Incontinence has a cause: name the type and treat the mechanism; containment is not treatment.

Habits, five of them: help the patient void when the urge first comes; give privacy unless they are too weak to be left; use their usual position (men standing, women often on a commode; the positioning rules are from Exam 2); stay with anyone who might fall and never lock the bathroom door; and keep fluids up, six to eight glasses a day.

Cutting fluids to leak less A patient with urge incontinence who drinks three cups a day “so I leak less” seems to be doing simple arithmetic, which is why the option tempts. Concentrated urine irritates the bladder and raises urgency, and invites UTI and constipation. Keep intake up, cut caffeine, alcohol and sweeteners, and limit fluid only near bedtime. Check that the cause the patient is treating actually drives the symptom.

Pelvic floor muscle training (Kegel). It reduces or eliminates stress incontinence and helps every type in women. The patient finds the muscle by imagining stopping urine midstream or holding back stool, then squeezes 5–10 seconds and relaxes 10, with the abdomen, thighs and buttocks relaxed, in sets through the day for at least 3 months. Stopping the stream is for finding the muscle once, not a daily exercise: repeated stop-start voiding can leave urine behind.

Scheduled voiding comes in three forms, and they differ by who runs the schedule:

Bladder trainingHabit training (timed voiding)Prompted voiding
Who drives itThe patientThe caregiverThe caregiver
CognitionIntact and motivated; can resist an urgeMay be impairedImpaired, but responds when asked
ScheduleStarts at the diary interval, then lengthens gradually; urges handled with distraction and relaxationFixed to the patient's own pattern; not lengthenedRegular checks: “wet or dry?”, prompt, help, praise
Best forUrge, mixedFunctional, cognitive impairmentFunctional, dementia

Two more techniques. Double voiding: void, wait a few minutes or lean forward, then void again, for incomplete emptying. Credé is firm downward pressure over the bladder for a flaccid bladder, used only with an order.

Incontinence-associated dermatitis (IAD). Bacteria turn urea into alkaline ammonia, which raises skin pH and breaks down the barrier. It looks like redness with poorly defined edges, warm and firm, with burning or itching. Inspect the perineum, buttocks and hips daily. Care, in four steps: prevent the incontinence; clean promptly after each episode with a pH-balanced cleanser (not soap and hot water), patting, never rubbing; moisturise, nothing with alcohol; a barrier (zinc oxide, petrolatum or a sealant) on all skin urine can reach, after every episode. Refer to a WOC nurse if it hasn't responded in 2 weeks.

Lab L9·2, L9·4 · Ch 38

Catheters: when, and which kind top-weighted topic

Every catheter needs an order (Exam 3), and urethral insertion is surgical asepsis (Exam 1). Nurses insert every type except the suprapubic and the coudé. Eight indications: acute retention; long-term drainage when nothing else works (neurogenic bladder); a sterile specimen from a patient who can't void; accurate output in the critically ill; healing an open sacral or perineal wound in an incontinent patient; around some surgery and tests; end-of-life comfort; prolonged immobilisation (unstable spine, multiple trauma). Not indications: managing incontinence, or staff convenience.

TypeWhat it isUsed forRemember
Intermittent (straight)In, drain, outRetention, post-op retention, self-catheterisationLower CAUTI risk than indwelling. Clean technique at home is supported.
Indwelling (Foley)Held by a balloon of sterile water. Double lumen: balloon and drainage. Triple: adds irrigation.Continuous drainage, decompression, irrigationUsual adult size 14F, 5–10 mL balloon. Smallest that drains; 18F and up erode the urethra.
CoudéCurved tipEnlarged prostateNot a nursing insertion (above); for when a straight catheter won't pass.
SuprapubicSurgically placed through the abdominal wall above the pubisLong-term drainage when the urethra is damaged or blockedLess contamination and lower CAUTI risk. Site care below.
External male (condom, urinary sheath)Sheath over the penis to a bagIncontinence without retentionClean technique; no CAUTI risk. Application and care below.
External femaleWick between the labia on low continuous suctionOutput, dry skin, no indwelling catheterKeep suction on while removing it.
Lab L9·2 · Ch 38

Inserting an indwelling catheter, female and male top-weighted topic

Before you open the kit: confirm the order, ask about latex and iodine allergy, get good light (a flashlight needs an assistant), use a firm surface, and get a second person if needed. The lab grades the steps below; the goal line is urine output of at least 30 mL/h with a bladder that is not distended.

Testable skill: catheter insertion, female and male Skills-lab outline, the highlighted testing line: Skills 37-5 / 37-6 in the lab outline (9th-edition numbering; 38-2 / 38-3 in Taylor 10e)
PhaseFemaleMale
PositionDorsal recumbent, knees flexed, feet about 2 ft apart; or Sims'Supine, thighs slightly apart
Pre-cleanClean gloves; front to back, a new corner of the cloth each stroke; gloves off, hand hygieneClean gloves; tip of the penis in circles from the meatus out, then the shaft downward; gloves off, hand hygiene
Sterile fieldOpen the kit on the overbed table; sterile gloves; drape cuffed over the gloved hands; fenestrated drape; attach the prefilled syringe to the balloon port; lubricate 1–2 in of the tip
ExposeNon-dominant hand spreads the labiaNon-dominant hand lifts the penis and retracts the foreskin
CleanseDominant hand, one swab per stroke: each labial fold top to bottom, then straight down over the meatus, meatus lastDominant hand: circles from the meatus down the glans, three times, a new swab each time
InsertHold 2–3 in from the tip; advance until urine returns (about 2–3 in), then 2–3 in morePenis perpendicular to the body with slight upward tension; instil 10 mL of lubricant; patient bears down; hold 1–2 in from the tip; advance to the Y (bifurcation)
InflateNever force: deep breaths and a gentle twist at resistance. Then inflate with the entire syringe of sterile water and tug gently to feel resistance. Male: replace the foreskin.
SecureInner thigh, with slackInner thigh or lower abdomen, with slack
BagBelow the bladder, hung on the bed frame, not the side rail; no kinks

The non-dominant hand is contaminated once it touches the patient; it stays in place until urine flows and never returns to the sterile field. Document the catheter type and size, the balloon volume, the urine returned and how the patient tolerated it.

Balloon: the checklist's set-up step says a prefilled sterile saline syringe and its inflation step says sterile water; Taylor and ATI use sterile water throughout. Neither the checklist nor Taylor pre-tests the balloon, though older test banks do.

Male, Taylor 10e: lubricate 5–7 in (anaesthetic gel 5–10 mL if used, after an allergy check), hold 3–4 in from the tip, advance 7–10 in until urine, then about 2 in more to the Y. Both versions stop at the Y.

Specimen at insertion: the checklist takes it from the new bag; the deck, Taylor and ATI say the port, never the bag (Part 6). Answer with the port.

FEMALE urethra about 4 cm bladder meatus 2–3 in: urine returns +2–3 in, then inflate balloon MALE urethra about 20 cm prostate bladder meatus the Y Checklist: advance to the Y, then inflate Taylor 10e: urine at 7–10 in, then about 2 in more
Not to scale. Urine shows the tip is in; the balloon sits behind the tip, so it needs the extra distance.

Female urethra: Taylor gives 4 cm (1.6 in) in its anatomy section and 1.5–2.5 in in the skill rationale. Use the skill's insertion depths.

Inflating the balloon at the first drop of urine Most people accept this, because urine does prove the catheter is in the bladder. It proves the tip is in; the balloon sits behind the tip, and inflating it in the urethra causes serious injury. Urine means the tip is in. Advance the extra distance so the balloon is in too, and when in doubt, don't inflate.
During insertionWhat you do
Male catheter won't pass beyond 3–4 inStop; don't force. Notify; expect a coudé placed by a urologist or advanced practitioner.
No urine where you expect itDon't inflate. Deep breath, rotate slightly, raise the head of the bed, check intake and kinks. Still unsure: remove it and notify.
Catheter in the vaginaLeave it as a landmark, insert a new sterile catheter into the meatus, then remove the first.
Sterility brokenStop and correct; what you replace depends on what was contaminated.
Lab L9·4 · Ch 38

Caring for the catheter, and taking it out top-weighted topic

Seven care rules:

  1. Meatal care with soap and water daily and after each bowel movement, cleaning the catheter from the meatus outward; no powders, lotions or antiseptics at the meatus.
  2. Generous fluids unless restricted: natural irrigation.
  3. Constant downward flow: no kinks, no dependent loops, not lying on the tubing.
  4. Bag below the bladder at all times, including transport, and off the floor.
  5. Keep the system closed. If it comes apart, replace the catheter and the bag.
  6. Empty into the patient's own graduated container at least every 8 hours; the spout touches nothing.
  7. Report CAUTI signs (UTI table, With a catheter). Change the catheter only when it fails, never on a schedule; remove it as soon as possible.

A leg bag is not a closed system, because it is opened to switch to a night bag; empty it at ½–⅔ full and clean the outlet each time.

Meatal care frequency: Taylor and ATI Engage say daily; ATI Skills Modules say at least once a shift. Follow facility policy.

Urine leaking around the catheter: “go up a size” Upsizing is the most-picked fix, because a bigger plug sounds like a better seal. Large catheters and balloons irritate the bladder and cause spasm. Check the balloon volume (underfilled, it slips into the urethra), constipation, and UTI. Leaking around a catheter is a symptom to investigate, not a size problem.

Three devices with care of their own:

DeviceCare
Suprapubic catheterInspect the site for redness, swelling, drainage or leaking; clean with soap and water from the site outward; dressing changed daily, clean or sterile per policy; tube secured so it can't pull. Bag and tubing as for a Foley. Report a dislodged tube, no drainage, or an inflamed site.
Condom catheterInspect the penis first: red, swollen or broken skin means don't apply it. Measure for size; perineal care, dry; trim hair, never shave. Leave 1–2 in at the tip; snug, never constricting, never taped around the shaft; check circulation in about 30 minutes. Bag below the bladder, no kinks, so urine can't pool at the glans. Off daily to wash, dry and inspect.
Bladder irrigationNot routine: fluids by mouth are preferred. When clots are expected (after prostate or bladder surgery): continuous through a triple-lumen catheter, or intermittent, 30–50 mL by syringe through the port, tubing clamped below; both closed. Sterile solution, aseptic technique, room temperature (cold causes spasm). True urine output = total drainage minus irrigant. Discomfort is expected; pain or slowing drainage is not: check for kinks and clots, then report.

Removal, in five steps:

  1. Check the record for the balloon volume; empty and measure the bag.
  2. Attach a syringe and let the balloon push the plunger back. Don't pull it, and never cut the tubing.
  3. Confirm the volume withdrawn matches the volume charted.
  4. Withdraw gently as the patient breathes out; stop and notify if you meet resistance.
  5. Perineal care.

After removal: encourage fluids; record intake and the time and amount of every void for 24 hours, voiding into a hat, and the volume of the first void. Slight burning, urgency or frequency on the first void or two is expected, and so is an accident. After a few days of catheter, watch for retention. If the patient hasn't voided within 8–10 hours, or per policy, notify; bladder-scan first if they feel full.

Many NCLEX sources teach 6–8 hours for the first void.

Objective W11·5 · Ch 38

Urinary diversions, stents and dialysis

DiversionWhat it isContinent?
Ileal conduit (urostomy)The most common. Ureters implanted into a piece of ileum brought out as a stoma.No: drains continuously into a pouch
Cutaneous ureterostomyThe ureters themselves brought to the skinNo
Continent diversion (Indiana or Kock pouch)Internal reservoir of bowel with a catheterisable stomaYes: catheterised on a schedule
NephrostomyTube through the back into the renal pelvis, often temporaryBag below kidney level; flush only if ordered

Stoma assessment. Normal is dark pink to red, moist and shiny, swollen at first and stable in size by about 6–8 weeks. Pale suggests anaemia; dusky, purple or black means poor circulation: notify promptly. Mucus in the urine is expected from any diversion built from bowel. Empty the pouch at ⅓–½ full; change it every 3–7 days, early in the morning before fluids, with the opening cut ⅛ in larger than the stoma and no lotion on the skin.

Know what is normal for the device Mucus strands in conduit urine look alarming and are expected; a stoma that darkens looks quieter and is losing its blood supply. Urine dripping from an ileal conduit is normal; from a continent pouch it is a failure. Judge a finding against what that diversion is built to do.

Stents and dialysis, briefly

DeviceWhat the nurse does
Ureteral stentKeeps urine flowing from kidney to bladder past a stone, mass, scar or swelling; usually temporary. Never irrigate it. Falling output may mean a blocked stent (hydronephrosis, kidney damage): act at once. Teach: report bright red urine, severe pain, a change in drainage, or infection.
Haemodialysis access (AV fistula or graft)Palpate the thrill and auscultate the bruit (bell); check distal pulses, capillary refill, colour, warmth, movement and sensation. Present means patent; weak or absent: notify, which outranks a trace of painless oedema. No BP, IV or blood draw in that arm (Part 5, Venous access).
Peritoneal dialysisDialysate in and out through an abdominal catheter; exit-site care is sterile, nurse and patient masked.
Topic: delegation · Ch 38

Who does what: delegating urinary care

Delegation turns on the judgement a task needs, not how routine it looks (Exam 3); the nurse validates and interprets what assistive personnel measure.

WhoTasks
Assistive personnel (AP)I&O, including emptying drainage bags and urostomy pouches; perineal and routine catheter care; toileting, scheduled and prompted voiding, pad changes, skin cleansing; routine specimens (UA, clean-catch, 24-hour); a condom catheter where policy allows. They report low output, a missed void, cloudy or bloody urine, red skin.
Per agency policyBladder scan; a sterile port specimen (often kept with licensed nurses); changing an ostomy appliance (LPN).
Licensed nurse (LPN within state scope)Inserting any catheter (surgical asepsis), straight or indwelling; irrigation.
RNAssessing (bladder, incontinence type, skin, stoma, whether a catheter is still needed); planning; teaching; evaluating; interpreting I&O.
“The AP collects specimens, so let the AP send one” An AP reports dark, cloudy urine in a catheter bag and offers to send a sample from it. Most people approve: AP do collect routine specimens, and it sounds like UTI. But judging a finding is assessment, and the bag is the wrong source: assess, then take a port specimen per order. The AP measures and reports; the nurse decides what a finding means.
Objective W11·7 · Ch 38

Writing the problem statements

ProblemRelated toAs evidenced by
Urinary retentionEnlarged prostate; anticholinergics or antidepressants; obstructionPVR 450 mL on bladder scan; hourly small voids; hesitancy
Stress urinary incontinenceWeak pelvic floor musclesLeaks with coughing, sneezing, laughing
Risk for urge incontinenceCaffeine, alcohol, faecal impaction, UTINone: a risk statement has no AEB
Caregiver role strainManaging a spouse's total incontinence“I haven't slept through the night in months”
Risk for infectionIndwelling urinary catheterNone (risk)

Urinary function can be the problem (incontinence, altered pattern, retention) or the cause of another (anxiety, caregiver role strain, risk for infection). “Catheterization R/T urinary retention” is wrong (Exam 3): a treatment is not a problem, though it can be a cause, as in the last row.

Six planned goals. Output about equal to intake; fluid and electrolyte balance; bladder emptied completely at regular intervals; reports ease of voiding; intact skin; self-care. Make each measurable: not “will void adequately” but “will void at least 200 mL within 8–10 hours of catheter removal, with a PVR under 50 mL”.

Check yourself · Part 7
A 79-year-old with moderate dementia has urge incontinence. Which plan fits: bladder training, prompted voiding, Kegels, or an indwelling catheter?
Prompted voiding. Bladder training treats urge but needs intact cognition; a catheter is never for incontinence alone. Choose on two axes: the type, and who can run the plan.
After prostate surgery, a patient on continuous bladder irrigation has 3,000 mL of irrigant in and 3,400 mL drained over 8 hours. Drainage has now slowed, with clots, and he reports cramping pain. What is his urine output, and what do you do first?
400 mL (3,400 minus 3,000), about 50 mL/h: adequate. Pain with slowing drainage is not expected: check for kinks and clots and palpate the bladder, then report. Most people pick an analgesic; here pain signals obstruction. Treat the cause of the pain before the pain.
Patient A's catheter came out 10 hours ago; she hasn't voided, is restless, and scans 700 mL. Patient B's came out 3 hours ago; he voided 250 mL with burning and urgency. Who first?
Patient A: no void past the 8–10-hour limit, with a full bladder, is retention; notify. B's burning and urgency are expected, but a complaint sounds more urgent than silence. Expected discomfort is not the problem; the missing void is.
Patient A, 84, is newly confused since breakfast, T 37.4 °C, urine dark and foul. Patient B, 26, has dysuria and frequency, T 38.1 °C, alert and drinking. Who first?
Patient A. New confusion may be the only sign of UTI or urosepsis in an older adult; it outranks a higher temperature in a patient who is compensating.
Topic: Rapid review

Numbers to know cold

Course figures only; another source's number is in grey in its part.

NumberWhat it is
Na 135–145 · K 3.5–5.0 · Cl 97–107 · Mg 1.3–2.3mEq/L. Potassium's range is only 1.5 wide. Sodium below 120 is severe: seizure precautions.
Ca 8.6–10.2 · phosphorus 2.5–4.5The two in mg/dL; they move in opposite directions.
Serum bicarbonate 25–29 · ABG HCO₃⁻ 22–26mEq/L. Two tests, two numbers: 27 is normal on a BMP, high on an ABG.
pH 7.35–7.45 · PaCO₂ 35–45 · PaO₂ 80–100PaCO₂ is the respiratory number; PaO₂ is oxygenation only.
20 : 1 · up to 90%Bicarbonate to carbonic acid · the share of ECF hydrogen ions the carbonic acid–bicarbonate buffer takes up. Lungs act in minutes, kidneys in hours to days.
285–295 mOsm/kgSerum osmolality: up in dehydration, down in overhydration. It follows the sodium, so it stays normal when salt and water are lost or gained together.
50–60% · about 45%Body water as a share of weight: adult · over 60. Infants have more, with more of it in the ECF, which is lost faster.
~70% · ~30%ICF · ECF, as shares of body water (35–40% and 15–20% of body weight).
~2,600 mL a dayIntake, and output, in health; urine is about 1,500. Judge the balance over 2–3 days. The care-plan outcome is about 2,500 mL a day, averaged over 3 days.
1.5–2 L · 1–2 LA drinking target to thin secretions · a heart-failure limit. Neither is the 2,600 turnover.
+500 · +1,000 mL a dayAt least this much extra loss with a fever of 101–103 °F · above 103 °F.
1 kg ≈ 1 L ≈ 2.2 lb · 1 g = 1 mLDaily weight, the most accurate measure · a weighed pad or diaper. Ice chips count at half.
5% · 10% · 15%Sudden weight loss: FVD in an adult · in an infant · life-threatening. A gain over 5%: FVE.
SBP ≥20 or DBP ≥10 mm HgOrthostatic drop: lying, sitting, standing.
≥ 30 mL/hMinimum adult urine output; below it, report. Per kilogram about 0.5 mL/kg/h; infants make 1–2.
< 400 · < 50 mL in 24 hOliguria · anuria.
SG 1.010–1.025 · pH 4.6–8.2Urine. 1.035 is high and 1.002 low under every source. Glucose and protein raise SG.
150–250 mLBladder volume at which the urge to void begins.
PVR < 50 · > 100 mLAdequate emptying · not emptying.
10 mEq/hIV potassium maximum: diluted, by pump, never pushed. 40 mEq takes at least 4 h.
mL/h = mL ÷ h · gtt/min = mL/h × drop factor ÷ 60Pump · gravity. Macrodrip 10, 15 or 20 gtt/mL; microdrip 60; blood set 10.
20–24 g · 18–20 gMost adults and routine blood · a rapid transfusion.
Every 4 h · 1–2 h · hourlyIV site check: routine · high-risk site · children. Assess the site and the patient, not the pump.
15 min · 4 hBlood: stay for the first 15 minutes, vital signs at 15 · finish within 4 hours.
14F · 5–10 mLUsual adult catheter and balloon, filled with sterile water.
~4 cm · ~20 cmUrethra: female, where you advance 2–3 in past the first urine · male, where you advance to the Y.
8–10 hNo void after catheter removal: notify.
Every 8 h at leastEmpty an indwelling bag into its own graduated container; a urimeter for hourly output.
1–2 in · dailyCondom catheter: space left at the tip · removed and the skin checked.
30–50 mL · drainage − irrigantIntermittent bladder irrigation · true urine output.
≤ 6 monthsTransient incontinence: appears suddenly, has a treatable cause.
Topic: Rapid review

Easy to confuse: the deciding hook

Seventeen pairsThe hook
Hypovolemia · dehydrationRead the sodium: normal when salt and water go together, high when water goes alone.
Hypervolemia · overhydrationSalt and water kept: sodium usually normal. Water alone gained: sodium low.
Fluid loss · third spacingThird-spaced fluid never left, so the weight does not fall.
Osmosis · active transportWater, free · a solute, paid for in ATP. Direction never decides.
ADH · aldosteroneADH keeps water; aldosterone keeps sodium, dumps potassium. ANP is aldosterone's opposite.
Hypotonic · hypertonicThe prefix describes the solution: cells swell · cells shrink. D5W acts hypotonic once its sugar is used.
Low · high Ca and MgLow is twitchy; high is sluggish.
The culprit · the rescueOnly the number that matches the pH names the imbalance.
Infiltration · extravasationThe same site. What leaked: non-vesicant · vesicant.
Infiltration · phlebitisCool and pale is a leak; warm and red is inflammation.
Midline · PICCSame arm vein, but a midline's tip stops at or below the axilla: not central.
Allergic · overload reactionHives · crackles and rising BP. Back pain and shock: hemolytic.
Retention · oliguriaScan: full bladder, emptying problem; empty bladder, production problem.
Clean-catch · port specimenClean, not sterile · sterile by its route.
Urge · overflowDribbling with a distended bladder or high PVR is overflow.
Stress · functionalA leak triggered by effort is stress; functional means the tract works but access doesn't.
Bladder training · prompted voidingWho runs it: the patient, with intact cognition · the caregiver.
Topic: Rapid review

What do you do first

Every option is a correct action; the order is the item. The tempting error is the famous step, or the call, jumping ahead of stopping the cause.

Fourteen situationsIn order  ·  the tempting error
InfiltrationStop the infusion → remove the catheter → outline, check circulation, elevate → new site
PhlebitisStop and remove → warm compress → restart elsewhere, never that vein
Air embolismClamp → left side, head down → call for help, vital signs, SpO₂  ·  positioning first
Fluid overload on an IVSlow the infusion → raise the head of the bed → notify
A bag running behindNever catch up → recalculate at the ordered rate → consult  ·  speeding it up
Transfusion reactionStop → keep the vein open with 0.9% NaCl, new tubing → stay, compare vital signs → notify provider and blood bank → send bag, tubing and samples → document  ·  calling first
K⁺ 6.2 with KCl runningStop the potassium → assess, monitor → notify → redraw  ·  calling or redrawing first
Breathing fast, tingling, frightenedCheck SpO₂ → coach slow breathing  ·  calling it anxiety
Hasn't voidedLast void and intake, palpate → bladder-scan → nursing measures → straight catheter per order. Bladder nearly empty: assess fluid status, notify
No urine at the YDon't inflate → deep breath, rotate, raise the head of the bed, check kinks → still unsure: remove, notify
Male catheter won't passStop; don't force → notify; expect a coudé
Catheter in the vaginaLeave it as a landmark → new sterile catheter into the meatus → remove the first
Dry, dizzy, wants the bathroomFall precautions: dangle, rise in stages, walk with help, call light in reach  ·  letting a patient who “walks fine at home” go alone
Magnesium drip, RR 10, reflexes goneStop the infusion → airway and breathing → notify; calcium gluconate within reach  ·  slowing it
Topic: Rapid review · archetype: same number, opposite meaning

Same finding, two answers

FindingIn this patientIn that patient
K 3.4Healthy, eating normally: diet teachingOn digoxin and furosemide: report before the dose
Na 128Heart failure, edema, crackles: restrict fluidDays of vomiting, orthostatic drop: sodium and volume
Tented skin, back of hand84, drinking normally: age; retest over the sternum28, after diarrhea: FVD
HCO₃⁻ 27BMP: normalABG: high
High PaCO₂Acid pH: respiratory acidosisAlkaline pH after NG suction, HCO₃⁻ 34: the rescue
Deep, rapid breathingAnxiety, hypoxemia ruled out: coach slow breathingDKA: Kussmaul, the rescue; don't slow it
D5W orderedNa 151 after poor intake: appropriateRising intracranial pressure: question it
Massive edemaHeart failure with crackles: FVEDay after a burn, urine 15 mL/h: vessels short; keep fluids running
SG 1.003After a litre of water: normal response15 mL/h, rising creatinine: cannot concentrate
Scan 150 mLJust after voiding: PVR over 100; report8 h post-op, no void: a production problem
Red urineAfter beets: harmlessOn warfarin: test for blood, report

Before you act on a value, find the test, drug, diagnosis or device it belongs to.

Topic: Rapid review · not yet settled

Still open: ask the instructor

The first row is flagged “Ask the instructor” where it is taught; the other four are source splits a part has settled for now. Meanwhile, use the third column.

QuestionThe versionsAnswer with, meanwhileWhere
How far does compensation go?Program outline: non-compensatory. The deck prints rescue values. Taylor grades partial or full.Name it from the number matching the pH; skip partial versus full.Part 4
Normal urine SGThe deck's 1.010–1.025; Taylor's 1.005–1.030 and 1.015–1.025.1.010–1.025Part 6
First void after removalTaylor 8–10 h; many NCLEX sources 6–8 h.8–10 h, or per policyPart 7
Balloon fluidChecklist: saline at set-up, water at inflation. Taylor and ATI: water.Sterile waterPart 7
PIVC changeATI: every 72–96 h. Taylor: when clinically indicated.When clinically indicatedPart 5

The Chapter 38 deck settled one former split: a catheter specimen comes from the sampling port, never the bag. The instructor's Chapter 41 lecture slides are still to come; this list will be updated once they are posted.

Read the recap
Part 1 · Ch 41

Recap: Where Body Water Lives, and How It Moves

Body water is 50 to 60 percent of an adult's weight. About 70 percent of it sits inside the cells, which is only 35 to 40 percent of body weight: mind the denominator. After 60, body water falls to about 45 percent; infants keep more of theirs in the ECF, which drains fastest. Intake and output each run about 2,600 mL a day, judged over two to three days. Osmosis moves water toward more particles for free; active transport pushes a solute uphill with ATP. Hydrostatic pressure pushes fluid out of a capillary and albumin pulls it back, so a low albumin lets fluid leak into the tissues while the vessels run short. ADH keeps water; aldosterone keeps sodium and dumps potassium.

Part 2 · Ch 41

Recap: Fluid Volume

Five volume words, sorted by sodium. Hypovolemia loses salt and water together, so sodium stays normal; dehydration loses water alone, so sodium rises. Hypervolemia keeps salt and water; overhydration gains water alone, so sodium falls. Third-spaced fluid has not left the body, so the patient looks dry while the weight holds steady: edema shows where fluid is, not how much the vessels hold. On the stick figure, wet runs high and full: a bounding pulse, engorged neck veins, crackles. Dry runs low and empty, and a fast, weak pulse is the earliest sign. The hematocrit rises when dry and falls when wet. Daily weight is the most accurate measure, a kilogram being about a litre. Decide wet or dry first, then read the sodium. Dry and wet care mirror each other: the dry patient gets fall precautions, oral then isotonic fluids and a close watch for overload during replacement; the wet patient gets Fowler’s, oxygen as ordered, fluid and sodium limits, diuretics with a potassium check, and turning every 2 hours. Third spacing is nursed as a deficit even though the weight holds. Assistive personnel measure, weigh and report; the nurse assesses, interprets and teaches.

Part 3 · Ch 41

Recap: Electrolytes

Learn the seven course normals: sodium 135 to 145, potassium 3.5 to 5.0, magnesium 1.3 to 2.3, chloride 97 to 107 and serum bicarbonate 25 to 29, in milliequivalents; calcium 8.6 to 10.2 and phosphorus 2.5 to 4.5, in milligrams per decilitre. Sodium is chief of the ECF, potassium of the ICF, and calcium is the most abundant in the body. Four patterns carry the grid: potassium threatens the heart, sodium the brain, low calcium and magnesium are twitchy, high ones sluggish. The kidneys cannot conserve potassium; loop diuretics lower it, while spironolactone, ACE inhibitors and renal failure raise it. Rank two abnormal values by the organ threatened, not by distance from normal. Every imbalance shares one care pattern: watch the heart for potassium, calcium and magnesium, the brain for sodium, and breathing and reflexes for magnesium and calcium. Weakness means fall precautions; a very low sodium, calcium or magnesium means seizure precautions; a high magnesium means calcium gluconate within reach. If an infusion produces signs of its own excess, stop it; don’t slow it.

Part 4 · Ch 41

Recap: Acid–Base

Blood pH lives between 7.35 and 7.45, set by a ratio of about 20 parts bicarbonate to 1 part carbonic acid. Buffers act first, and the carbonic acid–bicarbonate system takes up as much as 90 percent of the hydrogen ions; the lungs follow in minutes and the kidneys in hours to days. Read a gas in four steps: the pH, the carbon dioxide, the bicarbonate, then whichever matches the pH names the imbalance; a number reading the other way is the rescue. Vomiting loses acid, so alkalosis; diarrhea loses bicarbonate, so acidosis. In acidosis potassium leaves the cells, so the serum level rises. Bicarbonate is 25 to 29 on an electrolyte panel and 22 to 26 on a gas. Respiratory acidosis care is ventilation first: sit up, cough, use the spirometer, give oxygen as ordered, hold sedation and report. Rule out low oxygen before calling fast breathing anxiety, and stay with the patient. Insulin and fluids for an acidosis drive potassium back into the cells, so recheck it.

Part 5 · Ch 41

Recap: IV Therapy and Blood

Normal saline and lactated Ringer's are isotonic and stay in the vessels; LR contains potassium. Half-normal saline is hypotonic and swells cells; D10W is hypertonic and shrinks them. D5W is isotonic in the bag but acts hypotonic once its sugar is used, so keep it from a swelling brain. Choose a forearm vein, start distal and avoid the elbow crease. Read a site by temperature: cool and pale is a leak, warm and red is inflammation. Every complication starts by stopping the cause: for air embolism, clamp before positioning. Potassium is never pushed and never runs faster than 10 mEq an hour. For blood, two people check, prime with saline only, and stay for 15 minutes; at any reaction, stop, run saline through new tubing, stay, notify, send the bag and samples, and document. Assess the site and the patient, not the pump. Assistive personnel report IV complaints and record intake and output; they never start, adjust, flush or discontinue an IV.

Part 6 · Ch 38

Recap: Urine and Testing

Urine runs from kidney to ureter to bladder to urethra, and the short female urethra invites infection. The urge to void begins at 150 to 250 mL. Report adult output below 30 mL an hour; oliguria is under 400 mL a day and anuria under 50. Normal specific gravity is 1.010 to 1.025 and pH 4.6 to 8.2, but glucose and protein raise the gravity, and dilute urine reassures only when there is plenty of it. A clean-catch is clean, not sterile; a sterile specimen comes from a straight catheter or the sampling port, never the bag. For a 24-hour test, discard the first void and keep the last. A post-void residual over 100 mL means the bladder is not emptying. A urinary assessment runs ask, inspect, palpate or percuss, then measure: ask about the last void before you touch the bladder. Deep palpation of the kidneys belongs to an advanced practitioner; the nurse inspects the meatus, the skin and the urine and checks the bladder. Empty an indwelling bag at least every 8 hours, and weigh pads at 1 gram to 1 millilitre.

Part 7 · Ch 38

Recap: Urinary Problems and Nursing Care

Name incontinence by mechanism. Stress leaks with effort; urge cannot make it in time; overflow dribbles from a bladder that never empties; functional has a working tract and a broken path to the toilet; reflex has no sensation of fullness; transient has a treatable cause. Check emptying before you name the type, and never use an indwelling catheter for incontinence alone. For retention, scan, try nursing measures, then a straight catheter by order. In an older adult, new confusion may be the only sign of a UTI. Most CAUTIs start after placement: avoid the catheter, or remove it early. Advance 2 to 3 inches past the first urine in a female and to the Y in a male before inflating. Every urinary problem has the same five steps of care: assess, act, teach, delegate, evaluate. Urinary function can be the problem itself or the cause of another, such as anxiety or a risk for infection. Assistive personnel measure intake and output, give perineal and catheter care and run toileting schedules; inserting catheters, assessing and teaching stay with the nurse. Never take a blood pressure, start an IV or draw blood in a dialysis access arm.

What this is. A student-made study aid for NUR 110, not an official course document. It was built from the course's Chapter 41 deck, its exam and electrolyte-imbalance worksheets, the fluid-balance stick figure, its outlines and skills-lab catheter checklists, Taylor, Lynn & Bartlett, Fundamentals of Nursing, 10th ed., ATI Engage Fundamentals and Skills Modules, and the course's Lippincott resources. All wording is paraphrased; the deck, the textbook, ATI and Lippincott belong to their publishers and instructors, and a short phrase is quoted only where the exact wording is the point, cited to its source.

Which source wins. What is said in class outranks the instructor's own worksheets and checklists, which outrank the Chapter 41 deck, then Taylor, then ATI and Lippincott. The governing figure is printed at full weight and any other in small grey type.

Coverage and policy change by term; check your course site for anything time-sensitive.