Exam 4 Study Guide
Coverage: Taylor 10e Chapter 41, Fluid, Electrolyte, and Acid–Base Balance, including IV
therapy and blood transfusion, and Chapter 38, Urinary Elimination, bladder only. The guide is
ordered by concept, not by chapter: Parts 1–5 are Chapter 41, Parts 6–7 are Chapter 38.
Not on this exam: Chapter 31, Perioperative Nursing, which is taught after the exam, and bowel
elimination (Chapter 39).
Format: probably taken online and proctored, as earlier exams were; item types are not published.
About the blue boxes. Until the instructor's lecture slides are posted, a blue box marks
one of four things: the instructor's own worksheets (the exam worksheet's bold line and its questions, the
electrolyte-imbalance grid, the fluid-balance stick figure); an item the Chapter 41 deck marks or embeds (its
asterisk and its five built-in questions); one of the Chapter 38 deck's five review questions; or the highlighted
skills-lab line that makes catheter insertion
testable.
For exam dates and deadlines, see the semester calendar. Nothing in this guide is tied to a particular term.
What This Edition Covers
The seven parts, in the order the ideas build on each other:
| Part | Title | Chapter |
|---|---|---|
| 1 | Where Body Water Lives, and How It Moves | 41 |
| 2 | Fluid Volume: Too Little, Too Much, in the Wrong Place | 41 |
| 3 | Electrolytes: the Seven Numbers and What Happens When They Move | 41 |
| 4 | Acid–Base: Buffers, Lungs, Kidneys, and the Four Imbalances | 41 |
| 5 | IV Therapy and Blood | 41 |
| 6 | Urine: How It Is Made, What Normal Looks Like, How You Test It | 38 |
| 7 | Urinary Problems and Nursing Care | 38 |
How to Use This Guide
| Time | Route |
|---|---|
| 20 minutes | The Rapid Review: the numbers to know cold and the easy-to-confuse pairs. |
| An hour | The whole Rapid Review, then every section flagged top-weighted topic beside its title. They cover six areas: electrolyte imbalances, fluid-volume imbalances, IV solutions, IV complications, urinary problems and catheters. |
| The whole guide | Parts 1 to 7 in order, each ending with four scenario questions. Then the Summary Recap, then the practice quiz and practice exam: they are the real question bank. |
What the course says you must be able to do
The exam is written against the course outline's objectives. Chapter 41's two lecture weeks share one list of seven; the skills-lab rows are the ones an exam item can reach.
Which part covers each objective. Twenty-one rows, folded shut; open it to check that nothing in the outline is missing.
| # | Chapter | Objective, in short | Where it is covered |
|---|---|---|---|
| W9·1 | 41 | Composition of the major body-fluid compartments. | Part 1, Two compartments, and three spaces outside the cells |
| W9·2 | 41 | Compare diffusion, osmosis and filtration, and hydrostatic, osmotic and oncotic pressure. | Part 1, Four ways across a membrane; Push, pull and albumin · Part 2, Third spacing and edema |
| W9·3 | 41 | Pathophysiology and signs of sodium, potassium, calcium, magnesium and phosphorus imbalances. | Part 3, The imbalance grid; Potassium and sodium; Calcium, magnesium and phosphorus |
| W9·4 | 41 | Normal and abnormal lab and diagnostic findings in fluid and electrolyte balance. | Part 3, Seven numbers, seven jobs · Part 2, Measuring fluid status · Part 4, Reading a gas · Part 6, Normal versus abnormal urine |
| W9·5 | 41 | Tonicities, purposes and complications of IV solutions. | Part 1, Tonicity · Part 5, Solutions by tonicity; IV complications |
| W9·6 | 41 | Nursing management of fluid and electrolyte imbalances. | Part 2, Problems, outcomes and interventions · Part 3, The imbalance grid · Part 5 |
| W9·7 | 41 | The nursing process for fluid and electrolyte imbalances. | Part 2, Problems, outcomes and interventions |
| Topic | 41 | Regulatory mechanisms; the buffer, respiratory and renal defences of acid–base balance (topic bullets W9·T6–T7). | Part 1, Holding the balance · Part 4, Three defences, fastest first |
| W11·1 | 38 | The physiology of urination. | Part 6, The route, and how voiding works |
| W11·2 | 38 | Factors that influence urinary elimination and patterns. | Part 6, What changes voiding |
| W11·3 | 38 | Normal versus abnormal characteristics and constituents of urine. | Part 6, Normal versus abnormal urine; The eight terms |
| W11·4 | 38 | Causes and effects of incontinence, retention, urinary tract infection and kidney stones. | Part 7, Incontinence; Retention; UTI and CAUTI; Kidney stones |
| W11·5 | 38 | Urinary diversions. | Part 7, Urinary diversions |
| W11·6 | 38 | Nursing management of altered urinary elimination. | Part 7, Promoting voiding and continence; Catheters |
| W11·7 | 38 | The nursing process for altered urinary elimination. | Part 7, Writing the problem statements |
| L6·2 | 41 | skills lab Tell IV-fluid tonicities apart, and read IV labels correctly. | Part 5, Solutions by tonicity |
| L6·3 | 41 | skills lab Monitor IV sites and recognise IV complications. | Part 5, IV complications |
| L6·10 | 41 | skills lab IV safety: flow rates, drops per minute versus milliliters per hour, pumps, disposal of equipment. | Part 5, Running an infusion |
| L9·2 | 38 | skills lab Insert an indwelling and a straight catheter on a male and a female; the tested skills are 37-5 / 37-6 in the lab outline (9th-edition numbering; 38-2 / 38-3 in Taylor 10e). | Part 7, Inserting an indwelling catheter, female and male |
| L9·3 | 38 | skills lab Assess for bladder distention; discuss the bladder scanner. | Part 6, The bladder scanner and the post-void residual |
| L9·4 | 38 | skills lab Device care, I&O, specimens and cultures, removal. | Part 6, Collecting urine specimens · Part 7, Caring for the catheter, and taking it out |
Where Body Water Lives, and How It Moves
Where the body keeps its water, how much comes in and goes out each day, the four ways water and solutes cross a membrane, the pressures that keep fluid in a vessel, the hormones that hold the balance, and what tonicity does to a cell.
Two compartments, and three spaces outside the cells
Water is the body's most important nutrient. It does six jobs: it carries nutrients, wastes, hormones and blood cells; supports cell metabolism; dissolves electrolytes and nonelectrolytes; keeps body temperature normal; aids digestion and elimination; and lubricates tissue.
Two barriers divide it. The cell membrane separates the fluid inside cells from everything outside them, and the capillary wall separates the blood from the fluid around the cells. That gives two compartments, and the outer one has three spaces.
| Compartment | Where it is | Share of body water | Share of adult body weight |
|---|---|---|---|
| ICF (intracellular) | Inside the cells | ~70% | 35–40% |
| ECF (extracellular) | Outside the cells: the three spaces below | ~30% | 15–20% |
| · Interstitial | Around the cells, including lymph | 10–15% | |
| · Intravascular | Plasma, inside the vessels | 5% | |
| · Transcellular | CSF; pericardial, pleural, synovial and intraocular fluid; sweat; digestive secretions | small | |
| Total body water | — | 100% | 50–60% |
Age, sex and fat
Fat holds little water and muscle holds a lot, so body water varies with build and age. Three groups differ from the healthy adult's 50–60%:
| Who | Body water | Why it matters |
|---|---|---|
| Infant | Considerably more water, and more of it in the ECF | ECF is lost more easily than ICF, so infants slide into a deficit fast. |
| Women; people with obesity | Less | More of their weight is fat. |
| Over 60 | About 45% of body weight | More fat, less muscle: a smaller reserve (box below). |
After age 60, total body water falls to about 45% of body weight, and that raises the risk of fluid imbalance in the older adult. Expect an age-based risk item to turn on it.
Both ends of life are at risk, for opposite reasons. The infant has more water but keeps a large share in the compartment that drains fastest. The older adult has less water in reserve, and a weaker thirst to warn them.
In and out: about 2,600 mL a day
Water comes from three sources: drinks (the largest), food, and metabolism (water released as food is oxidized). In a healthy adult, intake and output both average about 2,600 mL a day, and urine output roughly tracks what is drunk. Output leaves by two kinds of loss, and one tempting third category is not a loss at all.
| Kind | Hook | Examples |
|---|---|---|
| Sensible | You can see or measure it | Urine (the largest output, about 1,500 mL a day), feces, wound drainage |
| Insensible | It evaporates unseen | Water vapour breathed out; evaporation through the skin |
| Not one of these: third-spaced fluid | It is not lost, only unavailable | Fluid trapped in a body space, such as ascites. It leaves the circulation without leaving the body, so weight does not fall (Part 2). |
Sources sort sweat differently: Taylor counts evaporation and perspiration through the skin as insensible and visible sweat as sensible; ATI lists sweat as sensible. Breath and unseen skin evaporation are insensible in every source.
Judge the balance over 2–3 days, not one shift: a single day can run short or long and mean nothing. Fever raises insensible loss: a temperature of 101–103 °F (38.3–39.4 °C) adds at least 500 mL a day, and anything above 103 °F adds at least 1,000 mL.
The deck and Taylor split the 2,600 differently between drinks and food, and among skin, lungs and gut. Only the totals and urine's 1,500 mL agree, so those are the figures to know.
Four ways across a membrane
Body fluid is solutes (electrolytes, and nonelectrolytes such as glucose and urea) dissolved in a solvent, water. Four processes move them. The exam worksheet matches each process to one phrase, shown in the last column.
| Process | What moves | Which way | Energy | Example | Worksheet phrase |
|---|---|---|---|---|---|
| Osmosis | Water | From fewer solute particles toward more | None | The main way body water moves between compartments | Lower → higher concentration |
| Diffusion | Solutes and gases | High concentration to low (“downhill”) | None | O₂ and CO₂ across the alveolar wall (Exam 3) | Gases move by this |
| Active transport | Solutes | Low concentration to high (“uphill”) | ATP | The sodium–potassium pump, which keeps potassium in the cells and sodium out | Requires ATP |
| Capillary filtration | Fluid and small solutes | Higher pressure to lower | Blood pressure | Fluid pushed out at the arterial end of a capillary | Fluid-permeable membrane |
Push, pull and albumin: why fluid leaves a capillary
Osmotic pressure is the pull that dissolved particles exert on water. When the particles are plasma proteins, chiefly albumin, which are too large to leave the vessel, it is called oncotic (colloid osmotic) pressure. Two pressures oppose each other across every capillary wall.
| Pressure | Push or pull | Made by | Wins at | Result |
|---|---|---|---|---|
| Hydrostatic | Push, out of the vessel | Blood pressing on the vessel wall | Arterial end | Filtration: fluid moves into the interstitium |
| Oncotic (colloid osmotic) | Pull, into the vessel | Plasma proteins, mainly albumin | Venous end | Reabsorption: fluid returns to the blood |
When the push outruns the pull, or the wall itself leaks, fluid collects in the tissues as edema. The four ways that happens, and third spacing, are in Part 2 (Third spacing and edema); the pitting scale was taught in NUR 120 (Exams 2 and 4).
The same pressures run in reverse when blood volume falls. Fluid moves from the interstitium into the vessels; the interstitium is left concentrated and pulls water out of the cells. The body protects the circulating volume first, at the expense of the tissues and then the cells. That is why the early signs of a deficit show in the mouth, the skin and the pulse before the blood pressure falls (Part 2).
Holding the balance: thirst, three hormones, nine organs
Thirst is the main control on intake. It is run from the hypothalamus and triggered by cells losing water (concentrated ECF draws water out of them) and by falling blood volume. It weakens with age, so older adults are offered fluid at regular intervals rather than when they ask. Output is controlled by three hormones, all acting on the kidney.
| Hormone | Where it comes from, and what releases it | What the kidney does | Net effect |
|---|---|---|---|
| ADH (antidiuretic hormone, vasopressin) | Made in the hypothalamus; stored and released by the posterior pituitary. Released when the ECF is concentrated (high sodium, high glucose) or volume is low; switched off when the ECF is dilute or volume is high. | Keeps water | Less urine, more concentrated; the ECF is diluted back toward normal |
| Aldosterone (the end of RAAS) | Poor kidney perfusion → renin → angiotensin I → (ACE) → angiotensin II, which constricts vessels and makes the adrenal glands release aldosterone. | Keeps sodium (and water with it); excretes potassium | Volume and blood pressure rise; potassium falls |
| ANP (atrial natriuretic peptide) | The atria, when stretched by a high volume. | Excretes sodium and water | Volume falls: the opposite of aldosterone |
Taylor does not name ANP, so don't expect an item keyed on it.
The exam worksheet pairs nine organs with one phrase each, as below.
| Organ | Worksheet phrase | What it means |
|---|---|---|
| Kidneys | Regulate the ECF | Adjust its volume, osmolality, electrolytes and pH; excrete wastes |
| Heart and vessels | Circulate nutrients and water | Perfuse the kidneys; stretch receptors sense low volume and trigger retention |
| Lungs | Promptly correct acid–base | Blow off or keep CO₂ (Part 4); lose about 300 mL of water a day as vapour |
| Adrenal glands | Regulate with aldosterone | Keep sodium and water, excrete potassium |
| Pituitary | Stores and releases ADH | ADH is made in the hypothalamus |
| Nervous system | Senses intracellular dehydration | Triggers thirst; osmoreceptors signal the pituitary to release or hold back ADH |
| GI tract | Absorbs water and nutrients | Also a route of loss, through vomiting and diarrhea |
| Parathyroids | Regulate calcium and phosphate | PTH raises calcium and lowers phosphate (Part 3) |
| Thyroid | Increases blood flow with thyroxine | More blood flow to the kidneys, so more urine |
Tonicity: what a solution does to a cell
Osmolality is the concentration of particles in a fluid. Tonicity compares a solution's particles with plasma's, and so predicts which way water will move by osmosis. Water always moves toward more particles. There are three tonicities:
| Tonicity | Particles compared with plasma | Water moves | The cell |
|---|---|---|---|
| Hypotonic | Fewer | Out of the vessel, into the cells | Swells, and may burst |
| Isotonic | The same | No net shift; the fluid stays in the vessel | Unchanged |
| Hypertonic | More | Out of the cells, into the vessel | Shrinks |
The item: true or false, a hypertonic solution, being more concentrated, draws water out of the cells into the intravascular space, and the cells shrink.
True. The solution has more particles than the cell, so water leaves the cell by osmosis and is drawn into the vessels. The cell shrinks while the circulating volume grows.
Not one of these: isotonic exercise (Exam 2) is constant muscle tension with movement. It shares the word with an isotonic solution, not the meaning. Which IV bags fall in each group, and the D5W exception, are in Part 5.
Taylor writes osmolality and puts plasma at about 280 mOsm/L; the deck writes osmolarity. The serum osmolality lab value is in Part 2.
Fluid Volume: Too Little, Too Much, in the Wrong Place
The five volume words, why edema forms, the wet/dry stick figure, how to measure fluid status, the three patient shapes, and the care plan.
Five volume words, sorted by what was lost or gained top-weighted topic
Fluid problems come in two kinds: volume (too little or too much) and distribution (a normal amount in the wrong place). There are five words to keep apart: two deficits, two excesses and one shift. Within each pair, the deciding feature is whether sodium moved in step with the water.
| Condition | What changed | Serum Na | Typical causes | Hook |
|---|---|---|---|---|
| Hypovolemia (FVD) | Water and electrolytes lost together, in near-normal proportion | Normal | Hemorrhage, vomiting, diarrhea, GI suction, burns, heavy sweating | Lost salt water |
| Dehydration | Water lost out of proportion to solute | High; serum osmolality up | No access to fluids, a patient who cannot sense or ask for a drink, fever, burns, hyperventilation | Lost water, kept salt |
| Hypervolemia (FVE) | Water and sodium retained together in the ECF | Usually normal | Heart failure, renal failure, excess sodium, too much IV fluid | Kept salt water |
| Overhydration | Water gained in excess of sodium (water intoxication) | Low | More water taken in than the kidneys can clear (Part 3, Potassium and sodium) | Gained water, not salt |
| Third-space shift | Fluid moves out of the vessels into a space where the body cannot use it | Varies | Burns, bowel obstruction, liver or heart failure, crush injury, surgery, peritonitis | Not lost, not available |
| Not one of these: edema | A location, not a volume: excess fluid in the interstitium. It comes with FVE and with third spacing, so it cannot tell you which. | |||
| Not one of these: interstitial-to-plasma shift | The body's rescue in FVD, not a disorder of its own (Part 1, Push, pull and albumin). | |||
Taylor files both deficits under “fluid volume deficit” and does not define dehydration separately. The deck and ATI treat dehydration and hypovolemia as two disorders, so a stem that pairs “dehydration” with a high sodium means the water-only loss.
Third spacing and edema: fluid in the wrong place
Edema is excess ECF in the interstitium. Fluid leaves a capillary when the hydrostatic push out beats the oncotic pull back in (Part 1, Push, pull and albumin). There are four ways the balance tips.
| Mechanism | What happens | Examples |
|---|---|---|
| More push: ↑ hydrostatic pressure | Blood backs up or volume rises, so more fluid is forced out of the capillary | Heart failure, renal failure, excess IV fluid |
| Less pull: ↓ oncotic pressure | Too little albumin to hold water in the vessels | Burns, liver disease, malnutrition |
| Leaky walls: ↑ capillary permeability | Protein and fluid escape together, so the pull is lost as well | Trauma, burns, crush injury |
| Sodium retention | The kidneys keep sodium, water follows it, and the ECF expands until it spills into the tissues | Heart failure, renal failure |
Third spacing. Fluid is trapped in the pleural, peritoneal (ascites) or pericardial space, the joints, the bowel or the interstitium. It has not left the body, but the circulation cannot use it, so the patient shows a deficit: tachycardia, falling BP, low urine output. The causes are low albumin, too much volume (excess IV fluid, kidney failure), high capillary pressure (heart failure), leaky capillaries (trauma, burns) and low serum sodium. The weight does not fall and the loss cannot be measured on the I&O.
Where edema shows. In the lower legs of a patient who walks and the presacral area of a patient in bed. It is not visible until 5–10 lb of fluid has been retained, which is why the scale catches it first. Edema is localized (a thrombophlebitis) or generalized (heart failure, cirrhosis, nephrotic syndrome). The pitting scale is NUR 120 material (Exams 2 and 4); measuring the limb with a tape at the same marked spot each day is more exact than grading it.
The wet/dry stick figure, head to toe
The assignment is a blank figure: label the signs of hypervolemia (wet) on one side and hypovolemia (dry) on the other. Fill your own in before reading the key; the act of placing each sign on a body part is what makes it stick.
| Area | Wet: hypervolemia (FVE) | Dry: hypovolemia (FVD) |
|---|---|---|
| Brain | Confusion, if the extra water dilutes sodium | Restlessness; change in mental status |
| Eyes, face | Puffy eyelids | Sunken, soft eyes (a 10% deficit); pinched face; no tears in a child |
| Mouth, tongue | — | Dry where the cheek meets the gum; extra furrows, smaller tongue; cracked lips; thirst |
| Neck | Engorged neck veins | Flat neck veins |
| Lungs | Moist crackles, dyspnea, faster breathing | Usually clear |
| Pulse | Bounding (high amplitude) | Fast and weak; tachycardia is the earliest sign |
| Blood pressure | Up | Down; orthostatic drop of SBP ≥20 or DBP ≥10 mm Hg |
| Temperature | — | Often up |
| Skin | Taut, shiny; pitting edema | Poor turgor; dry |
| Hands | Tight rings, swollen fingers | — |
| Sacrum, abdomen | Presacral edema if in bed; ascites | — |
| Legs, feet | Dependent edema | — |
| Urine | More, if the kidneys work | Scanty, dark, high specific gravity (e.g., 1.035) |
| Weight | Gain over 5% | Sudden loss: 5% in an adult, 10% in an infant; 15% is life-threatening |
| Labs | Hct down (diluted) | Hct up (concentrated); BUN and creatinine up |
Fifteen rows. Most signs split cleanly: wet is full (bounding pulse, distended veins, edema, weight up) and dry is empty (weak pulse, flat veins, weight down). The two that trip people up are the pulse rate and the hematocrit.
Measuring fluid status: what each measure is worth
Assessment rests on four parameters: the history and physical, intake and output, daily weights, and labs. Look hardest at the four risk groups: acute or chronic illness (diabetes, heart failure, renal failure); abnormal losses (vomiting, diarrhea, draining wounds, fistulas); burns, trauma and surgery; and therapies that shift fluid (diuretics, steroids, IV therapy, parenteral nutrition).
Daily weight, the most accurate measure
1 kg ≈ 1 L ≈ 2.2 lb (NUR 120 Exam 4). Weigh at the same time, on the same scale, in similar clothing, best in the morning after voiding and before breakfast. Weight is more accurate than I&O, and in an infant it is the most reliable indicator of fluid balance. A rapid loss of 5% in an adult or 10% in an infant signals FVD; a gain over 5% signals FVE. The lb↔kg arithmetic is in the Dosage Calculation guide.
Intake and output: what counts
| Record | Includes | Easy to miss |
|---|---|---|
| Intake | Oral fluids; IV fluids, including IVPB; tube feedings and flushes (NG, G, J); ice chips at about half their volume; gelatin, popsicles, water ice | The IVPB and the flushes, because they feel like medication |
| Output | Urine; vomitus; diarrhea; fistula and suction drainage; note heavy sweating and wound drainage | Liquid stool and drain output. Diapers are weighed: 1 g = 1 mL |
Tell the patient, the family and every caregiver that everything is measured. Normal turnover is about 2,600 mL in and out a day (Part 1, In and out), and the balance is judged over 2–3 days, not one shift. Intake well below output signals a risk of FVD; well above, a risk of FVE. Refresher (Dosage Calculation guide): report the balance with its sign, e.g. −350 mL; assistive personnel may measure, but the nurse interprets.
Urine output and specific gravity are fluid-status measures too; the report threshold, the SG range and how to read SG against the volume are in Part 6 (Normal versus abnormal urine).
Bedside technique, and what fools you
| Finding | How to check it | What fools you |
|---|---|---|
| Pulse | Rate and amplitude | The rate alone (the exceptions above) |
| Orthostatic BP | Lying, sitting, then standing; a sustained drop of SBP ≥20 or DBP ≥10 mm Hg is postural hypotension | A single seated reading misses it |
| Skin turgor | Over the sternum, inner thigh or forehead; the abdomen or medial thigh in a child | Normally reduced with age; severe malnutrition depresses it in infants without any fluid loss |
| Tongue | One lengthwise furrow is normal; more furrows and a smaller tongue mean FVD | Nothing: tongue turgor does not change with age |
| Mouth | Dryness where the cheek meets the gum | Mouth-breathing and dry air dry the mouth too |
| Tears, saliva | Absent in a child means FVD | Both decrease normally with age |
NUR 120 taught the back of the hand as the turgor site. For fluid status, use the sternum, inner thigh or forehead instead: hand skin loses elasticity with age, so it tents in older adults who are not dry. In an older adult, confirm with the tongue, the mucosa and an orthostatic BP.
Labs
| Lab | Dry (FVD) | Wet (FVE) | Note |
|---|---|---|---|
| Hematocrit | Up | Down | Hemoconcentration vs dilution; also down after bleeding or hemolysis (Exam 3) |
| BUN | Up | — | Also up in renal impairment, heart failure, burns, DKA |
| Creatinine | Up | — | More kidney-specific than BUN |
| BUN:creatinine | Over 20:1 | — | Low blood flow to the kidneys; both rising together in proportion points to kidney damage instead |
| Serum osmolality | Up in dehydration | Down in overhydration | 285–295 mOsm/kg; the lab most used to judge fluid status. It tracks the sodium, so it stays normal in a same-proportion loss or gain |
| Serum sodium | Up in dehydration | Down in overhydration | Normal in pure hypovolemia or hypervolemia (Part 3, Potassium and sodium) |
Taylor uses about 280 mOsm as the reference point for plasma.
Three patients, three shapes
The unit's three video cases are the shapes scenario items come in: an excess with electrolytes piling up, an excess from a failing pump, and a deficit in a child.
| Shape | What drives it | What you watch | The trap inside it |
|---|---|---|---|
| Chronic renal failure: FVE | The kidneys cannot excrete water, sodium, potassium, magnesium or phosphate; calcium falls as phosphate rises | Weight, I&O, lungs; potassium (Part 3); low output with low, fixed SG | A fluid bolus for low output overloads a kidney that cannot respond; question lactated Ringer's, which contains K (Part 5) |
| Heart failure: FVE | A weak pump raises hydrostatic pressure, and the kidneys retain sodium and water | Daily weight, crackles, neck veins, edema, I&O | “Encourage fluids” is wrong here; the HF picture itself is NUR 120 Exam 4 material |
| Pediatric dehydration: FVD | More ECF and surface area, no way to fetch a drink; vomiting and diarrhea | Daily weight; tears, eyes, urine output; sodium, because hypernatremia can leave permanent brain damage in a child | A 10% loss in an infant matches a 5% loss in an adult; oral rehydration comes first if the child can drink |
Problems, outcomes and interventions
There are three NANDA labels: Excess fluid volume, Deficient fluid volume and Risk for deficient fluid volume. Items may instead use the three newer problem labels below, each with its own cues.
| Problem label | Cues that point to it |
|---|---|
| Fluid overload | Tight wedding ring, clothes that no longer fit, dyspnea on exertion, pitting edema, taut shiny skin, adventitious breath sounds, BP up, a 10-lb (4.5-kg) gain in a month |
| Fluid imbalance | Mental status change, temperature and pulse up, BP down, dry mucosa, cracked lips, furrowed tongue, poor turgor, scanty dark urine, a sudden 5-lb (2–3 kg) loss |
| Impaired fluid intake | Limits drinks to avoid trips to the bathroom; SG 1.035; intake below 1,500 mL a day |
Exam 3's red rule still applies: no medical diagnosis and no symptom after R/T. The three expected outcomes: intake and output balanced at about 2,500 mL a day, averaged over 3 days (a round goal near the ~2,600 mL turnover); urine SG within 1.010–1.025; and the patient practises self-care that keeps the balance (for example, losing 4 lb a month after cutting sodium).
Nursing care, dry versus wet
One table for both problems, in the order you work: assess, act, teach, delegate, evaluate. Most rows are mirror images. The how-to for fluids, diet and diuretics is in the table after it.
| Step | Dry: deficient fluid volume (hypovolemia) | Wet: excess fluid volume (hypervolemia) |
|---|---|---|
| Assess and monitor | Heart rate first (the earliest sign), then orthostatic BP and pulse: lying, sitting, standing. LOC: restlessness comes early. Mucosa and turgor. Hourly urine output when the deficit is acute; report under 30 mL/h (Part 6). SG, Hct, BUN, sodium. | BP and pulse amplitude. Lung sounds, RR and SpO₂ at every assessment: crackles mean the fluid has reached the lungs. Neck veins; edema at the ankles, or the sacrum in bed. LOC, if sodium is diluted. Hct, sodium. |
| Monitor, both | Strict I&O and a daily weight by the technique above. A change of 1 kg is 1 L. | |
| Position, safety, oxygen | Fall precautions for orthostasis: bed low, call light and a drink within reach; the patient dangles at the bedside, rises slowly and walks with assistance. | Semi- or high-Fowler's for dyspnea, so the lungs can expand; oxygen as ordered. |
| Fluids and diet | Oral first if the patient can drink (Oral rehydration, below); otherwise isotonic IV fluid as ordered (Part 5). While replacing, watch for overload: lungs, neck veins, output. A water-only dehydration is corrected slowly. | Fluid and sodium restriction as ordered (Restrict fluids and Diet, below). IV fluids and flushes count against the allowance. |
| Medications | Treat the cause as ordered. A diuretic can be the cause: report dizziness, a falling BP or low output in a patient taking one. | Diuretics as ordered, then weight, I&O and potassium, which loop diuretics waste (Diuretics, below). |
| Skin and mouth | Frequent mouth care; lubricate cracked lips; recheck turgor and mucosa. | Stretched skin breaks down easily: turn at least every 2 hours, elevate swollen limbs, protect taut skin, and avoid tape and tight clothing. Thirst on a restriction: Restrict fluids, below. |
| Teach | Rise in stages. Drink to a goal tied to the day's routine. Avoid casual laxatives, enemas and “water pills” (below). | Weigh every morning at home and report a rapid gain. Keep to the fluid and sodium limits, taught to whoever cooks. |
| Delegate | Assistive personnel may measure and record I&O, weigh, take routine vital signs on a stable patient, offer fluids within the order, help the patient walk and turn, and report to the RN (dizziness, low output, shortness of breath). The RN keeps assessment (lung sounds, interpreting orthostatic readings), IV fluids, interpreting the I&O and weights, teaching and evaluation. | |
| Evaluate | Heart rate down, no orthostatic drop, output 30 mL/h or more, SG back in range, LOC clear, weight back up. | Weight down, lungs clearing, SpO₂ up, less edema, intake and output nearer even. |
| Age changes the action | Infant: the daily weight is the best indicator; weigh diapers (1 g = 1 mL); oral rehydration by syringe (below). Older adult: weak thirst, so offer fluids at set times; turgor misleads, so use the tongue, mucosa and orthostatic readings; orthostasis makes a fall likelier. | Older adult: a heart and kidneys with little reserve, so IV replacement can tip a dry patient wet. Listen to the lungs often and report a wet cough, dyspnea or new edema. Child: infusions by pump (Part 5). |
Third spacing. Nurse it as a deficit in the vessels: heart rate, BP, LOC, hourly output and fluids as ordered. Weight and I&O miss the loss (above), so they cannot show improvement either. When the cause settles and the fluid returns to the vessels, the risk flips to overload, so keep listening to the lungs.
| Intervention | How |
|---|---|
| Increase fluids | Split the 24-hour order into shift totals, heaviest by day (e.g., 1,200 day, 900 evening, 300 night for 2,400 mL), so the night is not spent voiding. Offer variety at the temperature the patient likes; gelatin and popsicles for children; keep a drink within reach; count tube flushes. |
| Restrict fluids | Set shift totals so one shift cannot use up the allowance. Use small cups; offer fluids every 1–2 hours between meals; avoid dry, salty or sweet foods; keep other drinks out of sight. For thirst: frequent mouth care, lip lubricant, sugar-free gum or hard candy. |
| Oral rehydration | First-line for anyone who can drink, at any age, if renal function is adequate: an electrolyte-and-glucose solution, with I&O recorded hourly or per protocol and SG watched. Child: check the swallow; 5 mL every 5–10 minutes by syringe into the side of the mouth, more as tolerated; 50 mL/kg over at least 4 hours for mild dehydration, 100 mL/kg for moderate; back to a normal diet once rehydrated and not vomiting. |
| Diet | FVD: high-water foods such as citrus, melons and celery. FVE: limit sodium. Give a written list and use teach-back with whoever cooks. |
| Diuretics | Risk of FVD and electrolyte loss, especially potassium; monitor I&O, daily weight and electrolytes (Exam 1 drug table; Part 3, Potassium and sodium). |
Teach the patient to report four things: a rapid weight gain or loss; swollen fingers, feet or ankles, or puffy eyelids; scanty or very heavy urine; and muscle weakness or a change in skin sensation. Warn against casual use of enemas, laxatives, antacids and over-the-counter “water pills”.
Evaluate with five questions: is intake about equal to output; do the weight and the I&O agree; are the abnormal losses responding; are the signs resolving without new ones caused by the therapy; can the patient manage self-care?
Electrolytes: the Seven Numbers and What Happens When They Move
The seven course normals and the job each electrolyte does, where each comes from and how it is lost, the imbalance grid from the course worksheet, how to rank two abnormal values, the nursing care every imbalance shares, and the drug, acid–base and immobility links that move the numbers.
Seven numbers, seven jobs
An electrolyte breaks apart in water into charged particles, ions. Cations are positive (Na⁺, K⁺, Ca²⁺, Mg²⁺, H⁺); anions are negative (Cl⁻, HCO₃⁻, phosphate). In health total cations equal total anions, and both are counted in milliequivalents (mEq), a unit of chemical activity. A nonelectrolyte stays intact in solution; glucose and urea are the two examples to know.
True or false: molecules in the body's compounds that stay intact are called electrolytes. False. Molecules that stay intact are nonelectrolytes; electrolytes are the ones that separate into ions.
Splits into charged ions: electrolyte. Stays whole (glucose, urea): nonelectrolyte.
Items match each electrolyte to its range and to its job, so learn the range and the job together. Two of the seven are in mg/dL (calcium and phosphorus); the other five are in mEq/L.
| Electrolyte | Normal (course) | Lives mainly | Chief job (the worksheet's matching phrase in bold) | Hook |
|---|---|---|---|---|
| Sodium, Na⁺ | 135–145 mEq/L | ECF | Chief electrolyte of the ECF. Controls and regulates the volume of body fluid; nerve and muscle function. | Where sodium goes, water follows. |
| Potassium, K⁺ | 3.5–5.0 mEq/L | ICF | Major cation of the ICF. Regulates cell enzyme activity and water content; carries nerve, cardiac, skeletal-muscle and intestinal impulses. | The heart electrolyte. |
| Calcium, Ca²⁺ | 8.6–10.2 mg/dL | ECF; 99% stored in bone and teeth | Most abundant electrolyte in the body. Clotting, nerve impulses, muscle contraction, bones and teeth. | mg/dL, like phosphorus. |
| Magnesium, Mg²⁺ | 1.3–2.3 mEq/L | ICF (the second ICF cation) | Neuromuscular function. Carbohydrate and protein metabolism; enzyme reactions. | Calcium's partner in nerve and muscle. |
| Chloride, Cl⁻ | 97–107 mEq/L | ECF | Major ECF anion. Holds osmotic pressure with sodium; makes stomach acid (HCl). | Travels with sodium. |
| Bicarbonate, HCO₃⁻ | 25–29 mEq/L | ECF | Anion chemical buffer, the body's primary buffer. | The serum value; an ABG uses a different range (Part 4, Reading a gas). |
| Phosphate (phosphorus) | 2.5–4.5 mg/dL | ICF | Major ICF anion and buffer. ATP and energy; bones; muscle and red-cell function. | Moves opposite to calcium. |
Taylor gives calcium as 8.6–10.5 mg/dL (ionized 4.5–5.6), and its imbalance table uses Mg above 2.1 and Cl below 98 or above 108. ATI uses Na 136–145, Ca 9–10.5 and Mg 1.3–2.1. Use the course numbers.
Inside or outside. Four live mainly in the ECF: sodium, chloride, calcium and bicarbonate. Three live mainly in the ICF: potassium, phosphate and magnesium. That is why a sodium problem shows up as water moving into or out of cells (Part 1, Tonicity).
Where each comes from, how it is lost, and what controls it
Most of the seven arrive in food and leave through the kidneys, the gut or the skin. The food column is what you name when you teach.
| Electrolyte | Food sources | Lost through, or blocked by | Controlled by |
|---|---|---|---|
| Na⁺ | Table salt, processed foods, preservatives: canned soups and vegetables, lunch meats, processed cheese, salty snacks. | GI tract, kidneys, skin (sweat). | Aldosterone keeps it (RAAS); kidneys. |
| K⁺ | Bananas, citrus, apricots, melons, raisins; broccoli, potatoes, lima beans; dried peas and beans, whole grains, milk, meat. | Kidneys (the main exit), stool, sweat, vomit. | Aldosterone excretes it. Shifts between ICF and ECF. |
| Ca²⁺ | Dairy, dried beans, green leafy vegetables, small fish eaten with the bones. | Absorbed only with vitamin D and stomach acid. | PTH raises it; calcitonin lowers it. |
| Mg²⁺ | Leafy greens, nuts, seafood, whole grains, dried peas and beans, cocoa. | Urine (loop diuretics), diarrhea, NG suction. | Kidneys; PTH. |
| Cl⁻ | Salt, processed foods. | Vomiting and gastric suction (it leaves as HCl), with sodium. | Follows sodium; aldosterone. |
| HCO₃⁻ | None to teach. Antacids and sodium bicarbonate add it. | Diarrhea, diuretics. | Kidneys. |
| Phosphate | Meat, poultry, eggs, milk, bread, cereal. | Absorption blocked by antacids containing calcium, magnesium or aluminum. | Kidneys, PTH, vitamin D. |
- Aldosterone keeps sodium (and water) and dumps potassium (Part 1, Holding the balance), so too little aldosterone is a cause of high K.
- The kidneys cannot conserve potassium. A patient who stops eating, vomits or takes a loop diuretic keeps losing it.
- Calcium and phosphate move in opposite directions. When the kidneys fail, phosphate climbs and calcium falls.
The imbalance grid: four patterns before the details top-weighted topic
The worksheet is a blank grid of ten imbalances, low and high for potassium, sodium, calcium, phosphorus and magnesium. Each row prints the cut-off and leaves room for risk factors, manifestations and interventions. The tables in the next two sections are that grid filled in, on the worksheet's own cut-offs. Fill a blank copy from memory first, then check it against them.
| Electrolyte | Hypo- (below) | Hyper- (above) |
|---|---|---|
| Potassium | 3.5 mEq/L | 5.0 mEq/L |
| Sodium | 135 mEq/L | 145 mEq/L |
| Calcium | 8.6 mg/dL | 10.2 mg/dL |
| Phosphorus | 2.5 mg/dL | 4.5 mg/dL |
| Magnesium | 1.3 mEq/L | 2.3 mEq/L |
No chloride row. Learn the grid as patterns first; the details hang on them.
The four patterns.
- Potassium, high or low, threatens the heart. Both directions cause muscle weakness, paresthesias and dysrhythmias. The history tells them apart: renal failure or a KCl infusion points high; vomiting, suction or a loop diuretic points low.
- Sodium, high or low, threatens the brain. Low sodium lets water into cells, which swell; high sodium pulls water out, and they shrink.
- Low calcium and low magnesium are twitchy: tingling, cramps, tetany, hyperactive reflexes, seizures.
- High calcium and high magnesium are sluggish: lethargy, weak or absent reflexes; high magnesium also slows the breathing.
Potassium and sodium: the heart and the brain top-weighted topic
Potassium
| Imbalance | Risk factors | Manifestations | Interventions |
|---|---|---|---|
| Hypokalemia K <3.5 | Vomiting, gastric (NG) suction, diarrhea, loop and thiazide diuretics, alkalosis, poor intake. | Skeletal muscle first: weakness, leg cramps, fatigue, paresthesias. Dysrhythmias. Raises the risk of digoxin toxicity. | K-rich foods. Oral K diluted, after meals. IV K only diluted, by pump (Part 5, IV potassium). Confirm urine output before replacing. Monitor K on diuretics. |
| Hyperkalemia K >5.0 | Renal failure (the commonest), too little aldosterone, KCl and salt substitutes, potassium-sparing diuretics, ACE inhibitors, NSAIDs, heparin. Often present in metabolic acidosis. | Muscle weakness, paresthesias, paralysis. Cardiac irregularities, up to arrest. | Stop every K source; question any K-containing IV, such as LR, and notify. Low-K diet. As ordered: calcium gluconate to protect the heart (it does not lower K), insulin with dextrose (watch the glucose), a K-binding resin, dialysis. |
Why potassium is the dangerous one. Its range is narrow, only 1.5 mEq/L from floor to ceiling, so a small move is a big one. It carries the heart's impulses. The kidneys cannot hold on to it when intake stops, yet they are its main exit, so when they fail it piles up. That is why IV potassium is never given as a bolus: a bolus raises the serum level suddenly and causes a fatal dysrhythmia. Never IV push (NUR 110 Exam 1). How it is diluted, run and capped: Part 5, IV potassium.
Sodium
Which imbalance is a sodium deficit in the ECF caused by a loss of sodium or a gain of water? Hyponatremia. Hypernatremia is a sodium surplus; hypokalemia and hyperkalemia are a potassium deficit and excess.
Low sodium is a ratio: too little salt, or too much water diluting it.
| Imbalance | Risk factors | Manifestations | Interventions |
|---|---|---|---|
| Hyponatremia Na <135 (severe <120) | Sodium loss: vomiting, diarrhea, fistulas, sweating, diuretics (thiazides are the commonest drug cause). Water gain: excess water, too much hypotonic IV fluid, heart failure, cirrhosis. | Water moves into cells, which swell. Lethargy and confusion come first; headache, cramps, weakness. Below 120: twitching, hyperreflexia, seizures, coma. | Treat the volume problem behind it: fluid restriction when water is diluting it; sodium and fluid (NS) when both were lost. Raise it slowly. |
| Hypernatremia Na >145 | Water loss or sodium excess: fluid deprivation; anyone who cannot sense, reach or ask for water (infants, confused or older adults); diarrhea; burns; hyperventilation; prolonged suction; rapid sodium-containing IV. | Cells shrink and the brain is hit hardest: restlessness, agitation, disorientation, hallucinations, seizures. Red, swollen tongue; dry, sticky membranes. Permanent brain damage, especially in children. | Hypotonic IV fluid (0.45% NaCl) lowers it without dropping it too fast. Avoid table salt and processed foods; watch sodium-containing IVs. Offer water on a schedule to anyone who cannot ask; mouth care. |
Calcium, magnesium and phosphorus: the nerve-and-muscle minerals top-weighted topic
Calcium
| Imbalance | Risk factors | Manifestations | Interventions |
|---|---|---|---|
| Hypocalcemia Ca <8.6 mg/dL | Low intake; poor absorption (low vitamin D); low PTH, most often after thyroid or neck surgery; renal failure; low albumin; many transfusions; loop diuretics. | Numbness and tingling of the fingers, toes and around the mouth; cramps; tetany; seizures; laryngeal spasm; dysrhythmias. Chvostek's sign (tap the face just in front of the ear: that side twitches) and Trousseau's sign (a BP cuff held above systolic: the hand goes into spasm). | Calcium and vitamin D foods. IV calcium gluconate as ordered. Check Chvostek and Trousseau. |
| Hypercalcemia Ca >10.2 mg/dL | Cancer and hyperparathyroidism (the two commonest); prolonged bed rest; thiazides; too much vitamin D. | GI first: nausea, vomiting, constipation. Bone pain; excessive urination and thirst; confusion, lethargy, slurred speech; weak reflexes; kidney stones. | NS IV, then a loop diuretic, as ordered. Mobilize, with weight-bearing. Fluids; strain the urine. |
Magnesium
| Imbalance | Risk factors | Manifestations | Interventions |
|---|---|---|---|
| Hypomagnesemia Mg <1.3 | NG suction, diarrhea, chronic alcohol use, loop diuretics, tube feeds or PN, burns, sepsis. | Weakness, tremors, tetany, seizures, hyperactive reflexes, dysrhythmias, mental-status change. The same Chvostek and Trousseau signs. | Mg foods. IV magnesium sulfate is high-alert (second-nurse check); during it, check reflexes and respiratory rate. It is working when restlessness, tremors and seizures settle. Low Mg, low K and low Ca travel together: correct them together. |
| Hypermagnesemia Mg >2.3 | Renal failure (the commonest); Mg-containing antacids and laxatives (milk of magnesia). | Flushing, nausea, weakness, lethargy; hypoactive or absent reflexes; respiratory depression; bradycardia, hypotension, arrest. | Stop Mg products. Calcium gluconate (the antidote) as ordered; dialysis. |
Phosphorus, and chloride in one line
| Imbalance | Risk factors | Manifestations | Interventions |
|---|---|---|---|
| Hypophosphatemia <2.5 mg/dL | Feeding a malnourished patient (refeeding); alcohol withdrawal; DKA; hyperventilation; malabsorption; diuretics. Antacids with Ca, Mg or aluminum block absorption. | Weakness, paresthesias, slurred speech, trouble swallowing, weak respiratory effort, confusion, seizures, coma. | Replace as ordered; phosphate foods; refeed slowly. |
| Hyperphosphatemia >4.5 mg/dL | Kidneys not excreting it (renal failure); low PTH. | Hyperreflexia, anorexia, weakness, dysrhythmias; and, because calcium falls as phosphate rises, low-calcium signs. | Phosphate binders with meals; limit dairy, meat and cola; dialysis. |
Chloride. It follows sodium. Low chloride (vomiting, gastric suction, diuretics, metabolic alkalosis) is twitchy like low calcium; high chloride travels with metabolic acidosis and high sodium. Care follows the partner problem: the sodium row, or the acid–base imbalance (Part 4).
Reading a patient: which electrolyte, and which one first
Read the deciding sign, then check the history for the cause.
| The picture in the stem | Think | Deciding feature |
|---|---|---|
| Leg cramps and weakness after days of furosemide | Low K | Skeletal muscle first, plus a K-wasting drug. |
| Weakness and rhythm changes in renal failure | High K | Same symptoms as low K; the kidneys decide. |
| Confusion and headache in a patient drinking litres of water | Low Na | Brain signs plus water gain. |
| Agitation, red swollen tongue, an older adult who cannot reach water | High Na | Brain signs plus water loss. |
| Tingling around the mouth, a facial twitch when the cheek is tapped | Low Ca (or low Mg) | Twitchy, with Chvostek's sign. |
| Constipation, thirst, passing a lot of urine, bone pain | High Ca | GI first; cancer or bed rest in the history. |
| Absent knee jerks and RR 10 on milk of magnesia | High Mg | Sluggish, plus an Mg source and weak kidneys. |
| Shallow breathing after tube feeding is started | Low phosphorus | Refeeding. |
| Not one of these: tingling after vomiting with a normal Ca | Metabolic alkalosis | An acid–base picture, not a grid row (Part 4, The four imbalances). |
When two values are abnormal, rank by the function each value threatens, not by how many points it sits outside its range. The order is the ABCDE framework from Exam 3, applied to electrolytes. Four tiers, in order:
- Airway and breathing. Low calcium with laryngeal spasm; high magnesium with a falling RR or lost reflexes; low phosphorus or a potassium below 2.5 with weak respiratory effort.
- Circulation: heart rhythm. Any potassium out of range with weakness or rhythm changes, or with a cause that will keep pushing it (renal failure, a KCl infusion running). Potassium is the commonest electrolyte answer to “who first”, because a breathing problem is rarer.
- Disability: the brain. Sodium with confusion or seizures, in either direction, or below 120; tetany or seizures from low calcium or magnesium.
- The rest. An abnormal value in an alert, stable patient whose cause is already being treated.
What the nurse does, across every imbalance top-weighted topic
Each grid row holds its own treatment; the care that repeats across rows lives here once, in nursing-process order.
| Step | What you do | Which imbalances |
|---|---|---|
| 1 Assess and monitor | Heart: apical pulse and rhythm; cardiac monitor as ordered. | Any K out of range; Ca and Mg, either direction. |
| Brain: neuro checks (level of consciousness, orientation, behaviour), more often if they change. | Na, either direction; high Ca; low phosphorus. | |
| Breathing and reflexes: RR and depth, DTRs; listen for stridor or wheeze. | High Mg; low Ca (laryngeal spasm); low phosphorus; K below 2.5. | |
| Volume: I&O and daily weight (Part 2); urine output before any K. | Na; renal failure; diuretics; high Ca on IV NS. | |
| Labs: recheck the level after each replacement. | All; check Mg when a low K will not correct. | |
| 2 Act | Fall precautions: bed low, call light in reach, help to walk. | The weak and the sluggish: low K, high Ca, high Mg; low phosphorus; confused Na. |
| Seizure precautions: padded rails, suction and oxygen at the bedside. | Na below 120 or with neuro change; low Ca; low Mg. | |
| Airway ready: suction and oxygen near; for high Mg, calcium gluconate available. | Low Ca (laryngeal spasm); high Mg (respiratory depression). | |
| Medication safety: potassium is never pushed (Part 5, IV potassium); know the K before digoxin; IV Mg is high-alert; stop every K or Mg source when high. | K and Mg. | |
| 3 Teach | A written food list for whoever cooks; report weakness, cramps, tingling, palpitations or confusion; no salt substitute with high K; no Mg antacids or laxatives with weak kidneys. | All (foods: the sources table). |
| 4 Delegate | Assistive personnel may weigh, measure I&O, take vital signs, walk the patient and offer fluids within the plan, and report new weakness, numbness or confusion. The RN keeps neuro and reflex checks, the rhythm, the labs, IV electrolytes, teaching and evaluation. | All. |
| 5 Evaluate | Level in range on recheck; signs gone (regular rhythm, oriented, normal reflexes, no tetany); no new problem from the treatment: sodium corrected too fast, low glucose after insulin, K overshooting after replacement. | All. |
The care-plan line. For a patient not yet out of range, the NANDA label is Risk for electrolyte imbalance: risk factors, and no “as evidenced by”. Example: Risk for electrolyte imbalance related to gastric fluid loss through NG suction and loop-diuretic therapy. Measurable outcome: serum K stays 3.5–5.0 mEq/L and Mg 1.3–2.3 mEq/L on daily labs, with a regular apical pulse and no leg cramps, through discharge.
The links: drugs, acid–base, the kidneys and immobility
Most electrolyte items hide the cause in the medication list or the history. Eight links to know:
| The link | What moves | What you do |
|---|---|---|
| Loop and thiazide diuretics (furosemide) | K falls; loops also lower Mg and Ca. Thiazides are the commonest drug cause of low Na, and can raise Ca. | Monitor K and I&O; teach K-rich foods. |
| Potassium-sparing diuretics (spironolactone), ACE inhibitors (lisinopril), NSAIDs, heparin, KCl, salt substitutes | K rises. | Question a new K source; report a rising K. |
| Digoxin | Low K raises the risk of digoxin toxicity. | Know the K before the dose (NUR 110 Exam 1). |
| Mg-containing antacids and laxatives | Mg rises when the kidneys cannot clear it. | Avoid them in renal failure. |
| Vomiting, gastric suction | Acid, K and Cl are lost: metabolic alkalosis, and low K travels with it (Part 4, The four imbalances). | Replace K as ordered. |
| Metabolic acidosis | High K is often present (the shift: Part 4). | Watch the K as the acidosis is treated. |
| Renal failure | K, Mg and phosphate rise; calcium falls. The kidneys are the exit for all three. | Stop every source; expect binders and dialysis. |
| Immobility | Calcium leaves demineralizing bone: high Ca and calcium kidney stones (NUR 110 Exam 2). | Mobilize with weight-bearing; fluids. Stones: Part 7. |
Acid–Base: Buffers, Lungs, Kidneys, and the Four Imbalances
What sets blood pH, the three defences that hold it between 7.35 and 7.45, the four imbalances with their causes and care, and how to read the number that comes to the rescue.
Acids, bases and the 20 : 1 ratio
An acid releases hydrogen ions (H⁺); a base traps them. The more free H⁺, the lower the pH. On the 1–14 scale 7 is neutral, but blood lives in a narrow band: 7.35–7.45, slightly alkaline. Below 7.35 is acidosis, above 7.45 is alkalosis, and below 6.80 or above 7.80 life cannot continue.
Two chemicals carry the whole story. Carbonic acid is the acid the lungs control, by blowing CO₂ off or holding it in, which is why CO₂ acts as an acid. Bicarbonate (HCO₃⁻) is the base, and the kidneys control it. What sets the pH is not the amount of either but their ratio: about 20 parts bicarbonate to 1 part carbonic acid. Every imbalance in this part is that ratio tipping one way; every rescue is the other side tipping to match.
Three defences, fastest first
Three systems guard the ratio. Learn them in order of speed, because that is how items ask for them.
| Defence | How it works | Speed |
|---|---|---|
| 1 · Buffers | Chemical pairs that take up extra H⁺ or release it. Three systems: the carbonic acid–sodium bicarbonate buffer (the main one); phosphate, which works inside cells, especially the kidney tubules; and protein, meaning plasma proteins and the globin part of hemoglobin. | Fast, alongside the lungs. |
| 2 · Lungs | Rising CO₂ stimulates chemoreceptors in the medulla and the carotid and aortic bodies. Breathing becomes faster and deeper, CO₂ is blown off, and the pH rises. Falling CO₂ makes breathing slower and shallower, so CO₂ is kept. | Minutes, but only short term. |
| 3 · Kidneys | In acidosis they excrete H⁺ and form or keep HCO₃⁻. In alkalosis they keep H⁺ and excrete HCO₃⁻. | Hours to days, up to about 3 days. Slow, but the lasting fix. |
Which buffer system matters most? The carbonic acid–sodium bicarbonate system, which buffers as much as 90% of the hydrogen ions in the ECF. Phosphate and protein are real buffers with smaller shares. The fourth option, a “hydrogen buffer system”, is not one of the three.
Across the lifespan. Newborns cannot acidify their urine well. In older adults the lungs and kidneys both work less efficiently, especially with lung disease, so both ends of life have less room for error.
Reading a gas: the refresher
The normals and method are unchanged from NUR 120 Exam 3 (and NUR 110 Exam 3 for the respiratory pair).
| Value | Normal | What it tells you |
|---|---|---|
| pH | 7.35 – 7.45 | Below 7.35 is acidosis, above 7.45 is alkalosis. |
| PaCO₂ | 35 – 45 mmHg | The respiratory number, controlled by the lungs, which respond in minutes. CO₂ acts as an acid. |
| HCO₃⁻ | 22 – 26 mEq/L | The metabolic number, controlled by the kidneys, which take hours to days. HCO₃ acts as a base. |
| PaO₂ | 80 – 100 mmHg | Does not affect acid–base balance. It is read separately, for oxygenation. |
| Step | What to do |
|---|---|
| 1 | Look at the pH. Is it on the acid side, the alkaline side, or normal? <7.35 acid · >7.45 alkaline |
| 2 | Look at the PaCO₂ — this is the respiratory number. High means acid, low means alkaline. |
| 3 | Look at the HCO₃ — this is the metabolic number. Low means acid, high means alkaline. |
| 4 | Which one matches the pH? That one names the imbalance. |
Say it as a sentence: “The pH is acid. The CO₂ is acid. They match — so it is respiratory acidosis.” ATI's ROME is the same rule seen through direction: Respiratory Opposite (pH and PaCO₂ move opposite ways), Metabolic Equal (pH and HCO₃ move the same way). If both numbers match an acid pH, both are causing it: a combined acidosis, the one picture the four steps cannot name alone.
Drawing one (NUR 110 Exam 3): Allen test before a radial stick; the O₂ setting on the requisition; the specimen on ice and to the lab promptly; pressure 5–10 minutes, longer on anticoagulants.
The four imbalances
Each name has two halves: which system started it (respiratory means carbonic acid is off; metabolic means bicarbonate is off) and which way the pH went. The respiratory pair is a refresher, one row each; the metabolic pair is new, and the next section gives its causes in depth.
| Imbalance | Matching number | Causes | Signs |
|---|---|---|---|
| Respiratory acidosis | pH <7.35 PaCO₂ >45 carbonic acid excess: hypoventilation |
Opioid or sedative overdose, anesthesia, COPD, asthma, pneumonia, atelectasis, pulmonary edema, neuromuscular weakness, abdominal distention | Drowsiness, mental cloudiness, confusion, dizziness, twitching; chronic: weakness, dull headache |
| Respiratory alkalosis | pH >7.45 PaCO₂ <35 carbonic acid deficit: hyperventilation |
Anxiety (the most common), pain, fever, hypoxemia, early sepsis, pregnancy, a ventilator set too fast | Lightheadedness, tingling, palpitations, tinnitus, sweating, tremor, poor concentration; convulsions if severe |
| Metabolic acidosis | pH <7.35 HCO₃⁻ <22 bicarbonate deficit, or acid gained |
Diarrhea, DKA, renal failure, starvation, lactic acidosis (shock, cardiac arrest), salicylate (aspirin) toxicity, fistulas | Headache, confusion, drowsiness, Kussmaul breathing (deep and rapid), nausea and vomiting, warm flushed skin; hyperkalemia often present |
| Metabolic alkalosis | pH >7.45 HCO₃⁻ >26 bicarbonate excess, or acid lost |
Vomiting, NG suction, potassium-wasting diuretics, hypokalemia, antacid overuse, steroids | Dizziness, tingling fingers and toes, twitching or hypertonic muscles, slow shallow breathing; hypokalemia |
Acidosis depresses the brain; alkalosis excites it (NUR 120 Exam 3): drowsy and confused on one side, tingling and twitchy on the other. The tingling and twitching of alkalosis look like low calcium: with a normal calcium after vomiting or suction, think metabolic alkalosis (Part 3, Calcium, magnesium and phosphorus).
Read five gases. Cover the right-hand columns, run the four steps, then check.
| pH / PaCO₂ / HCO₃⁻ | The number that matches the pH | Name |
|---|---|---|
| 7.30 / 36 / 17 | HCO₃⁻ low (acid) | Metabolic acidosis |
| 7.52 / 30 / 24 | PaCO₂ low (alkaline) | Respiratory alkalosis |
| 7.28 / 54 / 25 | PaCO₂ high (acid) | Respiratory acidosis |
| 7.49 / 41 / 32 | HCO₃⁻ high (alkaline) | Metabolic alkalosis |
| 7.33 / 35 / 18 | HCO₃⁻ low; a PaCO₂ of 35 sits on the edge but is still normal | Metabolic acidosis |
What the nurse does
| Imbalance | Assess and monitor | Act | Teach · delegate |
|---|---|---|---|
| Respiratory acidosis | Airway, rate and depth, lung sounds, SpO₂; LOC (drowsier means the CO₂ is climbing); repeat ABGs | Sit the patient upright; cough, deep breathing, incentive spirometer; fluids to thin secretions; O₂ and bronchodilators. Hold the next opioid or sedative dose and report; reversal agent if ordered. In a chronic CO₂ retainer (COPD), give exactly the O₂ ordered and watch for rising drowsiness. Lower CO₂ slowly. | Teach the spirometer and a splinted cough. Assistive personnel (AP) may reposition, prompt the spirometer, and take VS and SpO₂ if stable; LOC, lung sounds, O₂ changes and the gas stay with the RN. |
| Respiratory alkalosis | SpO₂ first; rate, pain, temperature, anxiety; tingling and dizziness; on a ventilator, the set rate | Stay with the patient, speak calmly, breathe slowly with them; treat the cause (analgesic, antipyretic, anxiolytic); rebreathe CO₂ (a rebreather mask or paper bag) only once hypoxemia is ruled out; report a ventilator rate set too fast; fall precautions. | Teach slow, paced breathing for the next episode. AP may stay with the patient and report; ruling out hypoxemia stays with the RN. |
| Metabolic acidosis | Cardiac rhythm and K⁺; LOC; breathing pattern; glucose in DKA; I&O; an ABG after each 50–100 mEq of sodium bicarbonate | Treat the cause: IV fluids and insulin in DKA, fluid replacement for diarrhea, sodium bicarbonate if ordered. Don't slow Kussmaul breathing; it is the rescue. Bed low, call light in reach, fall precautions for confusion. | Teach glucose checks and insulin as prescribed in diabetes; report diarrhea that won't stop. AP may record I&O and VS; the rhythm, K⁺ and insulin stay with the nurse. |
| Metabolic alkalosis | Rate and depth (slow, shallow); tingling, twitching, tetany-like spasms; K⁺ and rhythm; I&O including emesis and NG output | Reverse the cause: replace fluid, chloride (0.9% NaCl) and K⁺ (Part 5, IV potassium); antiemetic; irrigate an NG tube with normal saline, not water, and suction no more than ordered; no unneeded antacids; fall precautions. | Teach: no casual antacid use; potassium foods or supplements with a K-wasting diuretic as prescribed. AP may measure emesis and NG output and report a jump; NG irrigation and IV K⁺ stay with the nurse. |
Evaluate four things in every row: the repeat gas moving toward pH 7.35–7.45; mental status back to baseline; breathing at a normal rate and depth (in metabolic acidosis, easing as the acid clears); and K⁺ within 3.5–5.0.
Metabolic causes: what was lost, what was gained
Every metabolic cause comes down to one of four moves: acid lost, acid gained, base lost or base gained. Name the move and the imbalance follows.
| Cause | What happens | Result |
|---|---|---|
| Vomiting, NG suction | Stomach acid (HCl) leaves; potassium and chloride go with it. | Alkalosis |
| Diarrhea, intestinal fistula | Bicarbonate-rich intestinal fluid leaves; chloride is kept. | Acidosis |
| DKA, starvation | Ketones, acids from fat breakdown, build up. | Acidosis |
| Renal failure | The kidneys can no longer excrete H⁺ or rebuild bicarbonate, and potassium stays in. | Acidosis + hyperkalemia |
| Shock, cardiac arrest; aspirin overdose | Lactic acid, or salicylate, is added. | Acidosis |
| Antacid or bicarbonate overuse | Base is added. | Alkalosis |
| Loop or thiazide diuretics | Potassium and chloride are lost in the urine. | Alkalosis |
| Large volumes of 0.9% NaCl | Normal saline carries more chloride than plasma; as chloride rises, bicarbonate falls. | Acidosis |
The GI rule from NUR 120 Exam 3 still settles most items: vomiting loses acid, so alkalosis; diarrhea loses bicarbonate, so acidosis. Sort by what leaves, not by the route.
Potassium rides along
Hydrogen and potassium trade places across the cell membrane. In acidosis, H⁺ moves into cells and K⁺ moves out, so serum K⁺ rises; that is why hyperkalemia so often comes with metabolic acidosis. In alkalosis the trade reverses and serum K⁺ falls. Low potassium also feeds metabolic alkalosis, so the two sustain each other. Insulin pushes K⁺ into cells too. Treat every acid–base patient as a potassium patient, and so as a heart patient (Part 3, Potassium and sodium).
Compensation: the rescue
When one system tips the ratio, the other system pushes back. NUR 120's rule still holds: only the number that matches the pH names the imbalance; a number reading the other way is the rescue, not the disease. The lungs rescue metabolic problems within minutes. The kidneys rescue respiratory problems over hours to days; in respiratory alkalosis they start after about 6 hours.
Why culprit and rescue move together on paper. High CO₂ means acid but high HCO₃⁻ means base, so a rescue pushes the second number the same direction as the first: both up, or both down. A COPD patient who has retained CO₂ for years also carries a high bicarbonate. That is the kidneys' rescue, not a second disease.
How far this exam goes. The program limits ABG work to non-compensatory acid–base balance, so expect the partner number to be normal in most items. The course deck's ABG patterns, though, print the rescue in: respiratory acidosis with HCO₃⁻ “normal or slightly elevated”, respiratory alkalosis with HCO₃⁻ <22, metabolic acidosis with PaCO₂ <35, metabolic alkalosis with PaCO₂ >45. Meet one of these and step 4 still names it; the extra number is the rescue.
IV Therapy and Blood
What each bag does to the cells, where the line goes, how it runs, what goes wrong at the site and in the body, the potassium speed limit, and giving blood safely.
The exam worksheet has one bold line, “REVIEW IV SITES/PLACEMENT/FLUIDS!”, and seven open questions follow it. Each one is worked in its own blue box in this part:
| # | The question | Worked in |
|---|---|---|
| 1 | How do you choose the best place on an extremity for an IV? | Venous access |
| 2 | Give an example of a hypertonic, a hypotonic and an isotonic fluid. | Solutions |
| 3 | Infection vs infiltration vs phlebitis vs emboli. | Complications |
| 4 | The important concepts when giving blood. | Blood |
| 5 | The signs of a transfusion reaction. | Blood |
| 6 | How IV potassium is given, its maximum, and why. | IV potassium |
| 7 | The important concepts in caring for a central line. | Venous access |
Solutions by tonicity: what each bag does to the cells top-weighted topic
Tonicity compares a fluid's particle concentration with plasma's, and water moves toward the side with more particles (Part 1, Tonicity). So the label on the bag tells you which way water will move once it is running: into the cells, out of them, or nowhere.
| Tonicity | Example | Water and cells | Who gets it | Watch for |
|---|---|---|---|---|
| Isotonic | 0.9% NaCl (normal saline) | No net shift: it stays in the vessels and expands the ECF | Hypovolemia; the only fluid hung with blood. Not a routine maintenance fluid | Fluid overload in heart or kidney failure; hypernatremia |
| Isotonic | Lactated Ringer's | The same | Hypovolemia, burns, GI losses | It contains potassium: question it when K is high |
| Isotonic in the bag | D5W | Acts hypotonic once the dextrose is used (trap below) | Hypernatremia: it supplies free water | Not for raised intracranial pressure or for fluid resuscitation |
| Hypotonic | 0.45% NaCl (half-normal saline) | Out of the vessels into the cells, which swell | Maintenance; hypernatremia, lowered gently | Low BP as the vessels empty; avoid with raised intracranial pressure, burns, trauma |
| Hypertonic | D10W | Out of the cells into the vessels: cells shrink | Pulls fluid into the circulation; supplies glucose | Overload and pulmonary edema in heart or kidney disease; irritates veins, so use a large one |
Taylor's hypertonic examples are D5LR and D5 0.9% NaCl (and D5½NS): dextrose added to a saline or LR base makes the bag hypertonic.
Apply the deck's hypertonic question (Part 1, Tonicity) to the bedside: the water a hypertonic bag pulls out of the cells lands in the circulation, so the patient to watch is the one whose heart or kidneys cannot handle more volume. Listen for crackles.
Venous access: choosing the site and the device
Four factors decide the site: how accessible the vein is, what condition it is in, what fluid will run, and for how long. In practice they become seven rules:
- Use the nondominant arm, and involve the patient in the choice.
- Use the forearm for a short peripheral catheter: it dwells longer, hurts less and is harder to pull out. Hand veins are acceptable short-term.
- Start distal and work up: never below a recent site, a flexion area, or painful or damaged skin.
- Avoid the antecubital fossa if any other vein is available (trap below).
- Never use a limb with a same-side mastectomy with node removal, an AV fistula or dialysis catheter, or burns, infection or trauma.
- No leg veins in adults except in an emergency: they risk thrombophlebitis and ulceration.
- Hypertonic, irritant, fast or thick fluids, blood included, need a large vein.
Gauge. 20–24 g for most adults; 22–26 g for neonates, children, older adults and anyone with poor veins; blood 20–24 g, or 18–20 g for a rapid transfusion. Children: hand, forearm, or the foot if not yet walking; scalp veins only as a last resort. Older adults' thin veins infiltrate easily, so limit the tourniquet (or use a BP cuff), lower the insertion angle, and watch the rate closely: with less heart and kidney reserve, overload comes sooner, from a bag or a transfusion.
Technique, the testable points. A tourniquet 3–4 inches above the site, with the radial pulse still present. Skin prep with chlorhexidine in alcohol, left to air-dry: no wiping, fanning or blowing, and no touching the site afterwards. Confirm a blood return and a flush without resistance. Transparent dressing, labelled with date, time, site, and catheter type and size. Two attempts per nurse at most, then escalate. Starting an IV is surgical asepsis (Exam 1) and is never delegated to assistive personnel.
The six devices
| Device | Where it goes | Chosen for | Key facts |
|---|---|---|---|
| Short PIVC | Peripheral vein, forearm preferred | Short therapy, under about 4 days | No vesicants, irritants or parenteral nutrition (PN) |
| Midline | Upper-arm vein; tip at or below the axilla | 5–14 days | Not a central line: the same limits as a PIVC |
| PICC | Inserted in an upper-arm vein; tip central | Long term, weeks to months | A trained RN can insert one; least pneumothorax risk of the central lines; replaced only PRN |
| Nontunneled CVC | Internal jugular, subclavian or femoral vein | Under 14 days; critical, unstable patients | Pneumothorax risk at the subclavian |
| Tunneled CVC | Jugular or subclavian, run under the skin | Long term | A Dacron cuff anchors it; fewer infections than nontunneled; no dressing once healed |
| Implanted port | Under the skin of the upper chest | Long term | Lowest infection rate; accessed with a noncoring needle; nothing visible |
PICC veins: the deck lists basilic, median cubital, brachial and cephalic; Taylor lists basilic, brachial (preferred) and cephalic, above the antecubital fossa. Don't key the median cubital on its own.
A central device is considered when therapy will run longer than about 15 days, or the fluid would injure a small vein. Choose the least invasive device, with the smallest diameter and fewest lumens that will do the job.
True: a central venous access device carries IV fluids, medications, blood products and parenteral nutrition, and it also allows hemodynamic monitoring and blood sampling. That range is why it is chosen for the fluids a peripheral vein cannot take, and why its infection risk gets the care in the next box.
Seven concepts:
- X-ray confirms the tip before the first use.
- Assess the site, dressing and securement with each infusion and at least daily.
- Asepsis at every access: scrub the hub for 5–15 seconds with chlorhexidine-alcohol and let it dry.
- Flush and aspirate for a blood return before and after each use. Lock a CVAD with heparin 10 units/mL or saline; a PIVC is locked with saline.
- Change the dressing on schedule (table below, How often things are changed), and at once if damp, loose or soiled.
- Clamp the catheter whenever the system is opened, and never let a bag run dry: both let air in.
- Not changed on a schedule: review the need daily and remove it once it is no longer needed or infection is suspected. Never submerge it; cover it to shower.
Watch for the five complications: thrombosis, bloodstream infection (CLABSI), air embolism, phlebitis, occlusion. An RN may remove a PICC or midline; most other CVADs are removed by advanced practitioners.
Running an infusion: priming, labelling, piggybacks, pumps and rates
Hanging a bag uses the same rights and checks as a medication: compare it with the MAR, check the expiry date, scan it, and inspect the fluid for clarity and particles. Priming, in five steps:
- Close the roller clamp and spike the bag without touching the port.
- Fill the drip chamber at least halfway.
- Prime the whole tubing until every bubble is gone, then clamp.
- Label the bag (name, solution, additives, date, time) and add a time tape; label the tubing with the date of its next change.
- Recheck the site and rate 30 minutes after starting, then check the infusion at least hourly.
Pumps. They are standard care, and a smart pump does not take away your responsibility for programming it. The alarms mean air, occlusion, low volume or rising pressure; rising pressure can be an infiltrating site (trap at the end of this section). Keep a time tape on the bag even on a pump, and run children's infusions on a pump, not by gravity. Too fast causes fluid overload; too slow leaves a deficit.
| Order and set | Pump | Gravity |
|---|---|---|
| 1,000 mL over 5 h · 15 gtt/mL | 200 mL/h | 200 × 15 ÷ 60 = 50 gtt/min |
| IVPB 50 mL over 20 min · 10 gtt/mL | 150 mL/h | 150 × 10 ÷ 60 = 25 gtt/min |
| 360 mL over 6 h · 60 gtt/mL | 60 mL/h | 60 gtt/min: microdrip, the same number |
How often things are changed
| Item | Change |
|---|---|
| Continuous primary or secondary set | No more often than every 96 h, and at least every 7 days; also with a new catheter, or at once if contaminated |
| Intermittent (IVPB) set | Every 24 h |
| PN set · lipid set | Every 24 h · every 12 h |
| Blood set | A filter takes 4 units at most; don't reuse it after a unit that ran 4 h |
| Transparent dressing · gauze | At least every 7 days · every 2 days; either at once if damp, loose or soiled |
| Needleless connector | No more often than every 96 h; scrub it before every access |
| The PIVC itself | When clinically indicated: no longer needed, unused for 24 h, or a complication. Never by dwell time alone |
ATI and older test banks give a routine PIVC change every 72–96 h; Taylor replaces it when clinically indicated.
Discontinuing a PIVC, in six steps. (1) Clamp the tubing. (2) Remove the dressing while stabilising the hub. (3) Withdraw the catheter without bending it, and don't press on it as it comes out. (4) Then press dry sterile gauze on the site until the bleeding stops; an alcohol pad stings and does not stop bleeding. (5) Check that the tip is intact; if it is not, put a tourniquet high on the limb and notify at once. (6) Occlusive dressing, then document.
When something goes wrong at the site, five rules. Resistance on flushing: never force it; restart elsewhere. Catheter partly out: never re-advance it. Tubing disconnected: discard the set. Artery hit, or tingling on insertion: remove, apply pressure, notify. Hematoma forming: remove and use a new site.
Caring for a running IV: the nurse's five jobs
| Job | What you do |
|---|---|
| Assess | Beyond the 30-minute and hourly checks (step 5 above), a full site check, inspect then palpate for temperature, at least every 4 h; every 1–2 h with a vesicant or irritant, a confused patient or a site over a joint; hourly in children. Then the patient, not the pump: vital signs, lung sounds, I&O, daily weight (Part 2) |
| Act | Run the ordered rate on a pump; keep the bag, tubing and dressing labelled; change the bag before it runs dry; act on the first sign of a complication (table below) |
| Teach | “Press your call light if the skin around the tube hurts, burns, swells, feels cold, tight or wet, or turns red, even a little. Don't wait for the pump to beep, and don't touch the pump or the clamps.” Cover the site to shower |
| Delegate | Assistive personnel measure and record I&O and report any site complaint, wet dressing or alarm. They never start, adjust, flush or discontinue an IV, or restart a pump. An LPN's IV role is set by state law and facility policy |
| Evaluate | Is the fluid problem correcting (weight, I&O over 2–3 days, vital signs, labs), and is the site still quiet? |
IV complications: read the site, then act top-weighted topic
Local complications are more common; systemic ones (overload, air embolism, sepsis) are more dangerous. Pain and swelling go with every local complication, so they decide nothing. Temperature and colour decide. The table below has eight complications.
| Complication | What happened | Signs | First action | Then |
|---|---|---|---|---|
| Infiltration | Non-vesicant fluid leaks into tissue | Cool, pale, swollen, sore; flow slows; leaking at the site | Stop the infusion | Remove the catheter; outline the area; check circulation, sensation and movement below it; elevate; restart at a new site |
| Extravasation | Vesicant leaks into tissue | The same look, plus burning or stinging; tissue damage | Stop the infusion | Follow the protocol (aspirate, antidote if one exists); notify |
| Phlebitis | Vein wall inflamed, mechanically or chemically | Warm, red, tender along the vein; streak; palpable cord | Stop and remove | Warm compress; grade it; restart elsewhere, never in that vein |
| Thrombus | Clot from vein trauma | Like phlebitis; the flow may stop | Stop | Never rub, massage or flush it through; restart elsewhere |
| Infection | Organisms at the site, or in the blood | Redness, induration, pus; fever, chills, malaise | Stop and notify | Culture per protocol; strict asepsis |
| Speed shock | Too-rapid infusion | Pounding headache, fainting, racing pulse, apprehension, chills, back pain, dyspnea | Stop or slow; notify | Prevented by the right tubing, a pump and a time tape |
| Fluid overload | Too much volume | Engorged neck veins, rising BP, dyspnea, crackles, bounding pulse | Slow the infusion | Raise the head of the bed; notify; vital signs |
| Air embolism | Air enters, e.g. through a break in the system | Sudden respiratory distress, rising HR, cyanosis, falling BP, change in LOC | Close the system: clamp or pinch | Left side, head down; call for help; vital signs and SpO₂ |
Phlebitis is graded 0–4: 1 redness; 2 pain with redness or swelling; 3 adds a streak and a palpable cord; 4 a cord longer than 1 inch with pus.
“Emboli” is plural on the worksheet: three things can travel. Air, through a break in the system (the last row). A clot: a thrombus at the site can break loose, which is why a clotted line is never rubbed, massaged or flushed through. A piece of the catheter: inspect the tip whenever a catheter comes out; if it is not intact, apply a tourniquet high on the extremity and notify at once, to keep the fragment from travelling. Infiltration and phlebitis stay at the site; an embolus leaves it, and so can an infection, into the bloodstream.
IV potassium: the speed limit
| How | Never IV push or bolus (Exam 1: concentrated KCl is never pushed, under any circumstance). Always diluted, always on a pump. Check urine output first: the kidneys are the main way potassium leaves. |
| Maximum | No faster than 10 mEq per hour. So 20 mEq takes at least 2 hours, and 40 mEq at least 4. |
| Why | A bolus raises serum potassium suddenly, and sudden hyperkalemia can cause a fatal dysrhythmia (Part 3, Potassium and sodium). |
| Also | It can sting the vein: warn the patient, and watch the site. |
Blood: compatibility, giving it, and reactions
Typing identifies the blood type; crossmatching tests the donor's blood against the recipient's. A red cell carries antigens, and the plasma carries antibodies against the antigens the person lacks.
| Type | Antigen on red cells | Antibodies in plasma | Can receive red cells from |
|---|---|---|---|
| A | A | Anti-B | A, O |
| B | B | Anti-A | B, O |
| AB | A and B | None: universal recipient | A, B, AB, O |
| O | None: universal donor | Anti-A and anti-B | O only |
Rh is a second, separate match. The D antigen present means Rh-positive. An Rh-negative patient receives Rh-negative blood: Rh-positive blood makes them form antibodies, and a later transfusion then destroys red cells. An Rh-negative donor can give to an Rh-positive patient.
Components, five. Packed red cells raise the hemoglobin without much volume; fresh frozen plasma gives clotting factors and volume; platelets treat a low count; albumin treats low albumin and hypovolemic shock; cryoprecipitate treats low fibrinogen and hemophilia. Whole blood is rare.
Nine steps, in order:
- Verify the order and the informed consent.
- Before collecting the unit: vital signs within 30 minutes of starting, lung sounds, risk conditions (fever, heart failure, kidney disease) and a patent IV, 20–24 g, or 18–20 g for a rapid transfusion.
- Collect the unit and start it promptly, within about 30 minutes or per policy.
- Two qualified people check at the bedside, with the patient: two identifiers against the band, the patient's ABO and Rh against the unit's, the donation number and the expiry. Any mismatch: don't spike it; call the blood bank.
- Use a blood set with a filter, primed with 0.9% NaCl only. Never dextrose, which destroys red cells, and never add a medication to the line.
- Start slowly and stay for the first 15 minutes: major reactions usually appear within the first 50 mL.
- Vital signs at 15 minutes; with no reaction, raise the rate as ordered. Assess at least every 30 minutes, with vital signs per policy.
- Finish within 4 hours.
- Vital signs at the end and 1 hour later. Watch for 4–6 hours for a delayed fever or breathing trouble, and teach the patient those signs.
Document the donation (unit) number, the times started and finished, the volume (on the I&O), the vital signs and the patient's response. The bedside check and the monitoring stay with licensed nurses; assistive personnel report any new symptom at once.
ATI's modules give three different gauges for blood (18–20, 18–24, 20–24 g); Taylor's 20–24 g, or 18–20 g for a rapid transfusion, is the one to learn.
Every reaction starts with the same six steps: (1) stop the transfusion; (2) keep the vein open with 0.9% NaCl through new tubing, so the blood in the line is not pushed in; (3) stay, assess, and compare vital signs with the baseline; (4) notify the provider and the blood bank; (5) send the bag and tubing, and collect blood and urine samples per protocol; (6) document. The signature tells you which reaction it is. The one exception is circulatory overload: the problem is volume, so the textbook allows slowing rather than stopping, and the saline that keeps the vein open runs at the slowest rate.
| Reaction | Signature | After stop and saline |
|---|---|---|
| Allergic | Hives, itching; can progress to anaphylaxis | Notify; antihistamine as ordered |
| Febrile | Fever, chills, headache, malaise | Notify; treat the symptoms |
| Hemolytic | Incompatible blood, immediate: flushing, fever, chills, headache, low back pain, falling BP, shock | Notify at once; blood samples and the first voided urine; treat shock; send the unit, tubing and filter to the lab |
| Circulatory overload | Dyspnea, dry cough, crackles, rising BP | Slow or stop; sit upright with feet dependent; vital signs; notify |
| Bacterial | Fever, dry flushed skin, abdominal pain; blood pressure changes (see the note below) | Blood cultures; return the bag; antibiotics as ordered |
| Not one of these | Swelling only at the IV site | Infiltration: site care, as in the complications table |
Blood pressure is where the sources split. For bacterial contamination, Taylor's table prints hypertension while ATI lists hypotension, the usual septic picture; key on the fever and the contaminated unit, not on the blood pressure. For circulatory overload, the rising BP is ATI's; Taylor lists dyspnea, dry cough and pulmonary edema.
Urine: How It Is Made, What Normal Looks Like, How You Test It
The route urine takes and how voiding works, the output numbers, what changes voiding, how to assess urinary function, normal versus abnormal urine, the eight terms, the specimens and the bladder scanner.
The route, and how voiding works
Urine passes through four structures: kidney, ureter, bladder, urethra. How the kidneys decide how much to make is Part 1, Holding the balance.
Voiding (micturition) is a spinal reflex that the brain learns to control after infancy. Stretch receptors fire as the bladder fills, and the urge begins at about 150–250 mL. To void, the detrusor contracts, the internal sphincter, perineal muscles and external sphincter relax, and the abdominal wall tightens slightly. Voluntary control covers three things only: starting, holding back and interrupting. Normal voiding is painless. Embarrassment can stop it altogether, which matters when you need a specimen. A bladder cut off from the brain by injury or disease still empties by reflex: an autonomic bladder (Part 7, Incontinence).
The female urethra is short and close to the vagina and rectum, which is why women get more UTIs (Part 7). The male urethra is long and curved and passes through the prostate, which can obstruct it.
ATI says the adult bladder comfortably holds 300–400 mL and signals at 250–400 mL. Use 150–250 mL for the point at which the urge begins.
Normal voiding and normal output
Healthy adults void roughly every 3–4 hours while awake and usually not during sleep. The first morning void is the most concentrated, which is why some tests ask for it. How often someone voids depends on how much urine is being made, so a change from the person's usual pattern, not the pattern itself, is what suggests illness. Habitually holding urine for 8–12 waking hours breeds UTIs.
| Number | What it means | The arithmetic |
|---|---|---|
| ≥ 30 mL/h | The minimum adult output. Below it, report. | 240 mL per 8-hour shift · 720 mL a day |
| < 400 mL/24 h | Oliguria | about 17 mL/h |
| < 50 mL/24 h | Anuria | about 2 mL/h: essentially none |
| 6–8 wet diapers/day | Expected for a newborn | A diaper dry for more than 3 h signals dehydration |
Taylor gives typical adult output as about 1,500 mL a day, roughly 40–80 mL/h. Those describe average output; 30 mL/h is the report threshold.
What changes voiding
| Factor | What it does |
|---|---|
| Infants and children | Newborn urine is pale and odourless. Voluntary control comes at 18–24 months; most children are dry between 2 and 5, day before night, girls before boys. Regression in hospital is expected and short-lived. Nocturnal enuresis usually resolves by 6. |
| Older adults | Less concentrating ability: nocturia. Lower tone: frequency, urgency. Weaker contraction: retention, stasis, UTI. Enlarged prostate in men over 50. Incontinence is not a normal part of ageing: it is a treatable symptom. |
| Food and fluid | Low intake: less, darker urine. Alcohol inhibits ADH, and caffeine also increases output. High-sodium foods hold fluid back, so less urine. |
| Psychological | No privacy can stop voiding; stress can cause small, frequent voids. |
| Activity and tone | Immobility causes stasis and poor control. An indwelling catheter costs bladder tone, because the bladder is never stretched by filling. Childbirth and menopause weaken the pelvic floor. |
| Surgery and anaesthesia | Pelvic or lower abdominal surgery, epidural or spinal anaesthesia, opioids and delayed walking favour retention (Part 7). |
| Disease | Fever and sweating: less, concentrated urine. Heart failure: less urine. High blood glucose: osmotic diuresis, so polyuria. Arthritis and Parkinson's make toileting hard. |
Eight diseases linked to kidney problems: congenital urinary tract abnormalities, polycystic kidney disease, UTI, urinary stones, hypertension, diabetes mellitus, gout and connective tissue disorders. The last four are diseases of other systems, so they are the ones most often missed on a select-all item. Ask about all eight in the history (next section).
Drugs that change the amount or emptying. Retention: anticholinergics, antihistamines and OTC cold remedies, antidepressants, calcium-channel blockers, opioids and epidural analgesia. More urine: diuretics. Blunted reflex or awareness: analgesics, sedatives and tranquillisers. Nephrotoxic: NSAIDs such as aspirin and ibuprofen, and gentamicin. Watch I&O and the renal labs, because nephrotoxicity does not announce itself as frequency, incontinence or nocturia.
| Cause | Urine colour | Is it blood? |
|---|---|---|
| Anticoagulants | Pink or red | Possibly: assess for bleeding |
| Beets, blackberries, rhubarb | Red | No |
| Phenazopyridine (Pyridium), a urinary analgesic | Orange to orange-red | No; expected |
| Diuretics | Pale yellow | No; dilution |
| Amitriptyline; B-complex vitamins | Green or blue-green | No |
| Levodopa; injectable iron | Brown or black | No |
| Bile pigments (liver disease) | Brown, tea-coloured | No, but abnormal |
Nocturia, a refresher (NUR 120 Exam 4): alone in an older adult it is ageing; with ankle edema and orthopnea it points to heart failure.
Assessing urinary function: ask, inspect, palpate, measure
A focused urinary assessment runs in four steps, in this order: ask, inspect, palpate or percuss, measure. Judge every finding against the patient's own usual pattern.
1. Ask: the history, under five headings
| Heading | Ask, in plain words | What it gives you |
|---|---|---|
| Usual pattern | “How often do you pass water in the day? Do you get up at night to go? What does your urine usually look like?” | The baseline |
| Recent changes | “Any change in how often, how much, or how strong the stream is? Any leaking when you cough or sneeze, or on the way to the bathroom?” | A change, which is what suggests illness |
| Past or present problems | “Any urgency, burning, trouble starting, dribbling? Bladder infections or kidney trouble before? Do you use pads?” | Dysuria, hesitancy, incontinence; the renal history (the eight diseases above) |
| Aids | “Is there anything you do that helps you go?” | Routines to keep in the care plan |
| Urinary diversion | “How do you look after your stoma, and what equipment do you use?” | Their routine and supplies, recorded (Part 7, Urinary diversions) |
Use the patient's own words for urinating; for a child, record the child's word for needing to go, and for an infant count wet diapers. Review the medication list against the drugs above. When a problem comes up, explore how long, how bad, what brings it on and what they already do about it.
2–3. Inspect, then palpate and percuss: five sites
| Site | What you do | Report or act on |
|---|---|---|
| Skin | Inspect colour, texture, turgor (Part 2) and the perineal skin | Excoriation or IAD from incontinence (Part 7) |
| Urethral meatus | Inspect for inflammation, discharge and odour. Female: dorsal recumbent, labia retracted. Male: retract the foreskin if uncircumcised, then replace it | Infection cues: report |
| Urine | Inspect colour, odour, clarity and sediment; SG, pH and constituents when indicated. In acute care, at least once a shift, even if the patient measures their own | Anything on the abnormal list (next section) |
| Bladder | Ask when they last voided first. Inspect above the symphysis pubis for swelling, palpate for tenderness, percuss, or use the bedside scanner (The bladder scanner, below). An empty bladder can't be felt; a full one rises above the symphysis, sometimes to just below the umbilicus | Distended: scan, then retention care (Part 7) |
| Not yours: kidneys | Deep palpation, done by an advanced practitioner | — |
4. Measure: three situations
| Patient | How |
|---|---|
| Continent | Gloves. Void into a urinal, bedpan or specimen hat under the toilet seat. Pour into a calibrated container, set it on a flat surface and read it at eye level, record. Discard in the toilet unless a specimen is needed. |
| Incontinent | Count the episodes and note colour and odour. Weigh the pad: wet minus dry, 1 g = 1 mL. Toileting about every 2 hours gives urine to measure and prevents episodes. |
| Indwelling catheter | Drain the bag into the patient's own graduated container at least every 8 hours; the bag's own markings are only approximate (spout care: Part 7). For hourly output in the acutely ill, a urimeter: read it, then tip it into the main bag. |
Who does what. An AP may measure, empty and record. The nurse makes sure the AP knows the procedure, validates the accuracy, and does the assessment and the interpreting (Part 2, Measuring fluid status).
Normal versus abnormal urine
| Feature | Normal | Abnormal, and what it suggests |
|---|---|---|
| Volume | ≥ 30 mL/h | Less: report. Large volumes: polyuria (terms below). |
| Colour | Pale yellow, straw or amber, by concentration | Dark amber: concentrated. Red or pink: blood or food. Brown: bile. Drugs: the table above. |
| Clarity | Clear when fresh | Cloudy fresh urine: RBCs, WBCs, bacteria, vaginal discharge, sperm or prostatic fluid. |
| Odour | Faintly aromatic | Sweet: glucose. Fetid: heavy infection. Musty after asparagus. |
| Specific gravity | 1.010–1.025 | High: concentrated, usually dehydration. Low: dilute, usually overhydration. Glucose or protein raise it. |
| pH | 4.6–8.2 | A protein-rich diet acidifies; citrus, dairy and vegetables alkalinise. |
| Constituents | Urea, creatinine, uric acid, electrolytes | Abnormal: blood, pus, protein, glucose, ketones, casts, gross bacteria, bile. |
Taylor gives specific gravity as 1.005–1.030 in Ch 41 and 1.015–1.025 in Ch 38, and urine pH as 4.5–8 (usually 5–6). A value well outside every range, such as 1.035 or 1.002, means the same thing under all of them.
The reagent strip (dipstick) screens at the bedside. Hold the thick end, dip so every pad is soaked, tap off the excess, lay it on its side and read it against the colour chart at the time the manufacturer specifies. An abnormal strip needs lab confirmation.
| Pad positive | Suggests |
|---|---|
| Glucose | High blood glucose spilling over (diabetes); also sweet odour and a higher SG |
| Ketones | Fat breakdown: uncontrolled diabetes, starvation |
| Protein | Kidney damage |
| Blood | Catheter or cystoscopy trauma, stones, infection, anticoagulants. Note menstruation on the slip. |
| Leukocyte esterase, nitrites | WBCs and bacteria: infection (Part 7) |
| Bilirubin | Liver or biliary disease |
False. Fresh urine smells faintly aromatic. An ammonia smell develops as urine stands, from bacterial action, so it tells you about the specimen's age before it tells you anything about the patient.
The eight terms, as one family
| Term | Means | Hook: the question it answers |
|---|---|---|
| Anuria | Less than 50 mL in 24 h: kidney shutdown | How much is made? Essentially none |
| Oliguria | Less than 400 mL in 24 h | How much is made? Too little |
| Polyuria | Excessive output (diuresis); no fixed number | How much is made? Too much: high glucose, diuretics, alcohol, a big intake |
| Frequency | Voiding more often than usual | How often? |
| Urgency | A strong, sudden need to void | How hard does it push? |
| Nocturia | Waking at night to void | When? |
| Dysuria | Painful or difficult voiding | How does it feel? Expected briefly after catheter removal or cystoscopy |
| Hematuria | Blood in the urine | What is in it? |
| Not one of these: retention | Urine made normally but not emptied | A full bladder and an empty bag (Part 7, Retention) |
Also: glycosuria, proteinuria, pyuria (pus) and bacteriuria (bacteria without symptoms, not a UTI: Part 7).
Anuria, which the deck calls synonymous with kidney shutdown or renal failure. The three distractors each answer a different question in the table: dysuria is how voiding feels, glycosuria is sugar in the urine, pyuria is pus in it. And an empty bag over a full bladder is retention, not anuria. Anuria and oliguria are about how much the kidneys make; scan before you name it.
Some sources set anuria at less than 100 mL in 24 h. Taylor and the Ch 38 deck both use less than 50 mL.
Collecting urine specimens
Every specimen is labelled in front of the patient (two identifiers, date and time) and bagged. Urine changes as it stands, so it reaches the lab promptly (ideally within 2 hours) or is refrigerated.
| Specimen | Sterile? | How | Amount, use |
|---|---|---|---|
| Routine UA | No | Void into a clean bedpan, urinal or hat; keep out stool and toilet tissue. | ≥ 10 mL |
| Clean-catch midstream | Clean, not sterile | Clean the meatus (female: separate the labia, front to back; male: retract the foreskin, and replace it afterwards). Void the first ~30 mL into the toilet, catch the middle of the stream without touching the meatus, finish in the toilet. | 3–5 mL minimum; the usual culture specimen from a patient who can void |
| Straight catheter | Sterile | Surgical asepsis, for a patient who can't void. | Culture ~3 mL |
| Indwelling catheter | Sterile | From the sampling port with a syringe (steps below). Never from the drainage bag, where urine is stale, and never by opening the closed system. | Culture ~3 mL; UA ≥ 10 mL |
| 24-hour (timed) | No | Start by voiding and discarding; record the time. Keep every void for 24 h, cold, with any preservative the lab specifies. At the end, void and add it. A missed void restarts the test. Post a sign on the bathroom door. | Creatinine clearance, protein |
| First morning | No | The first void after waking. | Most concentrated; pregnancy test |
| Infant or child | Varies | Midstream is possible over age 2 with help. Infants: an adhesive collection bag; never squeeze a wet diaper. Suprapubic aspiration is not an RN skill. | — |
| Urinary diversion | UA no; culture yes | For a culture, never use the existing pouch; catheterise the stoma (Part 7). | — |
A culture and sensitivity (C&S) names the organism and the antibiotics that work on it, in 24–48 hours; a UA shows bacteria but not which ones.
From the port of an indwelling catheter, in six steps:
- Gather a syringe sized to the test, an antiseptic swab, a sterile container, clean gloves and a clamp.
- If the tubing is empty, clamp it below the port for no more than 30 minutes so fresh urine collects.
- Scrub the port with the swab and let it dry.
- Attach the syringe and aspirate slowly.
- Remove the syringe and release the clamp.
- Transfer to the sterile container, label at the bedside, bag and send.
False. It comes from the catheter itself, which in practice means the sampling port, by the six steps above. Bag urine has been standing, so it is stale and contaminated; disconnecting the tubing to reach fresher urine opens the closed system. A specimen is only as fresh and as sterile as the place you took it from: from a catheter, the port, never the bag.
The Ch 38 deck now confirms the port, as Taylor does. The only outlier is the insertion checklist, which takes a specimen at insertion from the brand-new drainage bag. For any exam item, answer with the port.
The bladder scanner and the post-void residual
A portable ultrasound bladder scanner estimates the volume in the bladder. It is non-invasive, painless and carries almost no infection risk, so it is preferred over catheterising just to find out, and it cuts catheter use and CAUTI. The post-void residual (PVR) is what is left straight after voiding.
Scanning, in seven steps:
- Patient supine and draped; a right-handed nurse stands on the right.
- Turn it on, choose SCAN and select the patient's biological sex.
- Clean the scanner head and palpate the symphysis pubis.
- Put a generous amount of ultrasound gel, never lubricant, midline 1–1.5 in above the symphysis.
- Point the direction icon toward the head and aim slightly down toward the coccyx.
- Centre the bladder on the crosshairs, rescanning until it is centred, then read the volume.
- Clean up; document why you scanned, the volume and the response.
A PVR under 50 mL is adequate emptying; over 100 mL, the bladder is not emptying properly. Measuring the residual does not help the patient void; what comes next is retention care (Part 7). Ascites makes the reading inaccurate.
The same scan tells retention (a full bladder) from low production (a nearly empty one): Part 7, Retention.
Other diagnostic tests, briefly
| Test | Before | After: watch for |
|---|---|---|
| Urodynamic studies | Bladder full; tell them a catheter will be inserted | 8–10 glasses of water over 24 h; teach the signs of UTI |
| Cystoscopy (urethra and bladder) | Informed consent; sedation and analgesia | Dysuria and hematuria are expected at first; generous fluids; output for at least 24 h; watch for retention and infection |
| IV pyelogram | NPO; allergy history; not with a raised BUN or creatinine, or in pregnancy; bowel prep; consent | Contrast reaction (rash, nausea, hives); contrast can cause acute kidney failure, so watch I&O |
| CT with contrast | NPO about 8 h; allergy history; remove metal | A delayed contrast reaction |
Urinary Problems and Nursing Care: Incontinence, Retention, Infection, Catheters
The four problems the objectives name, the nursing care for each, the catheter from the decision to insert it to the first void after it comes out, other devices (suprapubic and condom catheters, irrigation, stents and dialysis access), and what can be delegated.
Incontinence: sort it by where the failure is top-weighted topic
Three rules hold for every type. Behavioural measures come first. An indwelling catheter is the last choice, because of infection. Absorbent products are not a treatment: they protect skin and clothing, and used carelessly they cause skin breakdown and UTI. Choose one on six factors: functional disability, type and severity, gender, caregivers available, earlier treatment failed, and patient preference. Eight types:
| Type | Where it fails, and why | The cue in a stem | Key intervention | Hook |
|---|---|---|---|---|
| Stress | Weak pelvic floor or sphincter plus a rise in abdominal pressure. Childbirth, menopause, obesity, chronic constipation. | Small leak with cough, sneeze, laugh, lift or standing up. No urge first. | Kegels, weight loss, treat constipation; pessary; surgery last. | Effort squeezes it out. |
| Urge | Overactive bladder. Caffeine, alcohol, faecal impaction, UTI, poor toileting habits; diuretics, especially in older adults. | “I can't make it in time.” Frequency, nocturia. | Bladder training; cut caffeine, alcohol, artificial sweeteners; diuretic in the morning; treat UTI or impaction. | The urge wins the race. |
| Mixed | Stress and urge together. | Leaks with effort and with urgency. | Kegels plus bladder training. | Both. |
| Overflow | Bladder never empties: weak signal or a blocked outlet. Enlarged prostate, diabetes, neurological disease, drugs. | Constant dribbling, distended bladder, high PVR. | Empty the bladder: intermittent catheter, double voiding; treat the obstruction; review drugs. | A full cup spilling over. |
| Functional | The urinary tract works; reaching the toilet doesn't. Mobility limits, memory loss, disorientation, barriers. | Knows she needs to go; can't get there or undo her clothes in time. | Fix the environment: commode, clear path, call light, elastic or Velcro clothing; prompted or scheduled toileting. | Bladder fine, access broken. |
| Reflex | No sensation of fullness: the nerves to the brain are damaged (spinal cord injury, MS). Empties by reflex. | Empties without warning, at fairly predictable volumes. | Scheduled intermittent (self-)catheterisation; timed voiding; skin care. | No message gets through. |
| Transient | Appears suddenly and lasts 6 months or less. A temporary, treatable cause: confusion from acute illness or infection, diuretics, IV fluids. | New, during an admission. | Find and treat the cause; continence should return. | Here today, gone with the cause. |
| Total (continuous) | Unpredictable, constant loss: surgery, trauma, fistula. | Never dry. | Containment, external device, skin care; repair. | Never closes. |
| Not one of these | Retention without leaking (next section); enuresis, wetting past toilet-training age, is a developmental term, not a mechanism. | — | — | — |
True. A diuretic raises urine production, so the bladder fills faster and the urge comes sooner. Read the medication list before you name the type, and teach a morning dose so the peak doesn't fall at night. Leaking that starts suddenly with a new diuretic during an illness is still transient on the ladder.
Nursing care, any type:
| Step | What you do |
|---|---|
| Assess and monitor | History, including the environment and walking aids; diet (caffeine, sweeteners, too little fluid); a voiding diary of times, amounts and leaks; palpation, a PVR and a urine screen for UTI, a frequent cause; the perineal skin. |
| Act | The type's key intervention (table); call light and urinal or commode within reach; clean and protect the skin after every episode (IAD, below). |
| Teach | The type's plan (Kegels, bladder training); fluids up, irritants down; incontinence is not a normal part of ageing. |
| Delegate | AP: toileting schedules, perineal care, pads, I&O. The nurse: the type, the skin, the plan, teaching. |
| Evaluate | Fewer leaks and longer intervals in the diary; intact skin. |
Priority (Exam 3): incontinence is an actual problem and skin breakdown only a risk, so incontinence ranks first.
Retention: urine made but not emptied top-weighted topic
Six causes: an enlarged prostate (men far more often); drugs that block the emptying reflex, such as anticholinergics, antihistamines, antidepressants and opioids (Part 6, What changes voiding); surgery on the lower abdomen or pelvis, especially with an epidural, delayed walking or low intake; swelling after childbirth; nerve damage; and obstruction or prolapse. Immobility lowers bladder tone and leaves urine standing (Exam 2).
The cues: a bladder distended above the symphysis pubis, discomfort, frequent small voids, hesitancy, a weak stream, and dribbling (overflow). The proof is a high PVR: above 100 mL means the bladder is not emptying (Part 6, The bladder scanner). Untreated, retention leads to UTI, hydronephrosis and kidney damage.
Nursing care:
| Step | What you do |
|---|---|
| Assess and monitor | Ask when they last voided and what they have drunk, then palpate above the symphysis and bladder-scan (non-invasive, no infection risk); look for the causes above, and after surgery track the time since the last void. |
| Act | Nursing measures first: privacy, usual position, running water within earshot, warm water over the perineum (cold tightens the muscles), analgesia if pain holds them back; double voiding, or Credé with an order (both below). Then intermittent (straight) catheterisation per order, the preferred route, post-op too; indwelling only for gradual decompression or retention that won't resolve. Drain no more at once than facility policy allows; treat the cause; walk early after surgery. |
| Teach | Void at the first urge; report hesitancy, a weak stream or fullness; ask before taking OTC decongestants or antihistamines. |
| Delegate | AP records every void and reports a missed one; scan per agency policy. The nurse assesses and catheterises. |
| Evaluate | Voids comfortably in adequate amounts; PVR back in range on re-scan. |
UTI and CAUTI top-weighted topic
UTI is one of the commonest health care–associated infections and a common source of systemic infection in older adults. E. coli from the bowel is the usual organism. A lower UTI is the bladder (cystitis) and urethra; an upper UTI is the kidneys and ureters (pyelonephritis), and an untreated lower UTI can climb there.
Seven risk factors: female anatomy (a short urethra near the vagina and rectum) and sexual activity; a diaphragm or spermicide, which kills protective flora; menopause; an indwelling catheter; diabetes; older age, including an enlarged prostate; and stasis from retention, immobility or habitually putting off voiding.
| Picture | What you see |
|---|---|
| Lower UTI | Dysuria, frequency, urgency with small volumes, suprapubic discomfort, cloudy, foul-smelling urine, hematuria. |
| Upper UTI | Adds flank or low-back pain, fever and chills, nausea and vomiting. |
| With a catheter | Burning or irritation at the meatus, cloudy urine, strong odour, fever, chills. |
| Older adult | Classic signs may be missing. New confusion, agitation, falls, new incontinence or poor appetite may be the only sign, often without fever. |
Nursing care:
| Step | What you do |
|---|---|
| Assess and monitor | The signs above and temperature; mental status in an older adult; urine colour, clarity and odour; I&O. |
| Act | Culture before the first antibiotic dose (Exam 1; how: Part 6, Collecting urine specimens). Antibiotic as ordered, a short course for a lower UTI, longer for an upper; generous fluids unless restricted; ask whether any catheter is still needed. |
| Teach | Finish the whole course; the six prevention points below. |
| Delegate | AP: perineal and catheter care, fluids, I&O; reports fever, new confusion, cloudy or foul urine. The nurse assesses and teaches. |
| Evaluate | Symptoms and fever settle; mental status back to baseline. New flank pain, fever or chills mean it has climbed: report. |
Prevention, six points in a patient's words: six to eight 8-oz glasses of fluid a day; go when you first feel the urge and take time to empty fully; wipe front to back; urinate before and after sex; cotton-crotch underwear and no tight clothes; shower rather than bathe, with no douches or sprays. If infections recur, discuss alternatives to a diaphragm or spermicide.
CAUTI. About 75% of hospital UTIs are catheter-associated, and CMS treats CAUTI as preventable and does not pay for its treatment. Bacteria form a biofilm on the catheter and climb inside the lumen or along the outside. The bundle (Exam 1): insert only for a valid indication, with surgical asepsis; maintain it by the seven care rules (Caring for the catheter); remove it as early as possible.
Kidney stones: the calcium bridge top-weighted topic
Refresher (Exam 2): with immobility, calcium leaves demineralising bone and can precipitate as renal calculi. That is the bridge from high serum calcium (Part 3, The links) to this chapter. Other risks include low urine volume, a high urine calcium, hyperparathyroidism, excess calcium or calcium antacids, gout, obesity, diuretics, recurrent UTI, male sex and a prior or family history of stones.
The picture is sharp flank pain radiating to the lower abdomen or groin, hematuria, frequency, urgency and dysuria. A stone can block the outflow and cause retention. Fever and chills mean infection behind an obstruction: report. For a small stone, strain all urine and send any stone to the lab to be identified, push fluids unless restricted, give analgesia as ordered, and track I&O and temperature; a large or obstructing stone is broken up by shock-wave lithotripsy or removed.
Promoting voiding and continence
“No interventions are effective for preventing incontinence” is false. Four kinds work, and this section follows them: regular voiding habits; fluid intake, kept up, not cut; muscle tone (pelvic floor training); and help with toileting (scheduled voiding, the environment). Incontinence has a cause: name the type and treat the mechanism; containment is not treatment.
Habits, five of them: help the patient void when the urge first comes; give privacy unless they are too weak to be left; use their usual position (men standing, women often on a commode; the positioning rules are from Exam 2); stay with anyone who might fall and never lock the bathroom door; and keep fluids up, six to eight glasses a day.
Pelvic floor muscle training (Kegel). It reduces or eliminates stress incontinence and helps every type in women. The patient finds the muscle by imagining stopping urine midstream or holding back stool, then squeezes 5–10 seconds and relaxes 10, with the abdomen, thighs and buttocks relaxed, in sets through the day for at least 3 months. Stopping the stream is for finding the muscle once, not a daily exercise: repeated stop-start voiding can leave urine behind.
Scheduled voiding comes in three forms, and they differ by who runs the schedule:
| Bladder training | Habit training (timed voiding) | Prompted voiding | |
|---|---|---|---|
| Who drives it | The patient | The caregiver | The caregiver |
| Cognition | Intact and motivated; can resist an urge | May be impaired | Impaired, but responds when asked |
| Schedule | Starts at the diary interval, then lengthens gradually; urges handled with distraction and relaxation | Fixed to the patient's own pattern; not lengthened | Regular checks: “wet or dry?”, prompt, help, praise |
| Best for | Urge, mixed | Functional, cognitive impairment | Functional, dementia |
Two more techniques. Double voiding: void, wait a few minutes or lean forward, then void again, for incomplete emptying. Credé is firm downward pressure over the bladder for a flaccid bladder, used only with an order.
Incontinence-associated dermatitis (IAD). Bacteria turn urea into alkaline ammonia, which raises skin pH and breaks down the barrier. It looks like redness with poorly defined edges, warm and firm, with burning or itching. Inspect the perineum, buttocks and hips daily. Care, in four steps: prevent the incontinence; clean promptly after each episode with a pH-balanced cleanser (not soap and hot water), patting, never rubbing; moisturise, nothing with alcohol; a barrier (zinc oxide, petrolatum or a sealant) on all skin urine can reach, after every episode. Refer to a WOC nurse if it hasn't responded in 2 weeks.
Catheters: when, and which kind top-weighted topic
Every catheter needs an order (Exam 3), and urethral insertion is surgical asepsis (Exam 1). Nurses insert every type except the suprapubic and the coudé. Eight indications: acute retention; long-term drainage when nothing else works (neurogenic bladder); a sterile specimen from a patient who can't void; accurate output in the critically ill; healing an open sacral or perineal wound in an incontinent patient; around some surgery and tests; end-of-life comfort; prolonged immobilisation (unstable spine, multiple trauma). Not indications: managing incontinence, or staff convenience.
| Type | What it is | Used for | Remember |
|---|---|---|---|
| Intermittent (straight) | In, drain, out | Retention, post-op retention, self-catheterisation | Lower CAUTI risk than indwelling. Clean technique at home is supported. |
| Indwelling (Foley) | Held by a balloon of sterile water. Double lumen: balloon and drainage. Triple: adds irrigation. | Continuous drainage, decompression, irrigation | Usual adult size 14F, 5–10 mL balloon. Smallest that drains; 18F and up erode the urethra. |
| Coudé | Curved tip | Enlarged prostate | Not a nursing insertion (above); for when a straight catheter won't pass. |
| Suprapubic | Surgically placed through the abdominal wall above the pubis | Long-term drainage when the urethra is damaged or blocked | Less contamination and lower CAUTI risk. Site care below. |
| External male (condom, urinary sheath) | Sheath over the penis to a bag | Incontinence without retention | Clean technique; no CAUTI risk. Application and care below. |
| External female | Wick between the labia on low continuous suction | Output, dry skin, no indwelling catheter | Keep suction on while removing it. |
Inserting an indwelling catheter, female and male top-weighted topic
Before you open the kit: confirm the order, ask about latex and iodine allergy, get good light (a flashlight needs an assistant), use a firm surface, and get a second person if needed. The lab grades the steps below; the goal line is urine output of at least 30 mL/h with a bladder that is not distended.
| Phase | Female | Male |
|---|---|---|
| Position | Dorsal recumbent, knees flexed, feet about 2 ft apart; or Sims' | Supine, thighs slightly apart |
| Pre-clean | Clean gloves; front to back, a new corner of the cloth each stroke; gloves off, hand hygiene | Clean gloves; tip of the penis in circles from the meatus out, then the shaft downward; gloves off, hand hygiene |
| Sterile field | Open the kit on the overbed table; sterile gloves; drape cuffed over the gloved hands; fenestrated drape; attach the prefilled syringe to the balloon port; lubricate 1–2 in of the tip | |
| Expose | Non-dominant hand spreads the labia | Non-dominant hand lifts the penis and retracts the foreskin |
| Cleanse | Dominant hand, one swab per stroke: each labial fold top to bottom, then straight down over the meatus, meatus last | Dominant hand: circles from the meatus down the glans, three times, a new swab each time |
| Insert | Hold 2–3 in from the tip; advance until urine returns (about 2–3 in), then 2–3 in more | Penis perpendicular to the body with slight upward tension; instil 10 mL of lubricant; patient bears down; hold 1–2 in from the tip; advance to the Y (bifurcation) |
| Inflate | Never force: deep breaths and a gentle twist at resistance. Then inflate with the entire syringe of sterile water and tug gently to feel resistance. Male: replace the foreskin. | |
| Secure | Inner thigh, with slack | Inner thigh or lower abdomen, with slack |
| Bag | Below the bladder, hung on the bed frame, not the side rail; no kinks | |
The non-dominant hand is contaminated once it touches the patient; it stays in place until urine flows and never returns to the sterile field. Document the catheter type and size, the balloon volume, the urine returned and how the patient tolerated it.
Balloon: the checklist's set-up step says a prefilled sterile saline syringe and its inflation step says sterile water; Taylor and ATI use sterile water throughout. Neither the checklist nor Taylor pre-tests the balloon, though older test banks do.
Male, Taylor 10e: lubricate 5–7 in (anaesthetic gel 5–10 mL if used, after an allergy check), hold 3–4 in from the tip, advance 7–10 in until urine, then about 2 in more to the Y. Both versions stop at the Y.
Specimen at insertion: the checklist takes it from the new bag; the deck, Taylor and ATI say the port, never the bag (Part 6). Answer with the port.
Female urethra: Taylor gives 4 cm (1.6 in) in its anatomy section and 1.5–2.5 in in the skill rationale. Use the skill's insertion depths.
| During insertion | What you do |
|---|---|
| Male catheter won't pass beyond 3–4 in | Stop; don't force. Notify; expect a coudé placed by a urologist or advanced practitioner. |
| No urine where you expect it | Don't inflate. Deep breath, rotate slightly, raise the head of the bed, check intake and kinks. Still unsure: remove it and notify. |
| Catheter in the vagina | Leave it as a landmark, insert a new sterile catheter into the meatus, then remove the first. |
| Sterility broken | Stop and correct; what you replace depends on what was contaminated. |
Caring for the catheter, and taking it out top-weighted topic
Seven care rules:
- Meatal care with soap and water daily and after each bowel movement, cleaning the catheter from the meatus outward; no powders, lotions or antiseptics at the meatus.
- Generous fluids unless restricted: natural irrigation.
- Constant downward flow: no kinks, no dependent loops, not lying on the tubing.
- Bag below the bladder at all times, including transport, and off the floor.
- Keep the system closed. If it comes apart, replace the catheter and the bag.
- Empty into the patient's own graduated container at least every 8 hours; the spout touches nothing.
- Report CAUTI signs (UTI table, With a catheter). Change the catheter only when it fails, never on a schedule; remove it as soon as possible.
A leg bag is not a closed system, because it is opened to switch to a night bag; empty it at ½–⅔ full and clean the outlet each time.
Meatal care frequency: Taylor and ATI Engage say daily; ATI Skills Modules say at least once a shift. Follow facility policy.
Three devices with care of their own:
| Device | Care |
|---|---|
| Suprapubic catheter | Inspect the site for redness, swelling, drainage or leaking; clean with soap and water from the site outward; dressing changed daily, clean or sterile per policy; tube secured so it can't pull. Bag and tubing as for a Foley. Report a dislodged tube, no drainage, or an inflamed site. |
| Condom catheter | Inspect the penis first: red, swollen or broken skin means don't apply it. Measure for size; perineal care, dry; trim hair, never shave. Leave 1–2 in at the tip; snug, never constricting, never taped around the shaft; check circulation in about 30 minutes. Bag below the bladder, no kinks, so urine can't pool at the glans. Off daily to wash, dry and inspect. |
| Bladder irrigation | Not routine: fluids by mouth are preferred. When clots are expected (after prostate or bladder surgery): continuous through a triple-lumen catheter, or intermittent, 30–50 mL by syringe through the port, tubing clamped below; both closed. Sterile solution, aseptic technique, room temperature (cold causes spasm). True urine output = total drainage minus irrigant. Discomfort is expected; pain or slowing drainage is not: check for kinks and clots, then report. |
Removal, in five steps:
- Check the record for the balloon volume; empty and measure the bag.
- Attach a syringe and let the balloon push the plunger back. Don't pull it, and never cut the tubing.
- Confirm the volume withdrawn matches the volume charted.
- Withdraw gently as the patient breathes out; stop and notify if you meet resistance.
- Perineal care.
After removal: encourage fluids; record intake and the time and amount of every void for 24 hours, voiding into a hat, and the volume of the first void. Slight burning, urgency or frequency on the first void or two is expected, and so is an accident. After a few days of catheter, watch for retention. If the patient hasn't voided within 8–10 hours, or per policy, notify; bladder-scan first if they feel full.
Many NCLEX sources teach 6–8 hours for the first void.
Urinary diversions, stents and dialysis
| Diversion | What it is | Continent? |
|---|---|---|
| Ileal conduit (urostomy) | The most common. Ureters implanted into a piece of ileum brought out as a stoma. | No: drains continuously into a pouch |
| Cutaneous ureterostomy | The ureters themselves brought to the skin | No |
| Continent diversion (Indiana or Kock pouch) | Internal reservoir of bowel with a catheterisable stoma | Yes: catheterised on a schedule |
| Nephrostomy | Tube through the back into the renal pelvis, often temporary | Bag below kidney level; flush only if ordered |
Stoma assessment. Normal is dark pink to red, moist and shiny, swollen at first and stable in size by about 6–8 weeks. Pale suggests anaemia; dusky, purple or black means poor circulation: notify promptly. Mucus in the urine is expected from any diversion built from bowel. Empty the pouch at ⅓–½ full; change it every 3–7 days, early in the morning before fluids, with the opening cut ⅛ in larger than the stoma and no lotion on the skin.
Stents and dialysis, briefly
| Device | What the nurse does |
|---|---|
| Ureteral stent | Keeps urine flowing from kidney to bladder past a stone, mass, scar or swelling; usually temporary. Never irrigate it. Falling output may mean a blocked stent (hydronephrosis, kidney damage): act at once. Teach: report bright red urine, severe pain, a change in drainage, or infection. |
| Haemodialysis access (AV fistula or graft) | Palpate the thrill and auscultate the bruit (bell); check distal pulses, capillary refill, colour, warmth, movement and sensation. Present means patent; weak or absent: notify, which outranks a trace of painless oedema. No BP, IV or blood draw in that arm (Part 5, Venous access). |
| Peritoneal dialysis | Dialysate in and out through an abdominal catheter; exit-site care is sterile, nurse and patient masked. |
Who does what: delegating urinary care
Delegation turns on the judgement a task needs, not how routine it looks (Exam 3); the nurse validates and interprets what assistive personnel measure.
| Who | Tasks |
|---|---|
| Assistive personnel (AP) | I&O, including emptying drainage bags and urostomy pouches; perineal and routine catheter care; toileting, scheduled and prompted voiding, pad changes, skin cleansing; routine specimens (UA, clean-catch, 24-hour); a condom catheter where policy allows. They report low output, a missed void, cloudy or bloody urine, red skin. |
| Per agency policy | Bladder scan; a sterile port specimen (often kept with licensed nurses); changing an ostomy appliance (LPN). |
| Licensed nurse (LPN within state scope) | Inserting any catheter (surgical asepsis), straight or indwelling; irrigation. |
| RN | Assessing (bladder, incontinence type, skin, stoma, whether a catheter is still needed); planning; teaching; evaluating; interpreting I&O. |
Writing the problem statements
| Problem | Related to | As evidenced by |
|---|---|---|
| Urinary retention | Enlarged prostate; anticholinergics or antidepressants; obstruction | PVR 450 mL on bladder scan; hourly small voids; hesitancy |
| Stress urinary incontinence | Weak pelvic floor muscles | Leaks with coughing, sneezing, laughing |
| Risk for urge incontinence | Caffeine, alcohol, faecal impaction, UTI | None: a risk statement has no AEB |
| Caregiver role strain | Managing a spouse's total incontinence | “I haven't slept through the night in months” |
| Risk for infection | Indwelling urinary catheter | None (risk) |
Urinary function can be the problem (incontinence, altered pattern, retention) or the cause of another (anxiety, caregiver role strain, risk for infection). “Catheterization R/T urinary retention” is wrong (Exam 3): a treatment is not a problem, though it can be a cause, as in the last row.
Six planned goals. Output about equal to intake; fluid and electrolyte balance; bladder emptied completely at regular intervals; reports ease of voiding; intact skin; self-care. Make each measurable: not “will void adequately” but “will void at least 200 mL within 8–10 hours of catheter removal, with a PVR under 50 mL”.
Rapid Review
The numbers, the look-alike pairs, the what-first orders, the findings that flip with the patient, and the questions still open. Each line is taught in full in its part.
Numbers to know cold
Course figures only; another source's number is in grey in its part.
| Number | What it is |
|---|---|
| Na 135–145 · K 3.5–5.0 · Cl 97–107 · Mg 1.3–2.3 | mEq/L. Potassium's range is only 1.5 wide. Sodium below 120 is severe: seizure precautions. |
| Ca 8.6–10.2 · phosphorus 2.5–4.5 | The two in mg/dL; they move in opposite directions. |
| Serum bicarbonate 25–29 · ABG HCO₃⁻ 22–26 | mEq/L. Two tests, two numbers: 27 is normal on a BMP, high on an ABG. |
| pH 7.35–7.45 · PaCO₂ 35–45 · PaO₂ 80–100 | PaCO₂ is the respiratory number; PaO₂ is oxygenation only. |
| 20 : 1 · up to 90% | Bicarbonate to carbonic acid · the share of ECF hydrogen ions the carbonic acid–bicarbonate buffer takes up. Lungs act in minutes, kidneys in hours to days. |
| 285–295 mOsm/kg | Serum osmolality: up in dehydration, down in overhydration. It follows the sodium, so it stays normal when salt and water are lost or gained together. |
| 50–60% · about 45% | Body water as a share of weight: adult · over 60. Infants have more, with more of it in the ECF, which is lost faster. |
| ~70% · ~30% | ICF · ECF, as shares of body water (35–40% and 15–20% of body weight). |
| ~2,600 mL a day | Intake, and output, in health; urine is about 1,500. Judge the balance over 2–3 days. The care-plan outcome is about 2,500 mL a day, averaged over 3 days. |
| 1.5–2 L · 1–2 L | A drinking target to thin secretions · a heart-failure limit. Neither is the 2,600 turnover. |
| +500 · +1,000 mL a day | At least this much extra loss with a fever of 101–103 °F · above 103 °F. |
| 1 kg ≈ 1 L ≈ 2.2 lb · 1 g = 1 mL | Daily weight, the most accurate measure · a weighed pad or diaper. Ice chips count at half. |
| 5% · 10% · 15% | Sudden weight loss: FVD in an adult · in an infant · life-threatening. A gain over 5%: FVE. |
| SBP ≥20 or DBP ≥10 mm Hg | Orthostatic drop: lying, sitting, standing. |
| ≥ 30 mL/h | Minimum adult urine output; below it, report. Per kilogram about 0.5 mL/kg/h; infants make 1–2. |
| < 400 · < 50 mL in 24 h | Oliguria · anuria. |
| SG 1.010–1.025 · pH 4.6–8.2 | Urine. 1.035 is high and 1.002 low under every source. Glucose and protein raise SG. |
| 150–250 mL | Bladder volume at which the urge to void begins. |
| PVR < 50 · > 100 mL | Adequate emptying · not emptying. |
| 10 mEq/h | IV potassium maximum: diluted, by pump, never pushed. 40 mEq takes at least 4 h. |
| mL/h = mL ÷ h · gtt/min = mL/h × drop factor ÷ 60 | Pump · gravity. Macrodrip 10, 15 or 20 gtt/mL; microdrip 60; blood set 10. |
| 20–24 g · 18–20 g | Most adults and routine blood · a rapid transfusion. |
| Every 4 h · 1–2 h · hourly | IV site check: routine · high-risk site · children. Assess the site and the patient, not the pump. |
| 15 min · 4 h | Blood: stay for the first 15 minutes, vital signs at 15 · finish within 4 hours. |
| 14F · 5–10 mL | Usual adult catheter and balloon, filled with sterile water. |
| ~4 cm · ~20 cm | Urethra: female, where you advance 2–3 in past the first urine · male, where you advance to the Y. |
| 8–10 h | No void after catheter removal: notify. |
| Every 8 h at least | Empty an indwelling bag into its own graduated container; a urimeter for hourly output. |
| 1–2 in · daily | Condom catheter: space left at the tip · removed and the skin checked. |
| 30–50 mL · drainage − irrigant | Intermittent bladder irrigation · true urine output. |
| ≤ 6 months | Transient incontinence: appears suddenly, has a treatable cause. |
Easy to confuse: the deciding hook
| Seventeen pairs | The hook |
|---|---|
| Hypovolemia · dehydration | Read the sodium: normal when salt and water go together, high when water goes alone. |
| Hypervolemia · overhydration | Salt and water kept: sodium usually normal. Water alone gained: sodium low. |
| Fluid loss · third spacing | Third-spaced fluid never left, so the weight does not fall. |
| Osmosis · active transport | Water, free · a solute, paid for in ATP. Direction never decides. |
| ADH · aldosterone | ADH keeps water; aldosterone keeps sodium, dumps potassium. ANP is aldosterone's opposite. |
| Hypotonic · hypertonic | The prefix describes the solution: cells swell · cells shrink. D5W acts hypotonic once its sugar is used. |
| Low · high Ca and Mg | Low is twitchy; high is sluggish. |
| The culprit · the rescue | Only the number that matches the pH names the imbalance. |
| Infiltration · extravasation | The same site. What leaked: non-vesicant · vesicant. |
| Infiltration · phlebitis | Cool and pale is a leak; warm and red is inflammation. |
| Midline · PICC | Same arm vein, but a midline's tip stops at or below the axilla: not central. |
| Allergic · overload reaction | Hives · crackles and rising BP. Back pain and shock: hemolytic. |
| Retention · oliguria | Scan: full bladder, emptying problem; empty bladder, production problem. |
| Clean-catch · port specimen | Clean, not sterile · sterile by its route. |
| Urge · overflow | Dribbling with a distended bladder or high PVR is overflow. |
| Stress · functional | A leak triggered by effort is stress; functional means the tract works but access doesn't. |
| Bladder training · prompted voiding | Who runs it: the patient, with intact cognition · the caregiver. |
What do you do first
Every option is a correct action; the order is the item. The tempting error is the famous step, or the call, jumping ahead of stopping the cause.
| Fourteen situations | In order · the tempting error |
|---|---|
| Infiltration | Stop the infusion → remove the catheter → outline, check circulation, elevate → new site |
| Phlebitis | Stop and remove → warm compress → restart elsewhere, never that vein |
| Air embolism | Clamp → left side, head down → call for help, vital signs, SpO₂ · positioning first |
| Fluid overload on an IV | Slow the infusion → raise the head of the bed → notify |
| A bag running behind | Never catch up → recalculate at the ordered rate → consult · speeding it up |
| Transfusion reaction | Stop → keep the vein open with 0.9% NaCl, new tubing → stay, compare vital signs → notify provider and blood bank → send bag, tubing and samples → document · calling first |
| K⁺ 6.2 with KCl running | Stop the potassium → assess, monitor → notify → redraw · calling or redrawing first |
| Breathing fast, tingling, frightened | Check SpO₂ → coach slow breathing · calling it anxiety |
| Hasn't voided | Last void and intake, palpate → bladder-scan → nursing measures → straight catheter per order. Bladder nearly empty: assess fluid status, notify |
| No urine at the Y | Don't inflate → deep breath, rotate, raise the head of the bed, check kinks → still unsure: remove, notify |
| Male catheter won't pass | Stop; don't force → notify; expect a coudé |
| Catheter in the vagina | Leave it as a landmark → new sterile catheter into the meatus → remove the first |
| Dry, dizzy, wants the bathroom | Fall precautions: dangle, rise in stages, walk with help, call light in reach · letting a patient who “walks fine at home” go alone |
| Magnesium drip, RR 10, reflexes gone | Stop the infusion → airway and breathing → notify; calcium gluconate within reach · slowing it |
Same finding, two answers
| Finding | In this patient | In that patient |
|---|---|---|
| K 3.4 | Healthy, eating normally: diet teaching | On digoxin and furosemide: report before the dose |
| Na 128 | Heart failure, edema, crackles: restrict fluid | Days of vomiting, orthostatic drop: sodium and volume |
| Tented skin, back of hand | 84, drinking normally: age; retest over the sternum | 28, after diarrhea: FVD |
| HCO₃⁻ 27 | BMP: normal | ABG: high |
| High PaCO₂ | Acid pH: respiratory acidosis | Alkaline pH after NG suction, HCO₃⁻ 34: the rescue |
| Deep, rapid breathing | Anxiety, hypoxemia ruled out: coach slow breathing | DKA: Kussmaul, the rescue; don't slow it |
| D5W ordered | Na 151 after poor intake: appropriate | Rising intracranial pressure: question it |
| Massive edema | Heart failure with crackles: FVE | Day after a burn, urine 15 mL/h: vessels short; keep fluids running |
| SG 1.003 | After a litre of water: normal response | 15 mL/h, rising creatinine: cannot concentrate |
| Scan 150 mL | Just after voiding: PVR over 100; report | 8 h post-op, no void: a production problem |
| Red urine | After beets: harmless | On warfarin: test for blood, report |
Before you act on a value, find the test, drug, diagnosis or device it belongs to.
Still open: ask the instructor
The first row is flagged “Ask the instructor” where it is taught; the other four are source splits a part has settled for now. Meanwhile, use the third column.
| Question | The versions | Answer with, meanwhile | Where |
|---|---|---|---|
| How far does compensation go? | Program outline: non-compensatory. The deck prints rescue values. Taylor grades partial or full. | Name it from the number matching the pH; skip partial versus full. | Part 4 |
| Normal urine SG | The deck's 1.010–1.025; Taylor's 1.005–1.030 and 1.015–1.025. | 1.010–1.025 | Part 6 |
| First void after removal | Taylor 8–10 h; many NCLEX sources 6–8 h. | 8–10 h, or per policy | Part 7 |
| Balloon fluid | Checklist: saline at set-up, water at inflation. Taylor and ATI: water. | Sterile water | Part 7 |
| PIVC change | ATI: every 72–96 h. Taylor: when clinically indicated. | When clinically indicated | Part 5 |
The Chapter 38 deck settled one former split: a catheter specimen comes from the sampling port, never the bag. The instructor's Chapter 41 lecture slides are still to come; this list will be updated once they are posted.
Summary Recap
Every part in a few minutes, in plain sentences written to be listened to. The full text is folded below.
Read the recap
Recap: Where Body Water Lives, and How It Moves
Body water is 50 to 60 percent of an adult's weight. About 70 percent of it sits inside the cells, which is only 35 to 40 percent of body weight: mind the denominator. After 60, body water falls to about 45 percent; infants keep more of theirs in the ECF, which drains fastest. Intake and output each run about 2,600 mL a day, judged over two to three days. Osmosis moves water toward more particles for free; active transport pushes a solute uphill with ATP. Hydrostatic pressure pushes fluid out of a capillary and albumin pulls it back, so a low albumin lets fluid leak into the tissues while the vessels run short. ADH keeps water; aldosterone keeps sodium and dumps potassium.
Recap: Fluid Volume
Five volume words, sorted by sodium. Hypovolemia loses salt and water together, so sodium stays normal; dehydration loses water alone, so sodium rises. Hypervolemia keeps salt and water; overhydration gains water alone, so sodium falls. Third-spaced fluid has not left the body, so the patient looks dry while the weight holds steady: edema shows where fluid is, not how much the vessels hold. On the stick figure, wet runs high and full: a bounding pulse, engorged neck veins, crackles. Dry runs low and empty, and a fast, weak pulse is the earliest sign. The hematocrit rises when dry and falls when wet. Daily weight is the most accurate measure, a kilogram being about a litre. Decide wet or dry first, then read the sodium. Dry and wet care mirror each other: the dry patient gets fall precautions, oral then isotonic fluids and a close watch for overload during replacement; the wet patient gets Fowler’s, oxygen as ordered, fluid and sodium limits, diuretics with a potassium check, and turning every 2 hours. Third spacing is nursed as a deficit even though the weight holds. Assistive personnel measure, weigh and report; the nurse assesses, interprets and teaches.
Recap: Electrolytes
Learn the seven course normals: sodium 135 to 145, potassium 3.5 to 5.0, magnesium 1.3 to 2.3, chloride 97 to 107 and serum bicarbonate 25 to 29, in milliequivalents; calcium 8.6 to 10.2 and phosphorus 2.5 to 4.5, in milligrams per decilitre. Sodium is chief of the ECF, potassium of the ICF, and calcium is the most abundant in the body. Four patterns carry the grid: potassium threatens the heart, sodium the brain, low calcium and magnesium are twitchy, high ones sluggish. The kidneys cannot conserve potassium; loop diuretics lower it, while spironolactone, ACE inhibitors and renal failure raise it. Rank two abnormal values by the organ threatened, not by distance from normal. Every imbalance shares one care pattern: watch the heart for potassium, calcium and magnesium, the brain for sodium, and breathing and reflexes for magnesium and calcium. Weakness means fall precautions; a very low sodium, calcium or magnesium means seizure precautions; a high magnesium means calcium gluconate within reach. If an infusion produces signs of its own excess, stop it; don’t slow it.
Recap: Acid–Base
Blood pH lives between 7.35 and 7.45, set by a ratio of about 20 parts bicarbonate to 1 part carbonic acid. Buffers act first, and the carbonic acid–bicarbonate system takes up as much as 90 percent of the hydrogen ions; the lungs follow in minutes and the kidneys in hours to days. Read a gas in four steps: the pH, the carbon dioxide, the bicarbonate, then whichever matches the pH names the imbalance; a number reading the other way is the rescue. Vomiting loses acid, so alkalosis; diarrhea loses bicarbonate, so acidosis. In acidosis potassium leaves the cells, so the serum level rises. Bicarbonate is 25 to 29 on an electrolyte panel and 22 to 26 on a gas. Respiratory acidosis care is ventilation first: sit up, cough, use the spirometer, give oxygen as ordered, hold sedation and report. Rule out low oxygen before calling fast breathing anxiety, and stay with the patient. Insulin and fluids for an acidosis drive potassium back into the cells, so recheck it.
Recap: IV Therapy and Blood
Normal saline and lactated Ringer's are isotonic and stay in the vessels; LR contains potassium. Half-normal saline is hypotonic and swells cells; D10W is hypertonic and shrinks them. D5W is isotonic in the bag but acts hypotonic once its sugar is used, so keep it from a swelling brain. Choose a forearm vein, start distal and avoid the elbow crease. Read a site by temperature: cool and pale is a leak, warm and red is inflammation. Every complication starts by stopping the cause: for air embolism, clamp before positioning. Potassium is never pushed and never runs faster than 10 mEq an hour. For blood, two people check, prime with saline only, and stay for 15 minutes; at any reaction, stop, run saline through new tubing, stay, notify, send the bag and samples, and document. Assess the site and the patient, not the pump. Assistive personnel report IV complaints and record intake and output; they never start, adjust, flush or discontinue an IV.
Recap: Urine and Testing
Urine runs from kidney to ureter to bladder to urethra, and the short female urethra invites infection. The urge to void begins at 150 to 250 mL. Report adult output below 30 mL an hour; oliguria is under 400 mL a day and anuria under 50. Normal specific gravity is 1.010 to 1.025 and pH 4.6 to 8.2, but glucose and protein raise the gravity, and dilute urine reassures only when there is plenty of it. A clean-catch is clean, not sterile; a sterile specimen comes from a straight catheter or the sampling port, never the bag. For a 24-hour test, discard the first void and keep the last. A post-void residual over 100 mL means the bladder is not emptying. A urinary assessment runs ask, inspect, palpate or percuss, then measure: ask about the last void before you touch the bladder. Deep palpation of the kidneys belongs to an advanced practitioner; the nurse inspects the meatus, the skin and the urine and checks the bladder. Empty an indwelling bag at least every 8 hours, and weigh pads at 1 gram to 1 millilitre.
Recap: Urinary Problems and Nursing Care
Name incontinence by mechanism. Stress leaks with effort; urge cannot make it in time; overflow dribbles from a bladder that never empties; functional has a working tract and a broken path to the toilet; reflex has no sensation of fullness; transient has a treatable cause. Check emptying before you name the type, and never use an indwelling catheter for incontinence alone. For retention, scan, try nursing measures, then a straight catheter by order. In an older adult, new confusion may be the only sign of a UTI. Most CAUTIs start after placement: avoid the catheter, or remove it early. Advance 2 to 3 inches past the first urine in a female and to the Y in a male before inflating. Every urinary problem has the same five steps of care: assess, act, teach, delegate, evaluate. Urinary function can be the problem itself or the cause of another, such as anxiety or a risk for infection. Assistive personnel measure intake and output, give perineal and catheter care and run toileting schedules; inserting catheters, assessing and teaching stay with the nurse. Never take a blood pressure, start an IV or draw blood in a dialysis access arm.
Which source wins. What is said in class outranks the instructor's own worksheets and checklists, which outrank the Chapter 41 deck, then Taylor, then ATI and Lippincott. The governing figure is printed at full weight and any other in small grey type.
Coverage and policy change by term; check your course site for anything time-sensitive.